Stressed and Inflamed, Can GSK3 Be Blamed?

Trends in Biochemical Sciences

Volumn 42, Issue 3, 2017, Pages 180-192

  Jope, Richard S ^a   Cheng, Yuyan ^a   Lowell, Jeffrey A ^a   Worthen, Ryan J ^a   Sitbon, Yoel H ^a   Beurel, Eleonore ^a  

^a UNIVERSITY OF MIAMI MILLER SCHOOL OF MEDICINE   (United States)

Author keywords

Alzheimer's disease; depression; glycogen synthase kinase 3; inflammation; learning; stress

Indexed keywords

GLYCOGEN SYNTHASE KINASE 3; TOLL LIKE RECEPTOR 4; TRANSCRIPTION FACTOR; PROTEIN KINASE INHIBITOR;

ALZHEIMER DISEASE; BLOOD BRAIN BARRIER; CENTRAL NERVOUS SYSTEM DISEASE; COGNITIVE DEFECT; CYTOKINE PRODUCTION; CYTOKINE RELEASE; DEPRESSION; DISEASE ASSOCIATION; DISEASE PREDISPOSITION; ENZYME ACTIVATION; ENZYME ACTIVITY; ENZYME INHIBITION; HUMAN; HYPOTHALAMUS HYPOPHYSIS ADRENAL SYSTEM; INFECTION; INFLAMMATION; INJURY; LEARNING DISORDER; LONG TERM POTENTIATION; MEMORY DISORDER; MENTAL STRESS; MOOD DISORDER; NERVOUS SYSTEM INFLAMMATION; NONHUMAN; PATHOPHYSIOLOGY; PROTEIN EXPRESSION; REVIEW; SIGNAL TRANSDUCTION; ANIMAL; ANTAGONISTS AND INHIBITORS; METABOLISM;

ANIMALS; GLYCOGEN SYNTHASE KINASE 3; HUMANS; INFLAMMATION; PROTEIN KINASE INHIBITORS; STRESS, PSYCHOLOGICAL;

EID: 85008253473     PISSN: 09680004     EISSN: 13624326     Source Type: Journal    
DOI: 10.1016/j.tibs.2016.10.009     Document Type: Review

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