RPS23RG1 reduces Aβ oligomer-induced synaptic and cognitive deficits

Scientific Reports

Volumn 6, Issue , 2016, Pages

  Yan, Li ^a,b   Chen, Yaomin ^b   Li, Wubo ^c   Huang, Xiumei ^b   Badie, Hedieh ^b   Jian, Fan ^b   Huang, Timothy ^b   Zhao, Yingjun ^b   Cohen, Stanley N ^d   Li, Limin ^c   Zhang, Yun Wu ^b,e   Luo, Huanmin ^a   Tu, Shichun ^b   Xu, Huaxi ^b  

^a MEDICAL COLLEGE OF JINAN UNIVERSITY   (China)

^b BURNHAM INSTITUTE   (United States)

^c Functional Genetics Inc   (United States)

^d STANFORD UNIVERSITY SCHOOL OF MEDICINE   (United States)

^e XIAMEN UNIVERSITY   (China)

Author keywords

[No Author keywords available]

Indexed keywords

ADENYLATE CYCLASE; ADENYLYL CYCLASE 8; AMYLOID BETA PROTEIN; CYCLIC AMP DEPENDENT PROTEIN KINASE; MESSENGER RNA; PROTEIN AGGREGATE; PROTEIN BINDING; RIBOSOME PROTEIN; RPS23 PROTEIN, HUMAN; RPS23R1 PROTEIN, MOUSE; TAU PROTEIN;

ALZHEIMER DISEASE; ANIMAL; CHEMISTRY; COGNITIVE DEFECT; DISEASE MODEL; GENE EXPRESSION REGULATION; GENE SILENCING; GENETICS; HIPPOCAMPAL CA1 REGION; HUMAN; LONG TERM POTENTIATION; METABOLISM; MOLECULAR CLONING; MOUSE; NERVE CELL; NERVE CELL PLASTICITY; NUCLEOTIDE SEQUENCE; PATHOLOGY; PATHOPHYSIOLOGY; PHOSPHORYLATION; PROTEINOSIS; SYNAPSE; TRANSGENIC MOUSE;

ADENYLYL CYCLASES; ALZHEIMER DISEASE; AMYLOID BETA-PEPTIDES; ANIMALS; BASE SEQUENCE; CA1 REGION, HIPPOCAMPAL; CLONING, MOLECULAR; COGNITIVE DYSFUNCTION; CYCLIC AMP-DEPENDENT PROTEIN KINASES; DISEASE MODELS, ANIMAL; GENE EXPRESSION REGULATION; GENE KNOCKDOWN TECHNIQUES; HUMANS; LONG-TERM POTENTIATION; MICE; MICE, TRANSGENIC; NEURONAL PLASTICITY; NEURONS; PHOSPHORYLATION; PROTEIN AGGREGATES; PROTEIN AGGREGATION, PATHOLOGICAL; PROTEIN BINDING; RIBOSOMAL PROTEINS; RNA, MESSENGER; SYNAPSES; TAU PROTEINS;

EID: 84953297110     PISSN: None     EISSN: 20452322     Source Type: Journal    
DOI: 10.1038/srep18668     Document Type: Article

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