Chronic exposure of human mesangial cells to high glucose environments activates the p38 MAPK pathway

Kidney International

Volumn 60, Issue 3, 2001, Pages 858-871

  Wilmer, William A ^a   Dixon, Cynthia L ^a   Hebert, Courtney ^a  

^a THE OHIO STATE UNIVERSITY COLLEGE OF MEDICINE   (United States)

Author keywords

Activator protein 1; Diabetes; Mitogen activated protein kinase; Osmotic stress; Protein kinase C; Reactive oxygen species

Indexed keywords

2 [1 (3 DIMETHYLAMINOPROPYL) 3 INDOLYL] 3 (3 INDOLYL)MALEIMIDE; 3 [8 [(DIMETHYLAMINO)METHYL] 6,7,8,9 TETRAHYDROPYRIDO[1,2 A]INDOL 10 YL] 4 (1 METHYL 1H INDOL 3 YL) 1H PYRROLE 2,5 DIONE; 4 (4 FLUOROPHENYL) 2 (4 METHYLSULFINYLPHENYL) 5 (4 PYRIDYL)IMIDAZOLE; ACETYLCYSTEINE; DIPHENYLIODONIUM SALT; GLUCOSE; MANNITOL; MITOGEN ACTIVATED PROTEIN KINASE; PHORBOL ESTER; REACTIVE OXYGEN METABOLITE; ROTTLERIN; TRANSCRIPTION FACTOR AP 1;

ARTICLE; CELL CULTURE; CONTROLLED STUDY; DNA BINDING; ELECTROPHORETIC MOBILITY; ENZYME ACTIVATION; ENZYME ACTIVITY; ENZYME ASSAY; ENZYME INHIBITION; HUMAN; HUMAN CELL; IMMUNOPRECIPITATION; MESANGIUM CELL; OSMOTIC STRESS; PRIORITY JOURNAL; PROTEIN PHOSPHORYLATION; WESTERN BLOTTING;

EID: 0035720552     PISSN: 00852538     EISSN: None     Source Type: Journal    
DOI: 10.1046/j.1523-1755.2001.060003858.x     Document Type: Article

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