Astrocytes are more vulnerable than neurons to cellular Ca2+ overload induced by a mitochondrial toxin, 3-nitropropionic acid

Neuroscience

Volumn 87, Issue 2, 1998, Pages 497-507

  Fukuda, A ^a   Deshpande, S B ^a   Shimano, Y ^a   Nishino, H ^a  

^a NAGOYA CITY UNIV MEDICAL SCHOOL   (Japan)

Author keywords

3 nitropropionic acid; Energy metabolism; Intracellular Ca2+; Mitochondrial toxin; Na+ Ca2+ exchanger; Neuron glia relation

Indexed keywords

3 NITROPROPIONIC ACID; ADENOSINE TRIPHOSPHATE; CALCIUM ION; GLIAL FIBRILLARY ACIDIC PROTEIN;

ANIMAL CELL; APOPTOSIS; ARTICLE; ASTROCYTE; CALCIUM CELL LEVEL; CELL DEATH; CONTROLLED STUDY; ENERGY METABOLISM; FETUS; ION EXCHANGE; MITOCHONDRIAL MEMBRANE; NERVE CELL CULTURE; NONHUMAN; PRIORITY JOURNAL; RAT;

AMILORIDE; ANIMALS; ASTROCYTES; CALCIUM; CALCIUM SIGNALING; CELL DEATH; FEMALE; IMMUNOHISTOCHEMISTRY; NEURONS; NEUROTOXINS; NITRO COMPOUNDS; PREGNANCY; PROPIONIC ACIDS; RATS; RATS, WISTAR; SODIUM-CALCIUM EXCHANGER;

EID: 0032551687     PISSN: 03064522     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0306-4522(98)00139-0     Document Type: Article

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