Effect of overexpression of wild-type and mutant CU/ZN-superoxide dismutases on oxidative damage and antioxidant defences: Relevance to Down's syndrome and familial amyotrophic lateral sclerosis

Journal of Neurochemistry

Volumn 76, Issue 4, 2001, Pages 957-965

  Lee, Moonhee ^a   Hyun, Dong Hoon ^a   Jenner, Peter ^a   Halliwell, Barry ^a,b  

^a KING'S COLLEGE LONDON   (United Kingdom)

^b NATIONAL UNIVERSITY OF SINGAPORE   (Singapore)

Author keywords

Antioxidant defence; Down's syndrome; Familial amyotrophic lateral sclerosis; Mutant SOD1; Oxidative stress; Wild type SOD1

Indexed keywords

3 NITROTYROSINE; 8 HYDROXYGUANINE; ANTIOXIDANT; CARBONYL DERIVATIVE;

AMYOTROPHIC LATERAL SCLEROSIS; ARTICLE; CELL PROLIFERATION; CONTROLLED STUDY; DOWN SYNDROME; ENZYME ACTIVITY; GENETIC TRANSFECTION; GROWTH RATE; HUMAN; HUMAN CELL; LIPID PEROXIDATION; OXIDATIVE STRESS; PRIORITY JOURNAL;

ALDEHYDES; AMYOTROPHIC LATERAL SCLEROSIS; ANTIOXIDANTS; CELL DIVISION; CELL LINE; CELL SURVIVAL; DOWN SYNDROME; GENE EXPRESSION; GLUTATHIONE; GLUTATHIONE DISULFIDE; GUANINE; HUMANS; KETONES; LIPID PEROXIDATION; MUTATION; NEUROBLASTOMA; OXIDATIVE STRESS; SUPEROXIDE DISMUTASE; TERATOCARCINOMA; TRANSFECTION; TYROSINE;

MUS MUSCULUS;

EID: 0035118930     PISSN: 00223042     EISSN: None     Source Type: Journal    
DOI: 10.1046/j.1471-4159.2001.00107.x     Document Type: Article

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