-
1
-
-
0016762220
-
Natural” killer cells in the mouse. I. Cytotoxic cells with specificity for mouse Moloney leukemia cells. Specificity and distribution according to genotype
-
PID: 1234049
-
Kiessling, R., Klein, E. & Wigzell, H. “Natural” killer cells in the mouse. I. Cytotoxic cells with specificity for mouse Moloney leukemia cells. Specificity and distribution according to genotype. Eur. J. Immunol. 5, 112–117 (1975).
-
(1975)
Eur. J. Immunol.
, vol.5
, pp. 112-117
-
-
Kiessling, R.1
Klein, E.2
Wigzell, H.3
-
2
-
-
42449151214
-
Functions of natural killer cells
-
PID: 18425107
-
Vivier, E., Tomasello, E., Baratin, M., Walzer, T. & Ugolini, S. Functions of natural killer cells. Nat. Immunol. 9, 503–510 (2008).
-
(2008)
Nat. Immunol.
, vol.9
, pp. 503-510
-
-
Vivier, E.1
Tomasello, E.2
Baratin, M.3
Walzer, T.4
Ugolini, S.5
-
3
-
-
0022616253
-
Selective rejection of H-2-deficient lymphoma variants suggests alternative immune defence strategy
-
PID: 3951539
-
Kärre, K., Ljunggren, H. G., Piontek, G. & Kiessling, R. Selective rejection of H-2-deficient lymphoma variants suggests alternative immune defence strategy. Nature 319, 675–678 (1986).
-
(1986)
Nature
, vol.319
, pp. 675-678
-
-
Kärre, K.1
Ljunggren, H.G.2
Piontek, G.3
Kiessling, R.4
-
4
-
-
42449088897
-
Natural killer cell recognition of missing self
-
PID: 18425103
-
Kärre, K. Natural killer cell recognition of missing self. Nat. Immunol. 9, 477–480 (2008).
-
(2008)
Nat. Immunol.
, vol.9
, pp. 477-480
-
-
Kärre, K.1
-
5
-
-
0024315206
-
Severe herpesvirus infections in an adolescent without natural killer cells
-
PID: 2543925
-
Biron, C. A., Byron, K. S. & Sullivan, J. L. Severe herpesvirus infections in an adolescent without natural killer cells. N. Engl. J. Med. 320, 1731–1735 (1989).
-
(1989)
N. Engl. J. Med.
, vol.320
, pp. 1731-1735
-
-
Biron, C.A.1
Byron, K.S.2
Sullivan, J.L.3
-
6
-
-
78149419031
-
dim NK-cell differentiation uncoupled from NK-cell education
-
PID: 20696944
-
dim NK-cell differentiation uncoupled from NK-cell education. Blood 116, 3853–3864 (2010).
-
(2010)
Blood
, vol.116
, pp. 3853-3864
-
-
Björkström, N.K.1
-
7
-
-
84924984567
-
Cytomegalovirus infection drives adaptive epigenetic diversification of NK cells with altered signaling and effector function
-
PID: 25786176
-
Schlums, H. et al. Cytomegalovirus infection drives adaptive epigenetic diversification of NK cells with altered signaling and effector function. Immunity 42, 443–456 (2015).
-
(2015)
Immunity
, vol.42
, pp. 443-456
-
-
Schlums, H.1
-
8
-
-
85040809440
-
Adaptive natural killer cells integrate interleukin-18 during target-cell encounter
-
PID: 29387058
-
Hammer, Q., Rückert, T., Dunst, J. & Romagnani, C. Adaptive natural killer cells integrate interleukin-18 during target-cell encounter. Front. Immunol. 8, 1976 (2018).
-
(2018)
Front. Immunol.
, vol.8
, pp. 1976
-
-
Hammer, Q.1
Rückert, T.2
Dunst, J.3
Romagnani, C.4
-
9
-
-
84922590130
-
Cytomegalovirus generates long-lived antigen-specific NK cells with diminished bystander activation to heterologous infection
-
PID: 25422494
-
Min-Oo, G. & Lanier, L. L. Cytomegalovirus generates long-lived antigen-specific NK cells with diminished bystander activation to heterologous infection. J. Exp. Med. 211, 2669–2680 (2014).
-
(2014)
J. Exp. Med.
, vol.211
, pp. 2669-2680
-
-
Min-Oo, G.1
Lanier, L.L.2
-
10
-
-
0037108345
-
Coordinated and distinct roles for IFN-αβ, IL-12, and IL-15 regulation of NK cell responses to viral infection
-
PID: 12370359
-
Nguyen, K. B. et al. Coordinated and distinct roles for IFN-αβ, IL-12, and IL-15 regulation of NK cell responses to viral infection. J. Immunol. 169, 4279–4287 (2002).
-
(2002)
J. Immunol.
, vol.169
, pp. 4279-4287
-
-
Nguyen, K.B.1
-
11
-
-
84863804916
-
Proinflammatory cytokine signaling required for the generation of natural killer cell memory
-
PID: 22493516
-
Sun, J. C. et al. Proinflammatory cytokine signaling required for the generation of natural killer cell memory. J. Exp. Med. 209, 947–954 (2012).
-
(2012)
J. Exp. Med.
, vol.209
, pp. 947-954
-
-
Sun, J.C.1
-
12
-
-
84872225254
-
Sustained effector function of IL-12/15/18-preactivated NK cells against established tumors
-
PID: 23209317
-
Ni, J., Miller, M., Stojanovic, A., Garbi, N. & Cerwenka, A. Sustained effector function of IL-12/15/18-preactivated NK cells against established tumors. J. Exp. Med. 209, 2351–2365 (2012).
-
(2012)
J. Exp. Med.
, vol.209
, pp. 2351-2365
-
-
Ni, J.1
Miller, M.2
Stojanovic, A.3
Garbi, N.4
Cerwenka, A.5
-
13
-
-
84922572267
-
Cutting edge: stage-specific requirement of IL-18 for antiviral NK cell expansion
-
PID: 25589075
-
Madera, S. & Sun, J. C. Cutting edge: stage-specific requirement of IL-18 for antiviral NK cell expansion. J. Immunol. 194, 1408–1412 (2015).
-
(2015)
J. Immunol.
, vol.194
, pp. 1408-1412
-
-
Madera, S.1
Sun, J.C.2
-
15
-
-
84963954943
-
Critical role of CD2 co-stimulation in adaptive natural killer cell responses revealed in NKG2C-deficient humans
-
PID: 27117418
-
Liu, L. L. et al. Critical role of CD2 co-stimulation in adaptive natural killer cell responses revealed in NKG2C-deficient humans. Cell Reports 15, 1088–1099 (2016).
-
(2016)
Cell Reports
, vol.15
, pp. 1088-1099
-
-
Liu, L.L.1
-
16
-
-
84988409805
-
+ T cell help
-
PID: 27757318
-
+ T cell help. OncoImmunology 5, e1219009 (2016).
-
(2016)
OncoImmunology
, vol.5
-
-
Ni, J.1
-
17
-
-
85045118001
-
Peptide-specific recognition of human cytomegalovirus strains controls adaptive natural killer cells
-
+ NK cells
-
+ NK cells.
-
(2018)
Nat. Immunol.
, vol.19
, pp. 453-463
-
-
Hammer, Q.1
-
18
-
-
84901057914
-
The transcription factor Zbtb32 controls the proliferative burst of virus-specific natural killer cells responding to infection
-
PID: 24747678
-
Beaulieu, A. M., Zawislak, C. L., Nakayama, T. & Sun, J. C. The transcription factor Zbtb32 controls the proliferative burst of virus-specific natural killer cells responding to infection. Nat. Immunol. 15, 546–553 (2014).
-
(2014)
Nat. Immunol.
, vol.15
, pp. 546-553
-
-
Beaulieu, A.M.1
Zawislak, C.L.2
Nakayama, T.3
Sun, J.C.4
-
19
-
-
85044749133
-
Core-binding factor β and Runx transcription factors promote adaptive natural killer cell responses
-
PID: 29222089
-
Rapp, M. et al. Core-binding factor β and Runx transcription factors promote adaptive natural killer cell responses. Sci. Immunol. 2, eaan3796 (2017).
-
(2017)
Sci. Immunol.
, vol.2
, pp. eaan3796
-
-
Rapp, M.1
-
20
-
-
84956617633
-
Type I IFN promotes NK cell expansion during viral infection by protecting NK cells against fratricide
-
PID: 26755706
-
Madera, S. et al. Type I IFN promotes NK cell expansion during viral infection by protecting NK cells against fratricide. J. Exp. Med. 213, 225–233 (2016).
-
(2016)
J. Exp. Med.
, vol.213
, pp. 225-233
-
-
Madera, S.1
-
21
-
-
84924955835
-
Epigenetic modification and antibody-dependent expansion of memory-like NK cells in human cytomegalovirus-infected individuals
-
PID: 25786175
-
Lee, J. et al. Epigenetic modification and antibody-dependent expansion of memory-like NK cells in human cytomegalovirus-infected individuals. Immunity 42, 431–442 (2015).
-
(2015)
Immunity
, vol.42
, pp. 431-442
-
-
Lee, J.1
-
22
-
-
84873565353
-
Cutting edge: antibody-dependent memory-like NK cells distinguished by FcRγ deficiency
-
PID: 23345329
-
Zhang, T., Scott, J. M., Hwang, I. & Kim, S. Cutting edge: antibody-dependent memory-like NK cells distinguished by FcRγ deficiency. J. Immunol. 190, 1402–1406 (2013).
-
(2013)
J. Immunol.
, vol.190
, pp. 1402-1406
-
-
Zhang, T.1
Scott, J.M.2
Hwang, I.3
Kim, S.4
-
23
-
-
84923852558
-
bright NK cells against human cytomegalovirus
-
PID: 25667418
-
bright NK cells against human cytomegalovirus. J. Immunol. 194, 2715–2724 (2015).
-
(2015)
J. Immunol.
, vol.194
, pp. 2715-2724
-
-
Costa-Garcia, M.1
-
24
-
-
18844433996
-
Cellular ligands of activating NK receptors
-
PID: 15797513
-
Bottino, C., Castriconi, R., Moretta, L. & Moretta, A. Cellular ligands of activating NK receptors. Trends Immunol. 26, 221–226 (2005).
-
(2005)
Trends Immunol.
, vol.26
, pp. 221-226
-
-
Bottino, C.1
Castriconi, R.2
Moretta, L.3
Moretta, A.4
-
25
-
-
0034895155
-
Strategies for target cell recognition by natural killer cells
-
PID: 11513138
-
Diefenbach, A. & Raulet, D. H. Strategies for target cell recognition by natural killer cells. Immunol. Rev. 181, 170–184 (2001).
-
(2001)
Immunol. Rev.
, vol.181
, pp. 170-184
-
-
Diefenbach, A.1
Raulet, D.H.2
-
26
-
-
84875509728
-
Regulation of ligands for the NKG2D activating receptor
-
PID: 23298206
-
Raulet, D. H., Gasser, S., Gowen, B. G., Deng, W. & Jung, H. Regulation of ligands for the NKG2D activating receptor. Annu. Rev. Immunol. 31, 413–441 (2013).
-
(2013)
Annu. Rev. Immunol.
, vol.31
, pp. 413-441
-
-
Raulet, D.H.1
Gasser, S.2
Gowen, B.G.3
Deng, W.4
Jung, H.5
-
27
-
-
41149093018
-
Evolutionary struggles between NK cells and viruses
-
PID: 18340344
-
Lanier, L. L. Evolutionary struggles between NK cells and viruses. Nat. Rev. Immunol. 8, 259–268 (2008).
-
(2008)
Nat. Rev. Immunol.
, vol.8
, pp. 259-268
-
-
Lanier, L.L.1
-
28
-
-
85027998666
-
Cutting edge: NKG2D signaling enhances NK cell responses but alone is insufficient to drive expansion during mouse cytomegalovirus infection
-
PID: 28760883
-
Nabekura, T. et al. Cutting edge: NKG2D signaling enhances NK cell responses but alone is insufficient to drive expansion during mouse cytomegalovirus infection. J. Immunol. 199, 1567–1571 (2017).
-
(2017)
J. Immunol.
, vol.199
, pp. 1567-1571
-
-
Nabekura, T.1
-
29
-
-
0031572294
-
Mouse homolog of poliovirus receptor-related gene 2 product, mPRR2, mediates homophilic cell aggregation
-
PID: 9299162
-
Aoki, J. et al. Mouse homolog of poliovirus receptor-related gene 2 product, mPRR2, mediates homophilic cell aggregation. Exp. Cell Res. 235, 374–384 (1997).
-
(1997)
Exp. Cell Res.
, vol.235
, pp. 374-384
-
-
Aoki, J.1
-
30
-
-
2542458503
-
DNAM-1 and PVR regulate monocyte migration through endothelial junctions
-
PID: 15136589
-
Reymond, N. et al. DNAM-1 and PVR regulate monocyte migration through endothelial junctions. J. Exp. Med. 199, 1331–1341 (2004).
-
(2004)
J. Exp. Med.
, vol.199
, pp. 1331-1341
-
-
Reymond, N.1
-
31
-
-
78649855627
-
Critical role of DNAX accessory molecule-1 (DNAM-1) in the development of acute graft-versus-host disease in mice
-
PID: 20937876
-
Nabekura, T. et al. Critical role of DNAX accessory molecule-1 (DNAM-1) in the development of acute graft-versus-host disease in mice. Proc. Natl. Acad. Sci. USA 107, 18593–18598 (2010).
-
(2010)
Proc. Natl. Acad. Sci. USA
, vol.107
, pp. 18593-18598
-
-
Nabekura, T.1
-
32
-
-
0344926265
-
Characterization and identification of Tage4 as the murine orthologue of human poliovirus receptor/CD155
-
PID: 14652024
-
Ravens, I., Seth, S., Förster, R. & Bernhardt, G. Characterization and identification of Tage4 as the murine orthologue of human poliovirus receptor/CD155. Biochem. Biophys. Res. Commun. 312, 1364–1371 (2003).
-
(2003)
Biochem. Biophys. Res. Commun.
, vol.312
, pp. 1364-1371
-
-
Ravens, I.1
Seth, S.2
Förster, R.3
Bernhardt, G.4
-
33
-
-
65349131915
-
ATM-ATR-dependent up-regulation of DNAM-1 and NKG2D ligands on multiple myeloma cells by therapeutic agents results in enhanced NK-cell susceptibility and is associated with a senescent phenotype
-
PID: 19098271
-
Soriani, A. et al. ATM-ATR-dependent up-regulation of DNAM-1 and NKG2D ligands on multiple myeloma cells by therapeutic agents results in enhanced NK-cell susceptibility and is associated with a senescent phenotype. Blood 113, 3503–3511 (2009).
-
(2009)
Blood
, vol.113
, pp. 3503-3511
-
-
Soriani, A.1
-
34
-
-
84894261203
-
Costimulatory molecule DNAM-1 is essential for optimal differentiation of memory natural killer cells during mouse cytomegalovirus infection
-
PID: 24440149
-
Nabekura, T. et al. Costimulatory molecule DNAM-1 is essential for optimal differentiation of memory natural killer cells during mouse cytomegalovirus infection. Immunity 40, 225–234 (2014).
-
(2014)
Immunity
, vol.40
, pp. 225-234
-
-
Nabekura, T.1
-
35
-
-
85027922645
-
CD2-CD58 interactions are pivotal for the activation and function of adaptive natural killer cells in human cytomegalovirus infection
-
PID: 27469079
-
Rölle, A. et al. CD2-CD58 interactions are pivotal for the activation and function of adaptive natural killer cells in human cytomegalovirus infection. Eur. J. Immunol. 46, 2420–2425 (2016).
-
(2016)
Eur. J. Immunol.
, vol.46
, pp. 2420-2425
-
-
Rölle, A.1
-
36
-
-
85046669240
-
Suppression of costimulation by human cytomegalovirus promotes evasion of cellular immune defenses
-
PID: 29691324
-
Wang, E. C. Y. et al. Suppression of costimulation by human cytomegalovirus promotes evasion of cellular immune defenses. Proc. Natl. Acad. Sci. USA 115, 4998–5003 (2018).
-
(2018)
Proc. Natl. Acad. Sci. USA
, vol.115
, pp. 4998-5003
-
-
Wang, E.C.Y.1
-
37
-
-
85016146660
-
A viral immunoevasin controls innate immunity by targeting the prototypical natural killer cell receptor family
-
PID: 28340350
-
Aguilar, O. A. et al. A viral immunoevasin controls innate immunity by targeting the prototypical natural killer cell receptor family. Cell 169, 58–71 (2017).
-
(2017)
Cell
, vol.169
, pp. 58-71
-
-
Aguilar, O.A.1
-
38
-
-
0034991427
-
A review of genetic differences between limited and extensively passaged human cytomegalovirus strains
-
PID: 11376481
-
Prichard, M. N., Penfold, M. E., Duke, G. M., Spaete, R. R. & Kemble, G. W. A review of genetic differences between limited and extensively passaged human cytomegalovirus strains. Rev. Med. Virol. 11, 191–200 (2001).
-
(2001)
Rev. Med. Virol.
, vol.11
, pp. 191-200
-
-
Prichard, M.N.1
Penfold, M.E.2
Duke, G.M.3
Spaete, R.R.4
Kemble, G.W.5
-
39
-
-
0021147986
-
Pathogenesis of murine cytomegalovirus infection in natural killer cell-depleted mice
-
PID: 6207307
-
Bukowski, J. F., Woda, B. A. & Welsh, R. M. Pathogenesis of murine cytomegalovirus infection in natural killer cell-depleted mice. J. Virol. 52, 119–128 (1984).
-
(1984)
J. Virol.
, vol.52
, pp. 119-128
-
-
Bukowski, J.F.1
Woda, B.A.2
Welsh, R.M.3
-
40
-
-
0025313177
-
Cmv-1, a genetic locus that controls murine cytomegalovirus replication in the spleen
-
PID: 2159050
-
Scalzo, A. A., Fitzgerald, N. A., Simmons, A., La Vista, A. B. & Shellam, G. R. Cmv-1, a genetic locus that controls murine cytomegalovirus replication in the spleen. J. Exp. Med. 171, 1469–1483 (1990).
-
(1990)
J. Exp. Med.
, vol.171
, pp. 1469-1483
-
-
Scalzo, A.A.1
Fitzgerald, N.A.2
Simmons, A.3
La Vista, A.B.4
Shellam, G.R.5
-
41
-
-
0026750339
-
The effect of the Cmv-1 resistance gene, which is linked to the natural killer cell gene complex, is mediated by natural killer cells
-
PID: 1378069
-
Scalzo, A. A. et al. The effect of the Cmv-1 resistance gene, which is linked to the natural killer cell gene complex, is mediated by natural killer cells. J. Immunol. 149, 581–589 (1992).
-
(1992)
J. Immunol.
, vol.149
, pp. 581-589
-
-
Scalzo, A.A.1
-
42
-
-
0031568266
-
High-resolution linkage map in the proximity of the host resistance locus Cmv1
-
PID: 9027502
-
Depatie, C., Muise, E., Lepage, P., Gros, P. & Vidal, S. M. High-resolution linkage map in the proximity of the host resistance locus Cmv1. Genomics 39, 154–163 (1997).
-
(1997)
Genomics
, vol.39
, pp. 154-163
-
-
Depatie, C.1
Muise, E.2
Lepage, P.3
Gros, P.4
Vidal, S.M.5
-
43
-
-
0032113141
-
Ly-49D and Ly-49H associate with mouse DAP12 and form activating receptors
-
PID: 9647200
-
Smith, K. M., Wu, J., Bakker, A. B., Phillips, J. H. & Lanier, L. L. Ly-49D and Ly-49H associate with mouse DAP12 and form activating receptors. J. Immunol. 161, 7–10 (1998).
-
(1998)
J. Immunol.
, vol.161
, pp. 7-10
-
-
Smith, K.M.1
Wu, J.2
Bakker, A.B.3
Phillips, J.H.4
Lanier, L.L.5
-
44
-
-
3142742513
-
Gain of virulence caused by loss of a gene in murine cytomegalovirus
-
PID: 15220428
-
Bubić, I. et al. Gain of virulence caused by loss of a gene in murine cytomegalovirus. J. Virol. 78, 7536–7544 (2004).
-
(2004)
J. Virol.
, vol.78
, pp. 7536-7544
-
-
Bubić, I.1
-
45
-
-
0037123607
-
Direct recognition of cytomegalovirus by activating and inhibitory NK cell receptors
-
PID: 11950999
-
Arase, H., Mocarski, E. S., Campbell, A. E., Hill, A. B. & Lanier, L. L. Direct recognition of cytomegalovirus by activating and inhibitory NK cell receptors. Science 296, 1323–1326 (2002).
-
(2002)
Science
, vol.296
, pp. 1323-1326
-
-
Arase, H.1
Mocarski, E.S.2
Campbell, A.E.3
Hill, A.B.4
Lanier, L.L.5
-
46
-
-
0037172968
-
Recognition of a virus-encoded ligand by a natural killer cell activation receptor
-
PID: 12060703, refs. 45 and 46, the authors identify the MCMV protein m157 as the specific ligand for the NK cell activating receptor Ly49H and thereby for the first time describe a pathogen-specific recognition mechanism for NK cells
-
Smith, H. R. C. et al. Recognition of a virus-encoded ligand by a natural killer cell activation receptor. Proc. Natl. Acad. Sci. USA 99, 8826–8831 (2002). In refs. 45 and 46, the authors identify the MCMV protein m157 as the specific ligand for the NK cell activating receptor Ly49H and thereby for the first time describe a pathogen-specific recognition mechanism for NK cells.
-
(2002)
Proc. Natl. Acad. Sci. USA
, vol.99
, pp. 8826-8831
-
-
Smith, H.R.C.1
-
47
-
-
0034774942
-
Specific and nonspecific NK cell activation during virus infection
-
PID: 11550009
-
Dokun, A. O. et al. Specific and nonspecific NK cell activation during virus infection. Nat. Immunol. 2, 951–956 (2001).
-
(2001)
Nat. Immunol.
, vol.2
, pp. 951-956
-
-
Dokun, A.O.1
-
48
-
-
59049094662
-
Adaptive immune features of natural killer cells
-
+ NK cells after transfer and infection and, notably, demonstrate superior protection by these cells during secondary infection, showing that NK cells can display memory properties response to MCMV
-
+ NK cells after transfer and infection and, notably, demonstrate superior protection by these cells during secondary infection, showing that NK cells can display memory properties in response to MCMV.
-
(2009)
Nature
, vol.457
, pp. 557-561
-
-
Sun, J.C.1
Beilke, J.N.2
Lanier, L.L.3
-
49
-
-
80051604628
-
Comparing the kinetics of NK cells, CD4, and CD8 T cells in murine cytomegalovirus infection
-
PID: 21697462
-
Schlub, T. E. et al. Comparing the kinetics of NK cells, CD4, and CD8 T cells in murine cytomegalovirus infection. J. Immunol. 187, 1385–1392 (2011).
-
(2011)
J. Immunol.
, vol.187
, pp. 1385-1392
-
-
Schlub, T.E.1
-
50
-
-
0345686735
-
Murine cytomegalovirus m157 mutation and variation leads to immune evasion of natural killer cells
-
PID: 14597723
-
Voigt, V. et al. Murine cytomegalovirus m157 mutation and variation leads to immune evasion of natural killer cells. Proc. Natl. Acad. Sci. USA 100, 13483–13488 (2003).
-
(2003)
Proc. Natl. Acad. Sci. USA
, vol.100
, pp. 13483-13488
-
-
Voigt, V.1
-
51
-
-
2942579946
-
Escape of mutant double-stranded DNA virus from innate immune control
-
PID: 15189739
-
French, A. R. et al. Escape of mutant double-stranded DNA virus from innate immune control. Immunity 20, 747–756 (2004).
-
(2004)
Immunity
, vol.20
, pp. 747-756
-
-
French, A.R.1
-
52
-
-
84879416838
-
Targeting of a natural killer cell receptor family by a viral immunoevasin
-
PID: 23666294
-
Berry, R. et al. Targeting of a natural killer cell receptor family by a viral immunoevasin. Nat. Immunol. 14, 699–705 (2013).
-
(2013)
Nat. Immunol.
, vol.14
, pp. 699-705
-
-
Berry, R.1
-
53
-
-
79251589614
-
Functional consequences of natural sequence variation of murine cytomegalovirus m157 for Ly49 receptor specificity and NK cell activation
-
PID: 21187440
-
Corbett, A. J., Coudert, J. D., Forbes, C. A. & Scalzo, A. A. Functional consequences of natural sequence variation of murine cytomegalovirus m157 for Ly49 receptor specificity and NK cell activation. J. Immunol. 186, 1713–1722 (2011).
-
(2011)
J. Immunol.
, vol.186
, pp. 1713-1722
-
-
Corbett, A.J.1
Coudert, J.D.2
Forbes, C.A.3
Scalzo, A.A.4
-
54
-
-
84916910712
-
Viral MHC class I-like molecule allows evasion of NK cell effector responses in vivo
-
PID: 25392524
-
Pyzik, M. et al. Viral MHC class I-like molecule allows evasion of NK cell effector responses in vivo. J. Immunol. 193, 6061–6069 (2014).
-
(2014)
J. Immunol.
, vol.193
, pp. 6061-6069
-
-
Pyzik, M.1
-
55
-
-
20044375064
-
Epistasis between mouse Klra and major histocompatibility complex class I loci is associated with a new mechanism of natural killer cell-mediated innate resistance to cytomegalovirus infection
-
PID: 15895081
-
Desrosiers, M.-P. et al. Epistasis between mouse Klra and major histocompatibility complex class I loci is associated with a new mechanism of natural killer cell-mediated innate resistance to cytomegalovirus infection. Nat. Genet. 37, 593–599 (2005).
-
(2005)
Nat. Genet.
, vol.37
, pp. 593-599
-
-
Desrosiers, M.P.1
-
56
-
-
79956109908
-
Distinct MHC class I-dependent NK cell-activating receptors control cytomegalovirus infection in different mouse strains
-
PID: 21518798, ref. 56, additional receptors of the Ly49 family are reported to specifically recognize MCMV via the viral ligand m04 mouse strains other than C57BL/6, highlighting the general importance of this recognition mode of NK cells
-
Pyzik, M. et al. Distinct MHC class I-dependent NK cell-activating receptors control cytomegalovirus infection in different mouse strains. J. Exp. Med. 208, 1105–1117 (2011). In ref. 56, additional receptors of the Ly49 family are reported to specifically recognize MCMV via the viral ligand m04 in mouse strains other than C57BL/6, highlighting the general importance of this recognition mode of NK cells.
-
(2011)
J. Exp. Med.
, vol.208
, pp. 1105-1117
-
-
Pyzik, M.1
-
57
-
-
63449094484
-
Ly49P recognition of cytomegalovirus-infected cells expressing H2-Dk and CMV-encoded m04 correlates with the NK cell antiviral response
-
PID: 19255146
-
Kielczewska, A. et al. Ly49P recognition of cytomegalovirus-infected cells expressing H2-Dk and CMV-encoded m04 correlates with the NK cell antiviral response. J. Exp. Med. 206, 515–523 (2009).
-
(2009)
J. Exp. Med.
, vol.206
, pp. 515-523
-
-
Kielczewska, A.1
-
58
-
-
78649417356
-
Cytomegalovirus immunoevasin reveals the physiological role of ‘missing self’ recognition in natural killer cell dependent virus control in vivo
-
PID: 21078887
-
Babić, M. et al. Cytomegalovirus immunoevasin reveals the physiological role of ‘missing self’ recognition in natural killer cell dependent virus control in vivo. J. Exp. Med. 207, 2663–2673 (2010).
-
(2010)
J. Exp. Med.
, vol.207
, pp. 2663-2673
-
-
Babić, M.1
-
59
-
-
9444273448
-
Imprint of human cytomegalovirus infection on the NK cell receptor repertoire
-
+ NK cells, setting the stage for further studies of this population humans
-
+ NK cells, setting the stage for further studies of this population in humans.
-
(2004)
Blood
, vol.104
, pp. 3664-3671
-
-
Gumá, M.1
-
60
-
-
50949100398
-
Human NK cells can control CMV infection in the absence of T cells
-
PID: 18650467
-
Kuijpers, T. W. et al. Human NK cells can control CMV infection in the absence of T cells. Blood 112, 914–915 (2008).
-
(2008)
Blood
, vol.112
, pp. 914-915
-
-
Kuijpers, T.W.1
-
61
-
-
80052602721
-
hi natural killer cell subset during acute human cytomegalovirus infection
-
PID: 21825173
-
hi natural killer cell subset during acute human cytomegalovirus infection. Proc. Natl. Acad. Sci. USA 108, 14725–14732 (2011).
-
(2011)
Proc. Natl. Acad. Sci. USA
, vol.108
, pp. 14725-14732
-
-
Lopez-Vergès, S.1
-
62
-
-
84863338121
-
+ natural killer cells with potent function
-
PID: 22180440
-
+ natural killer cells with potent function. Blood 119, 2665–2674 (2012).
-
(2012)
Blood
, vol.119
, pp. 2665-2674
-
-
Foley, B.1
-
63
-
-
84868593211
-
+ NK cells are transplantable and expand in vivo in response to recipient CMV antigen
-
PID: 23077239
-
+ NK cells are transplantable and expand in vivo in response to recipient CMV antigen. J. Immunol. 189, 5082–5088 (2012).
-
(2012)
J. Immunol.
, vol.189
, pp. 5082-5088
-
-
Foley, B.1
-
64
-
-
84856055597
-
+ NK cells expressing self-specific KIRs in chronic hepatitis patients
-
PID: 22105371
-
+ NK cells expressing self-specific KIRs in chronic hepatitis patients. Eur. J. Immunol. 42, 447–457 (2012).
-
(2012)
Eur. J. Immunol.
, vol.42
, pp. 447-457
-
-
Béziat, V.1
-
65
-
-
84876878182
-
NK cell responses to cytomegalovirus infection lead to stable imprints in the human KIR repertoire and involve activating KIRs
-
PID: 23325834
-
Béziat, V. et al. NK cell responses to cytomegalovirus infection lead to stable imprints in the human KIR repertoire and involve activating KIRs. Blood 121, 2678–2688 (2013).
-
(2013)
Blood
, vol.121
, pp. 2678-2688
-
-
Béziat, V.1
-
66
-
-
84896716290
-
Human cytomegalovirus infection promotes rapid maturation of NK cells expressing activating killer Ig-like receptor in patients transplanted with NKG2C-/- umbilical cord blood
-
PID: 24442432
-
Della Chiesa, M. et al. Human cytomegalovirus infection promotes rapid maturation of NK cells expressing activating killer Ig-like receptor in patients transplanted with NKG2C-/- umbilical cord blood. J. Immunol. 192, 1471–1479 (2014).
-
(2014)
J. Immunol.
, vol.192
, pp. 1471-1479
-
-
Della Chiesa, M.1
-
67
-
-
0032100703
-
Association of DAP12 with activating CD94/NKG2C NK cell receptors
-
PID: 9655483
-
Lanier, L. L., Corliss, B., Wu, J. & Phillips, J. H. Association of DAP12 with activating CD94/NKG2C NK cell receptors. Immunity 8, 693–701 (1998).
-
(1998)
Immunity
, vol.8
, pp. 693-701
-
-
Lanier, L.L.1
Corliss, B.2
Wu, J.3
Phillips, J.H.4
-
68
-
-
0032546001
-
HLA-E binds to natural killer cell receptors CD94/NKG2A, B and C
-
PID: 9486650
-
Braud, V. M. et al. HLA-E binds to natural killer cell receptors CD94/NKG2A, B and C. Nature 391, 795–799 (1998).
-
(1998)
Nature
, vol.391
, pp. 795-799
-
-
Braud, V.M.1
-
69
-
-
0032574781
-
HLA-E is a major ligand for the natural killer inhibitory receptor CD94/NKG2A
-
PID: 9560253
-
Lee, N. et al. HLA-E is a major ligand for the natural killer inhibitory receptor CD94/NKG2A. Proc. Natl. Acad. Sci. USA 95, 5199–5204 (1998).
-
(1998)
Proc. Natl. Acad. Sci. USA
, vol.95
, pp. 5199-5204
-
-
Lee, N.1
-
70
-
-
0032933419
-
Specific recognition of HLA-E, but not classical, HLA class I molecules by soluble CD94/NKG2A and NK cells
-
PID: 9886400, The studies refs. 68–70 describe HLA-E as the ligand for receptors of the NKG2 family and how the presentation of leader peptides on HLA-E allows NK cells to surveil the expression of classical HLA class I
-
Brooks, A. G. et al. Specific recognition of HLA-E, but not classical, HLA class I molecules by soluble CD94/NKG2A and NK cells. J. Immunol. 162, 305–313 (1999). The studies in refs. 68–70 describe HLA-E as the ligand for receptors of the NKG2 family and how the presentation of leader peptides on HLA-E allows NK cells to surveil the expression of classical HLA class I.
-
(1999)
J. Immunol.
, vol.162
, pp. 305-313
-
-
Brooks, A.G.1
-
71
-
-
33646422721
-
+ NK cells in response to human cytomegalovirus-infected fibroblasts
-
PID: 16384928
-
+ NK cells in response to human cytomegalovirus-infected fibroblasts. Blood 107, 3624–3631 (2006).
-
(2006)
Blood
, vol.107
, pp. 3624-3631
-
-
Gumá, M.1
-
72
-
-
84915820649
-
+ NK cell expansion
-
PID: 25384219
-
+ NK cell expansion. J. Clin. Invest. 124, 5305–5316 (2014).
-
(2014)
J. Clin. Invest.
, vol.124
, pp. 5305-5316
-
-
Rölle, A.1
-
73
-
-
0030988540
-
The human major histocompatibility complex class Ib molecule HLA-E binds signal sequence-derived peptides with primary anchor residues at positions 2 and 9
-
PID: 9174606
-
Braud, V., Jones, E. Y. & McMichael, A. The human major histocompatibility complex class Ib molecule HLA-E binds signal sequence-derived peptides with primary anchor residues at positions 2 and 9. Eur. J. Immunol. 27, 1164–1169 (1997).
-
(1997)
Eur. J. Immunol.
, vol.27
, pp. 1164-1169
-
-
Braud, V.1
Jones, E.Y.2
McMichael, A.3
-
74
-
-
0037011156
-
A signal peptide derived from hsp60 binds HLA-E and interferes with CD94/NKG2A recognition
-
PID: 12461076
-
Michaëlsson, J. et al. A signal peptide derived from hsp60 binds HLA-E and interferes with CD94/NKG2A recognition. J. Exp. Med. 196, 1403–1414 (2002).
-
(2002)
J. Exp. Med.
, vol.196
, pp. 1403-1414
-
-
Michaëlsson, J.1
-
75
-
-
0031692643
-
HLA-E-bound peptides influence recognition by inhibitory and triggering CD94/NKG2 receptors: preferential response to an HLA-G-derived nonamer
-
PID: 9754572
-
Llano, M. et al. HLA-E-bound peptides influence recognition by inhibitory and triggering CD94/NKG2 receptors: preferential response to an HLA-G-derived nonamer. Eur. J. Immunol. 28, 2854–2863 (1998).
-
(1998)
Eur. J. Immunol.
, vol.28
, pp. 2854-2863
-
-
Llano, M.1
-
76
-
-
0345583696
-
Kinetics and peptide dependency of the binding of the inhibitory NK receptor CD94/NKG2-A and the activating receptor CD94/NKG2-C to HLA-E
-
PID: 10428963
-
Valés-Gómez, M., Reyburn, H. T., Erskine, R. A., López-Botet, M. & Strominger, J. L. Kinetics and peptide dependency of the binding of the inhibitory NK receptor CD94/NKG2-A and the activating receptor CD94/NKG2-C to HLA-E. EMBO J. 18, 4250–4260 (1999).
-
(1999)
EMBO J.
, vol.18
, pp. 4250-4260
-
-
Valés-Gómez, M.1
Reyburn, H.T.2
Erskine, R.A.3
López-Botet, M.4
Strominger, J.L.5
-
77
-
-
0034782592
-
Synergistic effect of IFN-γ and human cytomegalovirus protein UL40 in the HLA-E-dependent protection from NK cell-mediated cytotoxicity
-
PID: 11592068
-
Cerboni, C. et al. Synergistic effect of IFN-γ and human cytomegalovirus protein UL40 in the HLA-E-dependent protection from NK cell-mediated cytotoxicity. Eur. J. Immunol. 31, 2926–2935 (2001).
-
(2001)
Eur. J. Immunol.
, vol.31
, pp. 2926-2935
-
-
Cerboni, C.1
-
78
-
-
0034635414
-
Surface expression of HLA-E, an inhibitor of natural killer cells, enhanced by human cytomegalovirus gpUL40
-
PID: 10669413
-
Tomasec, P. et al. Surface expression of HLA-E, an inhibitor of natural killer cells, enhanced by human cytomegalovirus gpUL40. Science 287, 1031 (2000).
-
(2000)
Science
, vol.287
, pp. 1031
-
-
Tomasec, P.1
-
79
-
-
0034658344
-
Cutting edge: the human cytomegalovirus UL40 gene product contains a ligand for HLA-E and prevents NK cell-mediated lysis
-
+ NK cells
-
+ NK cells.
-
(2000)
J. Immunol.
, vol.164
, pp. 5019-5022
-
-
Ulbrecht, M.1
-
80
-
-
0037188543
-
UL40-mediated NK evasion during productive infection with human cytomegalovirus
-
PID: 12032324
-
Wang, E. C. Y. et al. UL40-mediated NK evasion during productive infection with human cytomegalovirus. Proc. Natl. Acad. Sci. USA 99, 7570–7575 (2002).
-
(2002)
Proc. Natl. Acad. Sci. USA
, vol.99
, pp. 7570-7575
-
-
Wang, E.C.Y.1
-
81
-
-
34548774511
-
Variability of UL18, UL40, UL111a and US3 immunomodulatory genes among human cytomegalovirus clinical isolates from renal transplant recipients
-
PID: 17827058
-
Garrigue, I. et al. Variability of UL18, UL40, UL111a and US3 immunomodulatory genes among human cytomegalovirus clinical isolates from renal transplant recipients. J. Clin. Virol. 40, 120–128 (2007).
-
(2007)
J. Clin. Virol.
, vol.40
, pp. 120-128
-
-
Garrigue, I.1
-
82
-
-
56049109070
-
UL40 human cytomegalovirus variability evolution patterns over time in renal transplant recipients
-
PID: 18813108
-
Garrigue, I. et al. UL40 human cytomegalovirus variability evolution patterns over time in renal transplant recipients. Transplantation 86, 826–835 (2008).
-
(2008)
Transplantation
, vol.86
, pp. 826-835
-
-
Garrigue, I.1
-
83
-
-
84875438461
-
Polymorphism in human cytomegalovirus UL40 impacts on recognition of human leukocyte antigen-E (HLA-E) by natural killer cells
-
PID: 23335510
-
Heatley, S. L. et al. Polymorphism in human cytomegalovirus UL40 impacts on recognition of human leukocyte antigen-E (HLA-E) by natural killer cells. J. Biol. Chem. 288, 8679–8690 (2013).
-
(2013)
J. Biol. Chem.
, vol.288
, pp. 8679-8690
-
-
Heatley, S.L.1
-
84
-
-
84871267470
-
Influence of congenital human cytomegalovirus infection and the NKG2C genotype on NK-cell subset distribution in children
-
PID: 22965785
-
Noyola, D. E. et al. Influence of congenital human cytomegalovirus infection and the NKG2C genotype on NK-cell subset distribution in children. Eur. J. Immunol. 42, 3256–3266 (2012).
-
(2012)
Eur. J. Immunol.
, vol.42
, pp. 3256-3266
-
-
Noyola, D.E.1
-
85
-
-
85017189025
-
Modulation of human leukocyte antigen-c by human cytomegalovirus stimulates KIR2DS1 recognition by natural killer cells
-
PID: 28424684
-
van der Ploeg, K. et al. Modulation of human leukocyte antigen-c by human cytomegalovirus stimulates KIR2DS1 recognition by natural killer cells. Front. Immunol. 8, 298 (2017).
-
(2017)
Front. Immunol.
, vol.8
, pp. 298
-
-
van der Ploeg, K.1
-
86
-
-
85019713163
-
Peptide-specific engagement of the activating NK cell receptor KIR2DS1
-
PID: 28546555
-
Chapel, A. et al. Peptide-specific engagement of the activating NK cell receptor KIR2DS1. Sci. Rep. 7, 2414 (2017).
-
(2017)
Sci. Rep.
, vol.7
-
-
Chapel, A.1
-
87
-
-
81055125785
-
Characterization of natural killer cell phenotype and function during recurrent human HSV-2 infection
-
PID: 22110712
-
Björkström, N. K., Svensson, A., Malmberg, K.-J., Eriksson, K. & Ljunggren, H.-G. Characterization of natural killer cell phenotype and function during recurrent human HSV-2 infection. PLoS One 6, e27664 (2011).
-
(2011)
PLoS One
, vol.6
-
-
Björkström, N.K.1
Svensson, A.2
Malmberg, K.J.3
Eriksson, K.4
Ljunggren, H.G.5
-
88
-
-
84887455253
-
A distinct subpopulation of human NK cells restricts B cell transformation by EBV
-
PID: 24108698
-
Lünemann, A., Vanoaica, L. D., Azzi, T., Nadal, D. & Münz, C. A distinct subpopulation of human NK cells restricts B cell transformation by EBV. J. Immunol. 191, 4989–4995 (2013).
-
(2013)
J. Immunol.
, vol.191
, pp. 4989-4995
-
-
Lünemann, A.1
Vanoaica, L.D.2
Azzi, T.3
Nadal, D.4
Münz, C.5
-
89
-
-
84908147824
-
Role for early-differentiated natural killer cells in infectious mononucleosis
-
PID: 25205117
-
Azzi, T. et al. Role for early-differentiated natural killer cells in infectious mononucleosis. Blood 124, 2533–2543 (2014).
-
(2014)
Blood
, vol.124
, pp. 2533-2543
-
-
Azzi, T.1
-
90
-
-
84875529000
-
Control of human viral infections by natural killer cells
-
PID: 23298212
-
Jost, S. & Altfeld, M. Control of human viral infections by natural killer cells. Annu. Rev. Immunol. 31, 163–194 (2013).
-
(2013)
Annu. Rev. Immunol.
, vol.31
, pp. 163-194
-
-
Jost, S.1
Altfeld, M.2
-
91
-
-
85041860975
-
Rewiring cellular networks by members of the Flaviviridae family
-
PID: 29430005
-
Neufeldt, C. J., Cortese, M., Acosta, E. G. & Bartenschlager, R. Rewiring cellular networks by members of the Flaviviridae family. Nat. Rev. Microbiol. 16, 125–142 (2018).
-
(2018)
Nat. Rev. Microbiol.
, vol.16
, pp. 125-142
-
-
Neufeldt, C.J.1
Cortese, M.2
Acosta, E.G.3
Bartenschlager, R.4
-
92
-
-
85042641418
-
KIR2DS2 recognizes conserved peptides derived from viral helicases in the context of HLA-C
-
PID: 28916719, ref. 92, the authors report a peptide from the HCV viralhelicase NS3 that is conserved among flaviviruses and specifically binds to KIR2DS2 and elicitsresponses by KIR2DS2 NK cells to HCV-infected cells
-
Naiyer, M. M. et al. KIR2DS2 recognizes conserved peptides derived from viral helicases in the context of HLA-C. Sci. Immunol. 2, eaal5296 (2017).In ref. 92, the authors report a peptide from the HCV viralhelicase NS3 that is conserved among flaviviruses and specifically binds to KIR2DS2 and elicitsresponses by KIR2DS2 NK cells to HCV-infected cells.
-
(2017)
Sci. Immunol.
, vol.2
, pp. eaal5296
-
-
Naiyer, M.M.1
-
93
-
-
0033920304
-
Hepatitis C virus-encoded enzymatic activities and conserved RNA elements in the 3′ nontranslated region are essential for virus replication in vivo
-
PID: 10644379
-
Kolykhalov, A. A., Mihalik, K., Feinstone, S. M. & Rice, C. M. Hepatitis C virus-encoded enzymatic activities and conserved RNA elements in the 3′ nontranslated region are essential for virus replication in vivo. J. Virol. 74, 2046–2051 (2000).
-
(2000)
J. Virol.
, vol.74
, pp. 2046-2051
-
-
Kolykhalov, A.A.1
Mihalik, K.2
Feinstone, S.M.3
Rice, C.M.4
-
94
-
-
72549104093
-
Natural killer cells are polarized toward cytotoxicity in chronic hepatitis C in an interferon-alfa-dependent manner
-
PID: 19747917
-
Ahlenstiel, G. et al. Natural killer cells are polarized toward cytotoxicity in chronic hepatitis C in an interferon-alfa-dependent manner. Gastroenterology 138, 325–35.e1–2 (2010).
-
(2010)
Gastroenterology
, vol.138
, pp. 325
-
-
Ahlenstiel, G.1
-
95
-
-
85019997687
-
Cytomegalovirus-driven adaptive-like natural killer cell expansions are unaffected by concurrent chronic hepatitis virus infections
-
PID: 28533779
-
Malone, D. F. G. et al. Cytomegalovirus-driven adaptive-like natural killer cell expansions are unaffected by concurrent chronic hepatitis virus infections. Front. Immunol. 8, 525 (2017).
-
(2017)
Front. Immunol.
, vol.8
, pp. 525
-
-
Malone, D.F.G.1
-
96
-
-
84962724284
-
Longitudinal analysis of natural killer cells in dengue virus-infected patients in comparison to chikungunya and chikungunya/dengue virus-infected patients
-
PID: 26938618
-
Petitdemange, C. et al. Longitudinal analysis of natural killer cells in dengue virus-infected patients in comparison to chikungunya and chikungunya/dengue virus-infected patients. PLoS Negl. Trop. Dis. 10, e0004499 (2016).
-
(2016)
PLoS Negl. Trop. Dis.
, vol.10
-
-
Petitdemange, C.1
-
97
-
-
84971656734
-
Sequence variations in HCV core-derived epitopes alter binding of KIR2DL3 to HLA-C*03:04 and modulate NK cell function
-
PID: 27057987
-
Lunemann, S. et al. Sequence variations in HCV core-derived epitopes alter binding of KIR2DL3 to HLA-C*03:04 and modulate NK cell function. J. Hepatol. 65, 252–258 (2016).
-
(2016)
J. Hepatol.
, vol.65
, pp. 252-258
-
-
Lunemann, S.1
-
98
-
-
0032528340
-
Direct binding and functional transfer of NK cell inhibitory receptors reveal novel patterns of HLA-C allotype recognition
-
PID: 9670929
-
Winter, C. C., Gumperz, J. E., Parham, P., Long, E. O. & Wagtmann, N. Direct binding and functional transfer of NK cell inhibitory receptors reveal novel patterns of HLA-C allotype recognition. J. Immunol. 161, 571–577 (1998).
-
(1998)
J. Immunol.
, vol.161
, pp. 571-577
-
-
Winter, C.C.1
Gumperz, J.E.2
Parham, P.3
Long, E.O.4
Wagtmann, N.5
-
99
-
-
3843075120
-
HLA and NK cell inhibitory receptor genes in resolving hepatitis C virus infection
-
PID: 15297676
-
Khakoo, S. I. et al. HLA and NK cell inhibitory receptor genes in resolving hepatitis C virus infection. Science 305, 872–874 (2004).
-
(2004)
Science
, vol.305
, pp. 872-874
-
-
Khakoo, S.I.1
-
100
-
-
77950626070
-
Consistent beneficial effects of killer cell immunoglobulin-like receptor 2DL3 and group 1 human leukocyte antigen-C following exposure to hepatitis C virus
-
PID: 20077564
-
Knapp, S. et al. Consistent beneficial effects of killer cell immunoglobulin-like receptor 2DL3 and group 1 human leukocyte antigen-C following exposure to hepatitis C virus. Hepatology 51, 1168–1175 (2010).
-
(2010)
Hepatology
, vol.51
, pp. 1168-1175
-
-
Knapp, S.1
-
101
-
-
0037384061
-
Innate immunity in human immunodeficiency virus infection: effect of viremia on natural killer cell function
-
PID: 12660917
-
Kottilil, S. et al. Innate immunity in human immunodeficiency virus infection: effect of viremia on natural killer cell function. J. Infect. Dis. 187, 1038–1045 (2003).
-
(2003)
J. Infect. Dis.
, vol.187
, pp. 1038-1045
-
-
Kottilil, S.1
-
102
-
-
0035942184
-
Control of HIV-1 viremia and protection from AIDS are associated with HLA-Bw4 homozygosity
-
PID: 11309482
-
Flores-Villanueva, P. O. et al. Control of HIV-1 viremia and protection from AIDS are associated with HLA-Bw4 homozygosity. Proc. Natl. Acad. Sci. USA 98, 5140–5145 (2001).
-
(2001)
Proc. Natl. Acad. Sci. USA
, vol.98
, pp. 5140-5145
-
-
Flores-Villanueva, P.O.1
-
103
-
-
0036699528
-
Epistatic interaction between KIR3DS1 and HLA-B delays the progression to AIDS
-
PID: 12134147
-
Martin, M. P. et al. Epistatic interaction between KIR3DS1 and HLA-B delays the progression to AIDS. Nat. Genet. 31, 429–434 (2002).
-
(2002)
Nat. Genet.
, vol.31
, pp. 429-434
-
-
Martin, M.P.1
-
104
-
-
36549019907
-
Differential natural killer cell-mediated inhibition of HIV-1 replication based on distinct KIR/HLA subtypes
-
PID: 18025129
-
Alter, G. et al. Differential natural killer cell-mediated inhibition of HIV-1 replication based on distinct KIR/HLA subtypes. J. Exp. Med. 204, 3027–3036 (2007).
-
(2007)
J. Exp. Med.
, vol.204
, pp. 3027-3036
-
-
Alter, G.1
-
105
-
-
67449101826
-
+ NK cells during acute human immunodeficiency virus type 1 infection
-
PID: 19386717
-
+ NK cells during acute human immunodeficiency virus type 1 infection. J. Virol. 83, 6798–6805 (2009).
-
(2009)
J. Virol.
, vol.83
, pp. 6798-6805
-
-
Alter, G.1
-
106
-
-
34247208503
-
Lack of KIR3DS1 binding to MHC class I Bw4 tetramers in complex with CD8+ T cell epitopes
-
PID: 17411378
-
Gillespie, G. M. A. et al. Lack of KIR3DS1 binding to MHC class I Bw4 tetramers in complex with CD8+ T cell epitopes. AIDS Res. Hum. Retroviruses 23, 451–455 (2007).
-
(2007)
AIDS Res. Hum. Retroviruses
, vol.23
, pp. 451-455
-
-
Gillespie, G.M.A.1
-
107
-
-
84928579211
-
Peptide-dependent recognition of HLA-B*57:01 by KIR3DS1
-
PID: 25740999
-
O’Connor, G. M. et al. Peptide-dependent recognition of HLA-B*57:01 by KIR3DS1. J. Virol. 89, 5213–5221 (2015).
-
(2015)
J. Virol.
, vol.89
, pp. 5213-5221
-
-
O’Connor, G.M.1
-
108
-
-
80052691225
-
Analysis of binding of KIR3DS1*014 to HLA suggests distinct evolutionary history of KIR3DS1
-
PID: 21804024
-
O’Connor, G. M. et al. Analysis of binding of KIR3DS1*014 to HLA suggests distinct evolutionary history of KIR3DS1. J. Immunol. 187, 2162–2171 (2011).
-
(2011)
J. Immunol.
, vol.187
, pp. 2162-2171
-
-
O’Connor, G.M.1
-
109
-
-
84980357055
-
Open conformers of HLA-F are high-affinity ligands of the activating NK-cell receptor KIR3DS1
-
PID: 27455421
-
Garcia-Beltran, W. F. et al. Open conformers of HLA-F are high-affinity ligands of the activating NK-cell receptor KIR3DS1. Nat. Immunol. 17, 1067–1074 (2016).
-
(2016)
Nat. Immunol.
, vol.17
, pp. 1067-1074
-
-
Garcia-Beltran, W.F.1
-
110
-
-
79961138654
-
HIV-1 adaptation to NK-cell-mediated immune pressure
-
PID: 21814282
-
Alter, G. et al. HIV-1 adaptation to NK-cell-mediated immune pressure. Nature 476, 96–100 (2011). In ref. 110, the authors elucidate the basis for a genetic correlation of KIR2DL2 with HIV polymorphisms by demonstrating differential inhibition of KIR2DL2 + NK cells by peptides encoded by these polymorphic regions of the HIV genome.
-
(2011)
Nature
, vol.476
, pp. 96-100
-
-
Alter, G.1
-
111
-
-
84864575731
-
HLA-Cw*0102-restricted HIV-1p24 epitope variants can modulate the binding of the inhibitory KIR2DL2 receptor and primary NK cell function
-
PID: 22807681
-
Fadda, L. et al. HLA-Cw*0102-restricted HIV-1p24 epitope variants can modulate the binding of the inhibitory KIR2DL2 receptor and primary NK cell function. PLoS Pathog. 8, e1002805 (2012).
-
(2012)
PLoS Pathog.
, vol.8
-
-
Fadda, L.1
-
112
-
-
84902275073
-
Sequence variations in HIV-1 p24 Gag-derived epitopes can alter binding of KIR2DL2 to HLA-C*03:04 and modulate primary natural killer cell function
-
PID: 24785948
-
van Teijlingen, N. H. et al. Sequence variations in HIV-1 p24 Gag-derived epitopes can alter binding of KIR2DL2 to HLA-C*03:04 and modulate primary natural killer cell function. AIDS 28, 1399–1408 (2014).
-
(2014)
AIDS
, vol.28
, pp. 1399-1408
-
-
van Teijlingen, N.H.1
-
113
-
-
84949473327
-
+ natural killer cells: data from an observational cohort in South Africa
-
PID: 26575988
-
+ natural killer cells: data from an observational cohort in South Africa. PLoS Med. 12, e1001900 (2015).
-
(2015)
PLoS Med.
, vol.12
-
-
Hölzemer, A.1
-
114
-
-
85029726801
-
– natural killer cells are associated with a lower viral set point and may predict disease progression in individuals with primary HIV infection
-
PID: 28979268
-
– natural killer cells are associated with a lower viral set point and may predict disease progression in individuals with primary HIV infection. Front. Immunol. 8, 1176 (2017).
-
(2017)
Front. Immunol.
, vol.8
, pp. 1176
-
-
Ma, M.1
-
115
-
-
85030259507
-
+ memory-like NK cells contribute to the control of HIV viremia during primary infection: Optiprim-ANRS 147
-
PID: 28791125
-
+ memory-like NK cells contribute to the control of HIV viremia during primary infection: Optiprim-ANRS 147. Clin. Transl. Immunology 6, e150 (2017).
-
(2017)
Clin. Transl. Immunology
, vol.6
-
-
Gondois-Rey, F.1
-
116
-
-
84864267713
-
NKG2C deletion is a risk factor of HIV infection
-
PID: 22074011
-
Thomas, R. et al. NKG2C deletion is a risk factor of HIV infection. AIDS Res. Hum. Retroviruses 28, 844–851 (2012).
-
(2012)
AIDS Res. Hum. Retroviruses
, vol.28
, pp. 844-851
-
-
Thomas, R.1
-
117
-
-
78651492046
-
Rapid expansion and long-term persistence of elevated NK cell numbers in humans infected with hantavirus
-
PID: 21173105
-
Björkström, N. K. et al. Rapid expansion and long-term persistence of elevated NK cell numbers in humans infected with hantavirus. J. Exp. Med. 208, 13–21 (2011).
-
(2011)
J. Exp. Med.
, vol.208
, pp. 13-21
-
-
Björkström, N.K.1
-
118
-
-
80053440812
-
Unconventional repertoire profile is imprinted during acute chikungunya infection for natural killer cells polarization toward cytotoxicity
-
PID: 21966274
-
Petitdemange, C. et al. Unconventional repertoire profile is imprinted during acute chikungunya infection for natural killer cells polarization toward cytotoxicity. PLoS Pathog. 7, e1002268 (2011).
-
(2011)
PLoS Pathog.
, vol.7
-
-
Petitdemange, C.1
-
119
-
-
33745211470
-
Human cytomegalovirus infection is associated with increased proportions of NK cells that express the CD94/NKG2C receptor in aviremic HIV-1-positive patients
-
PID: 16741880
-
Gumá, M. et al. Human cytomegalovirus infection is associated with increased proportions of NK cells that express the CD94/NKG2C receptor in aviremic HIV-1-positive patients. J. Infect. Dis. 194, 38–41 (2006).
-
(2006)
J. Infect. Dis.
, vol.194
, pp. 38-41
-
-
Gumá, M.1
-
120
-
-
73649147373
-
Chronic HIV-1 viremia reverses NKG2A/NKG2C ratio on natural killer cells in patients with human cytomegalovirus co-infection
-
PID: 19910789
-
Brunetta, E. et al. Chronic HIV-1 viremia reverses NKG2A/NKG2C ratio on natural killer cells in patients with human cytomegalovirus co-infection. AIDS 24, 27–34 (2010).
-
(2010)
AIDS
, vol.24
, pp. 27-34
-
-
Brunetta, E.1
-
121
-
-
84959564450
-
A conserved HIV-1-derived peptide presented by HLA-E renders infected T-cells highly susceptible to attack by NKG2A/CD94-bearing natural killer cells
-
PID: 26828202
-
Davis, Z. B. et al. A conserved HIV-1-derived peptide presented by HLA-E renders infected T-cells highly susceptible to attack by NKG2A/CD94-bearing natural killer cells. PLoS Pathog. 12, e1005421 (2016).
-
(2016)
PLoS Pathog.
, vol.12
-
-
Davis, Z.B.1
-
122
-
-
84939652785
-
Antigen-specific NK cell memory in rhesus macaques
-
PID: 26193080
-
Reeves, R. K. et al. Antigen-specific NK cell memory in rhesus macaques. Nat. Immunol. 16, 927–932 (2015).
-
(2015)
Nat. Immunol.
, vol.16
, pp. 927-932
-
-
Reeves, R.K.1
-
123
-
-
20944443289
-
Molecular and functional characterization of NKG2D, NKp80, and NKG2C triggering NK cell receptors in rhesus and cynomolgus macaques: monitoring of NK cell function during simian HIV infection
-
PID: 15843571
-
Biassoni, R. et al. Molecular and functional characterization of NKG2D, NKp80, and NKG2C triggering NK cell receptors in rhesus and cynomolgus macaques: monitoring of NK cell function during simian HIV infection. J. Immunol. 174, 5695–5705 (2005).
-
(2005)
J. Immunol.
, vol.174
, pp. 5695-5705
-
-
Biassoni, R.1
-
124
-
-
84994805839
-
Diversification of both KIR and NKG2 natural killer cell receptor genes in macaques — implications for highly complex MHC-dependent regulation of natural killer cells
-
PID: 27565739
-
Walter, L. & Petersen, B. Diversification of both KIR and NKG2 natural killer cell receptor genes in macaques — implications for highly complex MHC-dependent regulation of natural killer cells. Immunology 150, 139–145 (2017).
-
(2017)
Immunology
, vol.150
, pp. 139-145
-
-
Walter, L.1
Petersen, B.2
-
125
-
-
73949140796
-
Immune evasion proteins of murine cytomegalovirus preferentially affect cell surface display of recently generated peptide presentation complexes
-
PID: 19906905
-
Lemmermann, N. A. W. et al. Immune evasion proteins of murine cytomegalovirus preferentially affect cell surface display of recently generated peptide presentation complexes. J. Virol. 84, 1221–1236 (2010).
-
(2010)
J. Virol.
, vol.84
, pp. 1221-1236
-
-
Lemmermann, N.A.W.1
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