Bax targets mitochondria by distinct mechanisms before or during apoptotic cell death: a requirement for VDAC2 or Bak for efficient Bax apoptotic function

Cell death and differentiation

Volumn 21, Issue 12, 2014, Pages 1925-1935

  Ma, S B ^a   Nguyen, T N ^a   Tan, I ^a   Ninnis, R ^a   Iyer, S ^a   Stroud, D A ^a   Menard, M ^a   Kluck, R M ^a   Ryan, M T ^a   Dewson, G ^a  

^a UNIVERSITY OF MELBOURNE   (Australia)

Author keywords

[No Author keywords available]

Indexed keywords

BAK1 PROTEIN, MOUSE; BAX PROTEIN, MOUSE; PROTEIN BAK; PROTEIN BAX; VDAC2 PROTEIN, MOUSE; VOLTAGE DEPENDENT ANION CHANNEL 2;

129 MOUSE; ANIMAL; APOPTOSIS; C57BL MOUSE; CELL CULTURE; KNOCKOUT MOUSE; METABOLISM; MITOCHONDRION; PHYSIOLOGY; PROTEIN MULTIMERIZATION; PROTEIN TRANSPORT;

ANIMALS; APOPTOSIS; BCL-2 HOMOLOGOUS ANTAGONIST-KILLER PROTEIN; BCL-2-ASSOCIATED X PROTEIN; CELLS, CULTURED; MICE, 129 STRAIN; MICE, INBRED C57BL; MICE, KNOCKOUT; MITOCHONDRIA; PROTEIN MULTIMERIZATION; PROTEIN TRANSPORT; VOLTAGE-DEPENDENT ANION CHANNEL 2;

EID: 85027930466     PISSN: None     EISSN: 14765403     Source Type: Journal    
DOI: 10.1038/cdd.2014.119     Document Type: Article

References (0)