Transgenic animal models of familial amyotrophic lateral sclerosis

Journal of Neurology, Supplement

Volumn 244, Issue 2, 1997, Pages

  Gurney, Mark E ^a  

^a PFIZER INC   (United States)

Author keywords

Amyotrophic lateral sclerosis; Animal disease models; Excitotoxins; Superoxide dismutase; Transgenic mice

Indexed keywords

COPPER ZINC SUPEROXIDE DISMUTASE; FREE RADICAL; GLUTAMIC ACID; MUTANT PROTEIN;

AMINO ACID SUBSTITUTION; AMYOTROPHIC LATERAL SCLEROSIS; ANIMAL EXPERIMENT; ANIMAL MODEL; BRAIN STEM; CLINICAL FEATURE; CONFERENCE PAPER; DISEASE COURSE; GENE MUTATION; MISSENSE MUTATION; MOTONEURON; MOTOR DYSFUNCTION; MOUSE; NERVE DEGENERATION; NEUROTRANSMISSION; NONHUMAN; PATHOGENESIS; PRIORITY JOURNAL; SPINAL CORD; TRANSGENIC ANIMAL;

AMYOTROPHIC LATERAL SCLEROSIS; ANIMALS; DISEASE MODELS, ANIMAL; FOOD, FORTIFIED; MICE; MICE, TRANSGENIC; POINT MUTATION; SELENIUM; SUPEROXIDE DISMUTASE; VITAMIN E;

EID: 0030927253     PISSN: 09391517     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Conference Paper

References (2)

1. Mulder DW (1982) Clinical limits of amyotrophic lateral sclerosis. In: Rowland LP (ed) Human motor neuron diseases. Raven, New York, pp 15-22
2. Emery AEH, Holloway S (1982) Familial motor neuron diseases. In: Rowland LP (ed) Human motor neuron diserts. Raven, New York, pp 139-147