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Volumn 51, Issue 2, 2002, Pages 311-316

Interleukin-1 plus γ-interferon-induced pancreatic β-cell dysfunction is mediated by β-cell nitric oxide production

Author keywords

[No Author keywords available]

Indexed keywords

GAMMA INTERFERON; GAMMA INTERFERON RECEPTOR; INSULIN; INTERLEUKIN 1; NITRIC OXIDE; NITRIC OXIDE SYNTHASE;

EID: 0036068246     PISSN: 00121797     EISSN: None     Source Type: Journal    
DOI: 10.2337/diabetes.51.2.311     Document Type: Article
Times cited : (155)

References (41)
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    • An immunohistochemical study of macrophage influx and the co-localization of inducible nitric oxide synthase in the pancreas of non-obese diabetic (NOD) mice during disease acceleration with cyclophosphamide
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    • Reddy, S.1    Yip, S.2    Karanam, M.3    Poole, C.A.4    Ross, J.M.5
  • 17
    • 0028900805 scopus 로고
    • Intraislet release of interleukin 1 inhibits beta cell function by inducing beta cell expression of inducible nitric oxide synthase
    • (1995) J Exp Med , vol.181 , pp. 559-568
    • Corbett, J.A.1    McDaniel, M.L.2
  • 30
    • 0025685395 scopus 로고
    • Inhibition of insulin secretion by intefieukin-1 beta and tumour necrosis factor-alpha via an L-arginine-dependent nitric oxide generating mechanism
    • (1990) FEBS Lett , vol.276 , pp. 42-44
    • Southern, C.1    Schulster, D.2    Green, I.C.3
  • 40
    • 0030890588 scopus 로고    scopus 로고
    • Cell death mediators in autoimmune diabetes: No shortage of suspects
    • (1997) Cell , vol.89 , pp. 1-3
    • Benoist, C.1    Mathis, D.2


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.