Correction to: p53 induces formation of NEAT1 lncRNA-containing paraspeckles that modulate replication stress response and chemosensitivity (Nature Medicine, (2016), 22, 8, (861-868), 10.1038/nm.4135);P53 induces formation of NEAT1 lncRNA-containing paraspeckles that modulate replication stress response and chemosensitivity

Nature Medicine

Volumn 22, Issue 8, 2016, Pages 861-868

  Adriaens, Carmen ^a   Standaert, Laura ^a   Barra, Jasmine ^a   Latil, Mathilde ^b   Verfaillie, Annelien ^c   Kalev, Peter ^c   Boeckx, Bram ^a,c   Wijnhoven, Paul W G ^d   Radaelli, Enrico ^c   Vermi, William ^e,f   Leucci, Eleonora ^a   Lapouge, Gaëlle ^b   Beck, Benjamin ^b   Van Den Oord, Joost ^g   Nakagawa, Shinichi ^h,i   Hirose, Tetsuro ⁱ   Sablina, Anna A ^c   Lambrechts, Diether ^a,c   Aerts, Stein ^c   Blanpain, Cédric ^b   Marine, Jean Christophe ^a   more..

^a UNIVERSITY OF LEUVEN   (Belgium)

^b UNIVERSITÉ LIBRE DE BRUXELLES   (Belgium)

^c UNIVERSITY OF LEUVEN   (Belgium)

^d UNIVERSITY OF CAMBRIDGE   (United Kingdom)

^e UNIVERSITY OF BRESCIA   (Italy)

^f WASHINGTON UNIVERSITY SCHOOL OF MEDICINE   (United States)

^g UNIVERSITY HOSPITALS LEUVEN   (Belgium)

^h RIKEN   (Japan)

ⁱ HOKKAIDO UNIVERSITY   (Japan)

Author keywords

[No Author keywords available]

Indexed keywords

ATR PROTEIN; CHECKPOINT KINASE 1; CYCLIN DEPENDENT KINASE INHIBITOR 1A; CYTOKERATIN 14; HISTONE H2AX; LONG UNTRANSLATED RNA; NUCLEAR PARASPECKLE ASSEMBLY TRANSCRIPT 1 PROTEIN; NUTLIN 3; PROTEIN P53; REACTIVE OXYGEN METABOLITE; UNCLASSIFIED DRUG; ATM PROTEIN; ATR PROTEIN, MOUSE; NEAT1 LONG NON-CODING RNA, HUMAN; NEAT1 LONG NON-CODING RNA, MOUSE; NICOTINAMIDE ADENINE DINUCLEOTIDE ADENOSINE DIPHOSPHATE RIBOSYLTRANSFERASE INHIBITOR;

ADULT; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CELL DEATH; CELL DIFFERENTIATION; CELL NUCLEUS INCLUSION BODY; CELL PROLIFERATION; CELL STRESS; CELL VIABILITY; CHEMOSENSITIVITY; CONTROLLED STUDY; DNA DAMAGE; DOUBLE STRANDED DNA BREAK; EMBRYO; GENE TARGETING; GENOTOXICITY; HISTONE PHOSPHORYLATION; HUMAN; HUMAN CELL; HUMAN TISSUE; IN VIVO STUDY; MOUSE; NEGATIVE FEEDBACK; NONHUMAN; PRIORITY JOURNAL; PROTEIN MOTIF; SIGNAL TRANSDUCTION; SKIN CANCER; SKIN CARCINOGENESIS; TRANSCRIPTION INITIATION SITE; ANIMAL; CARCINOGENESIS; FLOW CYTOMETRY; FLUORESCENCE IN SITU HYBRIDIZATION; GENE EXPRESSION REGULATION; GENETICS; IMMUNOBLOTTING; IMMUNOHISTOCHEMISTRY; KAPLAN MEIER METHOD; MCF-7 CELL LINE; PRECANCER; PROPORTIONAL HAZARDS MODEL; REAL TIME POLYMERASE CHAIN REACTION; REVERSE TRANSCRIPTION POLYMERASE CHAIN REACTION; SKIN TUMOR; SURVIVAL ANALYSIS; TUMOR CELL LINE;

ANIMALS; ATAXIA TELANGIECTASIA MUTATED PROTEINS; CARCINOGENESIS; CELL LINE, TUMOR; DNA DAMAGE; FLOW CYTOMETRY; GENE EXPRESSION REGULATION, NEOPLASTIC; HUMANS; IMMUNOBLOTTING; IMMUNOHISTOCHEMISTRY; IN SITU HYBRIDIZATION, FLUORESCENCE; KAPLAN-MEIER ESTIMATE; MCF-7 CELLS; MICE; POLY(ADP-RIBOSE) POLYMERASE INHIBITORS; PRECANCEROUS CONDITIONS; PROPORTIONAL HAZARDS MODELS; REAL-TIME POLYMERASE CHAIN REACTION; REVERSE TRANSCRIPTASE POLYMERASE CHAIN REACTION; RNA, LONG NONCODING; SIGNAL TRANSDUCTION; SKIN NEOPLASMS; SURVIVAL ANALYSIS; TUMOR SUPPRESSOR PROTEIN P53;

EID: 84976902062     PISSN: 10788956     EISSN: 1546170X     Source Type: Journal    
DOI: 10.1038/s41591-024-02842-w     Document Type: Erratum

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