Hypoxia primes human normal prostate epithelial cells and cancer cell lines for the NLRP3 and AIM2 inflammasome activation

Oncotarget

Volumn 7, Issue 19, 2016, Pages 28183-28194

  Panchanathan, Ravichandran ^a,b   Liu, Hongzhu ^a,b   Choubey, Divaker ^a,b  

^a VETERANS AFFAIRS MEDICAL CENTER   (United States)

^b UNIVERSITY OF CINCINNATI COLLEGE OF MEDICINE   (United States)

Author keywords

Cancer; Hypoxia; Inflammasome; Inflammation; Prostate

Indexed keywords

AIM2 PROTEIN; AIM2 RECEPTOR; CELL RECEPTOR; CRYOPYRIN; INFLAMMASOME; INTERLEUKIN 1BETA; UNCLASSIFIED DRUG; AIM2 PROTEIN, HUMAN; DNA BINDING PROTEIN; NLRP3 PROTEIN, HUMAN;

ARTICLE; CELL HYPOXIA; CELL LINE; CHRONIC INFLAMMATION; CONTROLLED STUDY; HUMAN; HUMAN CELL; HYPOXIA; MALE; MOLECULAR DYNAMICS; PROSTATE CANCER CELL LINE; PROSTATE EPITHELIUM CELL; PROSTATIC CHRONIC INFLAMMATION; PROTEIN DETERMINATION; PROTEIN FUNCTION; THP 1 MACROPHAGE CELL LINE; COMPLICATION; EPITHELIUM CELL; METABOLISM; PATHOLOGY; PHYSIOLOGY; PROSTATE; PROSTATE TUMOR; PROSTATITIS; TUMOR CELL LINE;

CELL HYPOXIA; CELL LINE, TUMOR; DNA-BINDING PROTEINS; EPITHELIAL CELLS; HUMANS; INFLAMMASOMES; MALE; NLR FAMILY, PYRIN DOMAIN-CONTAINING 3 PROTEIN; PROSTATE; PROSTATIC NEOPLASMS; PROSTATITIS;

EID: 84968763900     PISSN: None     EISSN: 19492553     Source Type: Journal    
DOI: 10.18632/oncotarget.8594     Document Type: Article

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