Disordered clusters of bak dimers rupture mitochondria during apoptosis

eLife

Volumn 6, Issue , 2017, Pages

  Uren, Rachel T ^a,b   O’Hely, Martin ^a,b,c   Iyer, Sweta ^a,b   Bartolo, Ray ^a,d   Shi, Melissa X ^a,b   Brouwer, Jason M ^a,b   Alsop, Amber E ^a,b   Dewson, Grant ^a,b   Kluck, Ruth M ^a,b  

^a WALTER AND ELIZA HALL INSTITUTE OF MEDICAL RESEARCH   (Australia)

^b UNIVERSITY OF MELBOURNE   (Australia)

^c DEAKIN UNIVERSITY   (Australia)

^d ROYAL CHILDREN'S HOSPITAL   (Australia)

Author keywords

[No Author keywords available]

Indexed keywords

CYSTEINE; DIMER; ETOPOSIDE; PROTEIN BAK; PROTEIN BAX; BAK1 PROTEIN, HUMAN;

APOPTOSIS; ARTICLE; CONTROLLED STUDY; GENE CLUSTER; GENE EXPRESSION; GENE MUTATION; HUMAN; IMMUNOBLOTTING; MITOCHONDRIAL MEMBRANE POTENTIAL; MOLECULAR DOCKING; NONHUMAN; POLYACRYLAMIDE GEL ELECTROPHORESIS; PROTEIN EXPRESSION; PROTEIN STRUCTURE; RADIODENSITOMETRY; STRUCTURAL PROTEOMICS; ANIMAL; CHEMISTRY; METABOLISM; MITOCHONDRIAL MEMBRANE; MITOCHONDRION; MOUSE; PHYSIOLOGY; PROTEIN MULTIMERIZATION;

ANIMALS; APOPTOSIS; BCL-2 HOMOLOGOUS ANTAGONIST-KILLER PROTEIN; HUMANS; MICE; MITOCHONDRIA; MITOCHONDRIAL MEMBRANES; PROTEIN MULTIMERIZATION;

EID: 85012026146     PISSN: None     EISSN: 2050084X     Source Type: Journal    
DOI: 10.7554/eLife.19944     Document Type: Article

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