Aβ[25-35] peptide and iron promote apoptosis in lymphocytes by an oxidative stress mechanism: Involvement of H2O2, caspase-3, NF-κB, p53 and c-Jun

NeuroToxicology

Volumn 23, Issue 3, 2002, Pages 351-365

  Velez Pardo, Carlos ^a   Garcia Ospina, Gloria ^a   Jimenez del Rio, Marlene ^a  

^a UNIVERSIDAD DE ANTIOQUIA   (Colombia)

Author keywords

Amyloid ; Apoptosis; Caspase; Lymphocytes; Stress; Transcription factors

Indexed keywords

AMYLOID BETA PROTEIN; CASPASE 3; DACTINOMYCIN; DIAMINOBENZIDINE; HYDROGEN PEROXIDE; IMMUNOGLOBULIN ENHANCER BINDING PROTEIN; IRON; MESSENGER RNA; PROTEIN C JUN; PROTEIN P53; PYRROLIDINE DITHIOCARBAMATE; RHODAMINE; AMYLOID BETA PROTEIN (25 35); AMYLOID BETA-PROTEIN (25-35); CASP3 PROTEIN, HUMAN; CASPASE; PEPTIDE FRAGMENT; REACTIVE OXYGEN METABOLITE;

ALZHEIMER DISEASE; APOPTOSIS; ARTICLE; DEGENERATIVE DISEASE; ENZYME ACTIVATION; HUMAN; HUMAN CELL; LYMPHOCYTE; MALE; NEUROFIBRILLARY TANGLE; NEUROTOXICITY; OXIDATION REDUCTION REACTION; OXIDATIVE STRESS; PRIORITY JOURNAL; RNA SYNTHESIS; SENILE PLAQUE; ADULT; BIOSYNTHESIS; CELL DEATH; DRUG EFFECT; GEL MOBILITY SHIFT ASSAY; GENETICS; IMMUNOHISTOCHEMISTRY; IN VITRO STUDY; METABOLISM; ONCOGENE; PHYSIOLOGY; TUMOR SUPPRESSOR GENE; WESTERN BLOTTING;

ADULT; AMYLOID BETA-PROTEIN; APOPTOSIS; BLOTTING, WESTERN; CASPASE 3; CASPASES; CELL DEATH; ELECTROPHORETIC MOBILITY SHIFT ASSAY; GENES, JUN; GENES, P53; HUMANS; HYDROGEN PEROXIDE; IMMUNOHISTOCHEMISTRY; IRON; LYMPHOCYTES; MALE; NF-KAPPA B; OXIDATIVE STRESS; PEPTIDE FRAGMENTS; REACTIVE OXYGEN SPECIES; RNA, MESSENGER;

EID: 0036728812     PISSN: 0161813X     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0161-813X(02)00081-5     Document Type: Article

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