Hearts deficient in both Mfn1 and Mfn2 are protected against acute myocardial infarction

Cell Death and Disease

Volumn 7, Issue 5, 2016, Pages

  Hall, A R ^a   Burke, N ^a   Dongworth, R K ^a   Kalkhoran, S B ^a   Dyson, A ^a   Vicencio, J M ^a   Dorn, G W ^b   Yellon, D M ^a   Hausenloy, D J ^a,c,d  

^a UNIVERSITY COLLEGE LONDON   (United Kingdom)

^b WASHINGTON UNIVERSITY SCHOOL OF MEDICINE   (United States)

^c DUKE NUS MEDICAL SCHOOL   (Singapore)

^d DEPARTMENT OF CARDIOLOGY   (Singapore)

Author keywords

[No Author keywords available]

Indexed keywords

ANTIMYCIN A1; CALCIUM ION; CYTOCHROME C OXIDASE; MITOFUSIN 1; MITOFUSIN 2; REACTIVE OXYGEN METABOLITE; REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE DEHYDROGENASE (UBIQUINONE); SUCCINATE DEHYDROGENASE (UBIQUINONE); UBIQUINOL CYTOCHROME C REDUCTASE; CALCIUM; CARRIER PROTEIN; GUANOSINE TRIPHOSPHATASE; MFN1 PROTEIN, MOUSE; MFN2 PROTEIN, MOUSE; MITOCHONDRIAL PERMEABILITY TRANSITION PORE;

ACUTE HEART INFARCTION; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; CELL ORGANELLE; CONFOCAL MICROSCOPY; CONTROLLED STUDY; DISORDERS OF MITOCHONDRIAL FUNCTIONS; ECHOCARDIOGRAPHY; FRACTIONAL SHORTENING; HEART INFARCTION SIZE; HEART LEFT VENTRICLE HYPERTROPHY; HEART MITOCHONDRION; HEART OUTPUT; HEART PROTECTION; MITOCHONDRION SWELLING; MYOCARDIAL ISCHEMIA REPERFUSION INJURY; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; ANIMAL; CARDIAC MUSCLE; CARDIAC MUSCLE CELL; DEFICIENCY; DRUG EFFECTS; GENE EXPRESSION; GENETICS; KNOCKOUT MOUSE; METABOLISM; MITOCHONDRIAL DYNAMICS; MITOCHONDRION; MOUSE; MYOCARDIAL INFARCTION; MYOCARDIAL REPERFUSION INJURY; PATHOLOGY; SARCOPLASMIC RETICULUM; ULTRASTRUCTURE;

ANIMALS; ANTIMYCIN A; CALCIUM; GENE EXPRESSION; GTP PHOSPHOHYDROLASES; MICE; MICE, KNOCKOUT; MITOCHONDRIA; MITOCHONDRIAL DYNAMICS; MITOCHONDRIAL MEMBRANE TRANSPORT PROTEINS; MYOCARDIAL INFARCTION; MYOCARDIAL REPERFUSION INJURY; MYOCARDIUM; MYOCYTES, CARDIAC; REACTIVE OXYGEN SPECIES; SARCOPLASMIC RETICULUM;

EID: 84995901715     PISSN: None     EISSN: 20414889     Source Type: Journal    
DOI: 10.1038/cddis.2016.139     Document Type: Article

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