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Oncogene

Volumn 20, Issue 33, 2001, Pages 4507-4518

BAD/BCL-xL heterodimerization leads to bypass of G0/G1 arrest

(3) Chattopadhyay, Anuja a Chiang, Chi Wu a Yang, Elizabeth a

a VANDERBILT INGRAM CANCER CENTER (United States)

Author keywords

Apoptosis; BAD; BCL xL; Cell cycle arrest

Indexed keywords

BROXURIDINE; CYCLIN E; DNA; PROTEIN BCL 2;

ANIMAL CELL; APOPTOSIS; ARTICLE; CELL CYCLE G0 PHASE; CELL CYCLE G1 PHASE; CONTROLLED STUDY; DIMERIZATION; EMBRYO; ENZYME ACTIVITY; IMMUNOBLOTTING; IMMUNOPRECIPITATION; MOUSE; MUTATION; NONHUMAN; PHOSPHORYLATION; PRIORITY JOURNAL; PROTEIN BINDING; RAT;

ANIMALS; APOPTOSIS; BCL-ASSOCIATED DEATH PROTEIN; BCL-X PROTEIN; CARRIER PROTEINS; CDC2-CDC28 KINASES; CELL DIVISION; CELLS, CULTURED; CONTACT INHIBITION; CULTURE MEDIA, SERUM-FREE; CYCLIN-DEPENDENT KINASE 2; CYCLIN-DEPENDENT KINASES; DIMERIZATION; DNA REPLICATION; ENZYME ACTIVATION; FIBROBLASTS; G0 PHASE; G1 PHASE; PHOSPHORYLATION; PROTEIN CONFORMATION; PROTEIN HYBRIDIZATION; PROTEIN PROCESSING, POST-TRANSLATIONAL; PROTEIN-SERINE-THREONINE KINASES; PROTO-ONCOGENE PROTEINS C-BCL-2; RATS; RECOMBINANT FUSION PROTEINS; STRUCTURE-ACTIVITY RELATIONSHIP;

ANIMALIA;

EID: 0035958570 PISSN: 09509232 EISSN: None Source Type: Journal
DOI: 10.1038/sj.onc.1204584 Document Type: Article

Times cited : (54)

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