CD2–CD58 interactions are pivotal for the activation and function of adaptive natural killer cells in human cytomegalovirus infection

European Journal of Immunology

Volumn 46, Issue 10, 2016, Pages 2420-2425

  Rölle, Alexander ^a   Halenius, Anne ^b   Ewen, Eva Maria ^a   Cerwenka, Adelheid ^a   Hengel, Hartmut ^b   Momburg, Frank ^a  

^a GERMAN CANCER RESEARCH CENTER DKFZ   (Germany)

^b UNIVERSITY OF FREIBURG   (Germany)

Author keywords

Adaptive NK cells; ADCC; CD2; CD57; CD58; HCMV; Human cytomegalovirus; LFA 3; NKG2C

Indexed keywords

BLOCKING ANTIBODY; CD2 ANTIGEN; CD57 ANTIGEN; GAMMA INTERFERON; HLA DR ANTIGEN; LYMPHOCYTE FUNCTION ASSOCIATED ANTIGEN 3; NATURAL KILLER CELL RECEPTOR; NATURAL KILLER CELL RECEPTOR NKG2C; TUMOR NECROSIS FACTOR; UNCLASSIFIED DRUG;

ADAPTIVE IMMUNITY; ANTIGEN BINDING; ANTIVIRAL ACTIVITY; ARTICLE; COCULTURE; CONTROLLED STUDY; CYTOKINE PRODUCTION; CYTOMEGALOVIRUS INFECTION; FETUS; FIBROBLAST; HUMAN; HUMAN CELL; NATURAL KILLER CELL; NONHUMAN; PERIPHERAL BLOOD MONONUCLEAR CELL; PRIORITY JOURNAL; PROTEIN PROTEIN INTERACTION; UPREGULATION;

EID: 85027922645     PISSN: 00142980     EISSN: 15214141     Source Type: Journal    
DOI: 10.1002/eji.201646492     Document Type: Article

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