Suppression of basal and carbon nanotube-induced oxidative stress, inflammation and fibrosis in mouse lungs by Nrf2

Nanotoxicology

Volumn 10, Issue 6, 2016, Pages 699-709

  Dong, Jie ^a   Ma, Qiang ^a  

^a NATIONAL INSTITUTE FOR OCCUPATIONAL SAFETY AND HEALTH   (United States)

Author keywords

Lung fibrosis; Lung inflammation; Multi walled carbon nanotubes; Nanotoxicity; Nuclear factor erythroid 2 related factor 2; Oxidative stress

Indexed keywords

CALVASCULIN; CD63 ANTIGEN; COLLAGEN TYPE 1; FAS LIGAND; FIBRONECTIN; HEME OXYGENASE 1; HISTONE H2AX; INTERLEUKIN 10; INTERLEUKIN 13; INTERLEUKIN 1BETA; INTERLEUKIN 5; MACROPHAGE INFLAMMATORY PROTEIN 1ALPHA; MATRIX METALLOPROTEINASE 14; MESSENGER RNA; MONOCYTE CHEMOTACTIC PROTEIN 5; MULTI WALLED NANOTUBE; REACTIVE OXYGEN METABOLITE; TISSUE INHIBITOR OF METALLOPROTEINASE 1; TISSUE INHIBITOR OF METALLOPROTEINASE 4; TRANSCRIPTION FACTOR NRF2; CARBON NANOTUBE; CYTOKINE; NFE2L2 PROTEIN, MOUSE;

ADULT; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; B LYMPHOCYTE; CD3+ T LYMPHOCYTE; CELL NUCLEUS; CELL PROLIFERATION; COLLAGEN FIBER; CONTROLLED STUDY; DOSE RESPONSE; GENE EXPRESSION; GRANULOCYTE; IN VIVO STUDY; LIPID PEROXIDATION; LUNG ALVEOLUS MACROPHAGE; LUNG FIBROSIS; LUNG INFILTRATE; LUNG LAVAGE; LUNG TOXICITY; LYMPHOCYTIC INFILTRATION; MALE; MOLECULAR WEIGHT; MOUSE; NONHUMAN; OXIDATIVE STRESS; PNEUMONIA; PRIORITY JOURNAL; UBIQUITINATION; UPREGULATION; YOUNG ADULT; ANIMAL; C57BL MOUSE; CHEMICALLY INDUCED; DRUG EFFECTS; GENETICS; IMMUNOLOGY; KNOCKOUT MOUSE; LUNG; MACROPHAGE; METABOLISM; PATHOLOGY; PHYSIOLOGY;

ANIMALS; CYTOKINES; DOSE-RESPONSE RELATIONSHIP, DRUG; LUNG; MACROPHAGES; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; NANOTUBES, CARBON; NF-E2-RELATED FACTOR 2; OXIDATIVE STRESS; PNEUMONIA; PULMONARY FIBROSIS;

EID: 84947931461     PISSN: 17435390     EISSN: 17435404     Source Type: Journal    
DOI: 10.3109/17435390.2015.1110758     Document Type: Article

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