Receptor for advanced glycation end products and its inflammatory ligands are upregulated in amyotrophic lateral sclerosis

Frontiers in Cellular Neuroscience

Volumn 9, Issue DEC, 2015, Pages

  Juranek, Judyta K ^a,b   Daffu, Gurdip K ^b   Wojtkiewicz, Joanna ^c   Lacomis, David ^d   Kofler, Julia ^d   Schmidt, Ann Marie ^a,b  

^a NewYork Presbyterian Hospital   (United States)

^b NEW YORK UNIVERSITY SCHOOL OF MEDICINE   (United States)

^c UNIVERSITY OF WARMIA AND MAZURY IN OLSZTYN   (Poland)

^d UNIVERSITY OF PITTSBURGH SCHOOL OF MEDICINE   (United States)

Author keywords

Amyotrophic lateral sclerosis; CML; HMGB1; RAGE; S100B; Spinal cord

Indexed keywords

6 N CARBOXYMETHYLLYSINE; ADVANCED GLYCATION END PRODUCT RECEPTOR; HIGH MOBILITY GROUP B1 PROTEIN; PROTEIN S100B;

ADULT; AMYOTROPHIC LATERAL SCLEROSIS; ARTICLE; CONTROLLED STUDY; FEMALE; HUMAN; HUMAN TISSUE; IMMUNOBLOTTING; IMMUNOHISTOCHEMISTRY; IMMUNOREACTIVITY; MALE; MIDDLE AGED; PROTEIN EXPRESSION; REVERSE TRANSCRIPTION POLYMERASE CHAIN REACTION; RNA ISOLATION; UPREGULATION; WESTERN BLOTTING;

EID: 84954039309     PISSN: 16625102     EISSN: None     Source Type: Journal    
DOI: 10.3389/fncel.2015.00485     Document Type: Article

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