-
1
-
-
0028915702
-
The effects of phosphorylation of cardiac troponin-I on its interactions with actin and troponin-C
-
Al-Hillawi, E., Bhandari, D.G., Trayer, H.R., and Trayer, I.P. (1995). The effects of phosphorylation of cardiac troponin-I on its interactions with actin and troponin-C. EurJ Biochem 228, 962-970.
-
(1995)
EurJ Biochem
, vol.228
, pp. 962-970
-
-
Al-Hillawi, E.1
Bhandari, D.G.2
Trayer, H.R.3
Trayer, I.P.4
-
2
-
-
0032530637
-
Phosphorylation-specific antibodies for human cardiac troponin-I Eur
-
Al-Hillawi, E., Chilton, D., Trayer, I.P., and Cummins, P. (1998). Phosphorylation-specific antibodies for human cardiac troponin-I Eur JBiochem 256, 535-540.
-
(1998)
J Biochem
, vol.256
, pp. 535-540
-
-
Al-Hillawi, E.1
Chilton, D.2
Trayer, I.P.3
Cummins, P.4
-
3
-
-
84903591114
-
Desensitization of Myofilaments to Ca2+ as a Therapeutic Target for Hypertrophic Cardiomyopathy with Mutations in Thin Filament Proteins
-
doi: 10.1161/CIRCGENETICS.113.000324.
-
Alves, M.L., Dias, F.A.L, Gaffin, R.D., Simon, J.N., Montminy, E.M., Biesiadecki, B.J., Hinken, A.C., Warren, CM., Utter, M.S., Davis, R.T., Sakthivel, S., Robbins, J., Wieczorek, D.F., Solaro, R.J., and Wolska, B.M. (2014). Desensitization of Myofilaments to Ca2+ as a Therapeutic Target for Hypertrophic Cardiomyopathy with Mutations in Thin Filament Proteins. Circ: Cardiovasc Gen. doi: 10.1161/CIRCGENETICS.113.000324.
-
(2014)
Circ: Cardiovasc Gen
-
-
Alves, M.L.1
Dias, F.A.L.2
Gaffin, R.D.3
Simon, J.N.4
Montminy, E.M.5
Biesiadecki, B.J.6
Hinken, A.C.7
Warren, C.M.8
Utter, M.S.9
Davis, R.T.10
Sakthivel, S.11
Robbins, J.12
Wieczorek, D.F.13
Solaro, R.J.14
Wolska, B.M.15
-
4
-
-
0031923552
-
Microanalysis and distribution of cardiac troponin I phosphospecies in heart areas
-
Ardelt, P., Dorka, P., Jaquet, K., Heilmeyer, L.M.G., Kortke, H., Korfer, R., and Notohamiprodjo, G. (1998). Microanalysis and distribution of cardiac troponin I phosphospecies in heart areas. Biol Chem 379, 341-347.
-
(1998)
Biol Chem
, vol.379
, pp. 341-347
-
-
Ardelt, P.1
Dorka, P.2
Jaquet, K.3
Heilmeyer, L.M.G.4
Kortke, H.5
Korfer, R.6
Notohamiprodjo, G.7
-
5
-
-
69249106065
-
In Vivo Phosphorylation Site Mapping in Mouse Cardiac Troponin I by High Resolution Top-Down Electron Capture Dissociation Mass Spectrometry: Ser22/23 Are the Only Sites Basally Phosphorylated
-
Ayaz-Guner, S., Zhang, J., Li, L, Walker, J.W., and Ge, Y. (2009). In Vivo Phosphorylation Site Mapping in Mouse Cardiac Troponin I by High Resolution Top-Down Electron Capture Dissociation Mass Spectrometry: Ser22/23 Are the Only Sites Basally Phosphorylated. Biochemistry 48, 8161-8170.
-
(2009)
Biochemistry
, vol.48
, pp. 8161-8170
-
-
Ayaz-Guner, S.1
Zhang, J.2
Li, L.3
Walker, J.W.4
Ge, Y.5
-
6
-
-
0018341935
-
Phosphorylation of a bovine cardiac actin complex
-
Bailin, G. (1979). Phosphorylation of a bovine cardiac actin complex. Am J Physiol 236, C41-46.
-
(1979)
Am J Physiol
, vol.236
-
-
Bailin, G.1
-
7
-
-
37349117667
-
Modulation of cardiac troponin C function by the cardiac-specific N-terminus of troponin I: influence of PKA phosphorylation and involvement in cardiomyopathies
-
doi: 10.1016/j.jmb.2007.10.062.
-
Baryshnikova, O.K., Li, M.X., and Sykes, B.D. (2008). Modulation of cardiac troponin C function by the cardiac-specific N-terminus of troponin I: influence of PKA phosphorylation and involvement in cardiomyopathies./Mo/£/o/375, 735-751. doi: 10.1016/j.jmb.2007.10.062.
-
(2008)
J Mol Biol
, pp. 735-751
-
-
Baryshnikova, O.K.1
Li, M.X.2
Sykes, B.D.3
-
8
-
-
84906552537
-
*Functional investigation of troponin with the homozygous HCM mutation, TNNT2 K280M, obtained from an explanted heart
-
Bayliss, C, Messer, A., Leung, M.-C, Ward, D., Van Der Velden, J., Poggesi, C, Redwood, C, and Marston, S. (2012a). *Functional investigation of troponin with the homozygous HCM mutation, TNNT2 K280M, obtained from an explanted heart. Cardiovasc Res 93, S107.
-
(2012)
Cardiovasc Res
, vol.93
-
-
Bayliss, C.1
Messer, A.2
Leung M.-C Ward, D.3
Van Der Velden, J.4
Poggesi, C.5
Redwood, C.6
Marston, S.7
-
9
-
-
84874257414
-
Myofibrillar Ca2+-Sensitivity Is Uncoupled From Troponin I Phosphorylation In Hypertrophic Obstructive Cardiomyopathy Due To Abnormal Troponin T
-
doi: 10.1093/cvr/cvs322
-
Bayliss, C.R., Jacques, A.M., Leung, M.-C, Ward, D.G., Redwood, C.S., Gallon, C.E., Copeland, O.N., Mckenna, W.J., Dos Remedios, C, Marston, S.B., and Messer, A.E. (2012b). Myofibrillar Ca2+-Sensitivity Is Uncoupled From Troponin I Phosphorylation In Hypertrophic Obstructive Cardiomyopathy Due To Abnormal Troponin T. Cardiovasc Res 97, 500-508. doi: 10.1093/cvr/cvs322.
-
(2012)
Cardiovasc Res
, vol.97
, pp. 500-508
-
-
Bayliss, C.R.1
Jacques, A.M.2
Leung, M.-C.3
Ward, D.G.4
Redwood, C.S.5
Gallon, C.E.6
Copeland, O.N.7
Mckenna, W.J.8
Dos Remedios, C.9
Marston, S.B.10
Messer, A.E.11
-
10
-
-
34249676905
-
The troponin C G159D mutation blunts myofilament desensitization induced by troponin I Ser23/24 phosphorylation
-
Biesiadecki, B.J., Kobayashi, T., Walker, J.S., John Solaro, R., and De Tombe, P.P. (2007). The troponin C G159D mutation blunts myofilament desensitization induced by troponin I Ser23/24 phosphorylation. Circ Res 100,1486-1493.
-
(2007)
Circ Res
, vol.100
, pp. 1486-1493
-
-
Biesiadecki, B.J.1
Kobayashi, T.2
Walker, J.S.3
John Solaro, R.4
De Tombe, P.P.5
-
11
-
-
0030773407
-
Troponin I phosphorylation in the normal and failing adult human heart
-
Bodor, G.S., Oakeley, A.E., Allen, P.D., Crimmins, D.L., Ladenson, J.H., and Anderson, P.A.W. (1997). Troponin I phosphorylation in the normal and failing adult human heart. Circulation 96,1495-1500.
-
(1997)
Circulation
, vol.96
, pp. 1495-1500
-
-
Bodor, G.S.1
Oakeley, A.E.2
Allen, P.D.3
Crimmins, D.L.4
Ladenson, J.H.5
Anderson, P.A.W.6
-
12
-
-
0023834450
-
Physiologic mechanisms governing hemodynamic responses to positive inotropic therapy in patients with dilated cardiomyopathy
-
Borow, K.M., Lang, R.M., Neumann, A., Carroll, J.D., and Rajfer, S.I. (1988). Physiologic mechanisms governing hemodynamic responses to positive inotropic therapy in patients with dilated cardiomyopathy. Circulation 77, 625-637.
-
(1988)
Circulation
, vol.77
, pp. 625-637
-
-
Borow, K.M.1
Lang, R.M.2
Neumann, A.3
Carroll, J.D.4
Rajfer, S.I.5
-
13
-
-
84880852474
-
A computational exploration of the interactions of the green tea polyphenol (-)- epigallocatechin 3-gallate with cardiac muscle troponin C
-
doi: 10.1371/journal.pone.0070556
-
Botten, D., Fugallo, G., Fraternali, F., and Molteni, C. (2013). A computational exploration of the interactions of the green tea polyphenol (-)- epigallocatechin 3-gallate with cardiac muscle troponin C. PLoS ONE 8, e70556. doi: 10.1371/journal.pone.0070556.
-
(2013)
PLoS ONE
, vol.8
-
-
Botten, D.1
Fugallo, G.2
Fraternali, F.3
Molteni, C.4
-
14
-
-
70349250125
-
Identification and functional characterization of cardiac troponin I as a novel disease gene in autosomal dominant dilated cardiomyopathy
-
Carballo, S., Robinson, P., Otway, R., Fatkin, D., Jongbloed, J.D., De Jonge, N., Blair, E., Van Tintelen, J.P., Redwood, C., and Watkins, H. (2009). Identification and functional characterization of cardiac troponin I as a novel disease gene in autosomal dominant dilated cardiomyopathy. Circ Res 105, 375-382.
-
(2009)
Circ Res
, vol.105
, pp. 375-382
-
-
Carballo, S.1
Robinson, P.2
Otway, R.3
Fatkin, D.4
Jongbloed, J.D.5
De Jonge, N.6
Blair, E.7
Van Tintelen, J.P.8
Redwood, C.9
Watkins, H.10
-
15
-
-
3142570567
-
Asymmetric septal hypertrophy in heterozygous cMyBPC null mice
-
Carrier, L, Knoll, R., Vignier, N., Keller, D.I., Bausero, P., Prudhon, B., Isnard, R., Ambroisine, M.L., Fiszman, M., Ross, J., Jr., Schwartz, K., and Chien, K.R. (2004). Asymmetric septal hypertrophy in heterozygous cMyBPC null mice. Cardiovasc Res 63, 293-304.
-
(2004)
Cardiovasc Res
, vol.63
, pp. 293-304
-
-
Carrier, L.1
Knoll, R.2
Vignier, N.3
Keller, D.I.4
Bausero, P.5
Prudhon, B.6
Isnard, R.7
Ambroisine, M.L.8
Fiszman, M.9
Ross Jr., J.10
Schwartz, K.11
Chien, K.R.12
-
16
-
-
17644394261
-
Targeting protein phosphatase 1 in heart failure
-
Champion, H.C. (2005). Targeting protein phosphatase 1 in heart failure. Circ Res 96, 708-710.
-
(2005)
Circ Res
, vol.96
, pp. 708-710
-
-
Champion, H.C.1
-
17
-
-
30944435503
-
Sarcomeric protein mutations in dilated cardiomyopathy
-
Chang, A.N., and Potter, J.D. (2005). Sarcomeric protein mutations in dilated cardiomyopathy. Heart Fail Rev 10, 225-235.
-
(2005)
Heart Fail Rev
, vol.10
, pp. 225-235
-
-
Chang, A.N.1
Potter, J.D.2
-
18
-
-
77957555832
-
Investigation of changes in skeletal muscle alpha-actin expression in normal and pathological human and mouse hearts
-
doi: 10.1007/sl0974-010-9224-7
-
Copeland, 0., Nowak, K., Laing, N., Ravenscroft, G., Messer, A.E., Bayliss, C.R., and Marston, S.B. (2010a). Investigation of changes in skeletal muscle alpha-actin expression in normal and pathological human and mouse hearts. J Muse Res Cell Motil 31, 207-214. doi: 10.1007/sl0974-010-9224-7.
-
(2010)
J Muse Res Cell Motil
, vol.31
, pp. 207-214
-
-
Copeland, 0.1
Nowak, K.2
Laing, N.3
Ravenscroft, G.4
Messer, A.E.5
Bayliss, C.R.6
Marston, S.B.7
-
19
-
-
78149416262
-
Analysis of cardiac myosin binding protein-C phosphorylation in human heart muscle.
-
doi: 10.1016/j.yjmcc.2010.09.007.
-
Copeland, 0., Sadayappan, S., Messer, A.E., Stienen, G.J., Velden, J., and Marston, S.B. (2010b). Analysis of cardiac myosin binding protein-C phosphorylation in human heart muscle. J Mol Cell Cctrdiol 49,1003-1011. doi: 10.1016/j.yjmcc.2010.09.007.
-
(2010)
J Mol Cell Cctrdiol
, vol.49
, pp. 1003-1011
-
-
Copeland, 0.1
Sadayappan, S.2
Messer, A.E.3
Stienen, G.J.4
Velden, J.5
Marston, S.B.6
-
20
-
-
0142257957
-
Phosphorylation of human cardiac troponin I G203S and K206Q linked to familial hypertrophic cardiomyopathy affects actomyosin interaction in different ways
-
Deng, Y., Schmidtmann, A., Kruse, S., Filatov, V., Heilmeyer, L.M., Jr., Jaquet, K., and Thieleczek, R. (2003). Phosphorylation of human cardiac troponin I G203S and K206Q linked to familial hypertrophic cardiomyopathy affects actomyosin interaction in different ways. J Mol Cell Cardiol 35, 1365-1374.
-
(2003)
J Mol Cell Cardiol
, vol.35
, pp. 1365-1374
-
-
Deng, Y.1
Schmidtmann, A.2
Kruse, S.3
Filatov, V.4
Heilmeyer Jr., L.M.5
Jaquet, K.6
Thieleczek, R.7
-
21
-
-
0035807934
-
Effects of Phosphorylation and Mutation R145G on Human Cardiac Troponin I Function
-
Deng, Y., Schmidtmann, A., Redlich, A., Westerdorf, B., Jaquet, K., and Thieleczek, R. (2001). Effects of Phosphorylation and Mutation R145G on Human Cardiac Troponin I Function. Biochemistry 40,14593-14602.
-
(2001)
Biochemistry
, vol.40
, pp. 14593-14602
-
-
Deng, Y.1
Schmidtmann, A.2
Redlich, A.3
Westerdorf, B.4
Jaquet, K.5
Thieleczek, R.6
-
22
-
-
0029561016
-
Reconstitution of skinned cardiac fibres with human recombinant cardiac troponin-I mutants and troponin-C
-
Dohet, C, Al-Hillawi, E., Trayer, I.P., and Ruegg, J.C. (1995). Reconstitution of skinned cardiac fibres with human recombinant cardiac troponin-I mutants and troponin-C. FEBS Lett 377,131-134.
-
(1995)
FEBS Lett
, vol.377
, pp. 131-134
-
-
Dohet, C.1
Al-Hillawi, E.2
Trayer, I.P.3
Ruegg, J.C.4
-
23
-
-
41249091458
-
Structural kinetics of Cardiac troponin C mutants linked to familial hypertrophic and dilated cardiomyopathy in troponin complexes
-
Dong, W., Xing, J., Ouyang, Y., An, J., and Cheung, H.C. (2008). Structural kinetics of Cardiac troponin C mutants linked to familial hypertrophic and dilated cardiomyopathy in troponin complexes. J Biol Chem 283, 3424-3432.
-
(2008)
J Biol Chem
, vol.283
, pp. 3424-3432
-
-
Dong, W.1
Xing, J.2
Ouyang, Y.3
An, J.4
Cheung, H.C.5
-
24
-
-
34548211785
-
Effects of PKA phosphorylation of cardiac troponin I and strong crossbridge on conformational transitions of the N-domain of cardiac troponin C in regulated thin filaments
-
Dong, W.J., Jayasundar, J.J., An, J., Xing, J., and Cheung, H.C. (2007). Effects of PKA phosphorylation of cardiac troponin I and strong crossbridge on conformational transitions of the N-domain of cardiac troponin C in regulated thin filaments. Biochemistry 46, 9752-9761.
-
(2007)
Biochemistry
, vol.46
, pp. 9752-9761
-
-
Dong, W.J.1
Jayasundar, J.J.2
An, J.3
Xing, J.4
Cheung, H.C.5
-
25
-
-
34547610522
-
Knock-In Mouse Model of Dilated Cardiomyopathy Caused by Troponin Mutation
-
Du, C.K., Morimoto, S., Nishii, K., Minakami, R., Ohta, M., Tadano, N., Lu, Q.W., Wang, Y.Y., Zhan, D.Y., Mochizuki, M., Kita, S., Miwa, Y., Takahashi- Yanaga, F., Iwamoto, T., Ohtsuki, I., and Sasaguri, T. (2007). Knock-In Mouse Model of Dilated Cardiomyopathy Caused by Troponin Mutation. CircRes 101,185-194.
-
(2007)
CircRes
, vol.101
, pp. 185-194
-
-
Du, C.K.1
Morimoto, S.2
Nishii, K.3
Minakami, R.4
Ohta, M.5
Tadano, N.6
Lu, Q.W.7
Wang, Y.Y.8
Zhan, D.Y.9
Mochizuki, M.10
Kita, S.11
Miwa, Y.12
Takahashi-Yanaga, F.13
Iwamoto, T.14
Ohtsuki, I.15
Sasaguri, T.16
-
26
-
-
0026756820
-
Betaadrenergic contractile reserve as a predictor of clinical outcome in patients with idiopathic dilated cardiomyopathy
-
Dubois-Rande, J.L., Merlet, P., Roudot, F., Benvenuti, C, Adnot, S., Hittinger, L, Duval, A.M., Syrota, A., Castaigne, A., and Loisance, D. (1992). Betaadrenergic contractile reserve as a predictor of clinical outcome in patients with idiopathic dilated cardiomyopathy. Am Heart J 124, 679-685.
-
(1992)
Am Heart J
, vol.124
, pp. 679-685
-
-
Dubois-Rande, J.L.1
Merlet, P.2
Roudot, F.3
Benvenuti, C.4
Adnot, S.5
Hittinger, L.6
Duval, A.M.7
Syrota, A.8
Castaigne, A.9
Loisance, D.10
-
27
-
-
70350448975
-
Functional Analysis of a Unique Troponin C Mutation, Glyl59Asp that Causes Familial Dilated Cardiomyopathy, Studied in Explanted Heart Muscle
-
doi: 10.1161/CIRCHEARTFAILURE.108.818237
-
Dyer, E., Jacques, A., Hoskins, A., Ward, D., Gallon, C, Messer, A., Kaski, J., Burch, M., Kentish, J., and Marston, S. (2009). Functional Analysis of a Unique Troponin C Mutation, Glyl59Asp that Causes Familial Dilated Cardiomyopathy, Studied in Explanted Heart Muscle. Circ: Heart Fail 2, 456-464. doi: 10.1161/CIRCHEARTFAILURE.108.818237.
-
(2009)
Circ: Heart Fail
, vol.2
, pp. 456-464
-
-
Dyer, E.1
Jacques, A.2
Hoskins, A.3
Ward, D.4
Gallon, C.5
Messer, A.6
Kaski, J.7
Burch, M.8
Kentish, J.9
Marston, S.10
-
28
-
-
84906547079
-
*In vitro motility studies of HCM and DCM mutations in cardiac muscle actin
-
Dyer, E., Wells, D., Redwood, C, and Marston, S.B. (2007). *In vitro motility studies of HCM and DCM mutations in cardiac muscle actin. Biophys J 92,481A.
-
(2007)
BiophysJ
, vol.92
-
-
Dyer, E.1
Wells, D.2
Redwood, C.3
Marston, S.B.4
-
29
-
-
34447526253
-
Decreased phosphorylation levels of cardiac myosin-binding protein-C in human and experimental heart failure
-
El-Armouche, A., Pohlmann, L, Schlossarek, S., Starbatty, J., Yeh, Y.H., Nattel, S., Dobrev, D., Eschenhagen, T., and Carrier, L. (2007). Decreased phosphorylation levels of cardiac myosin-binding protein-C in human and experimental heart failure. J Mol Cell Cardiol 43, 223-229.
-
(2007)
J Mol Cell Cardiol
, vol.43
, pp. 223-229
-
-
El-Armouche, A.1
Pohlmann, L.2
Schlossarek, S.3
Starbatty, J.4
Yeh, Y.H.5
Nattel, S.6
Dobrev, D.7
Eschenhagen, T.8
Carrier, L.9
-
30
-
-
0017146487
-
Studies on the phosphorylation of the inhibitory subunit of troponin during modification of contraction in perfused rat heart
-
England, P.J. (1976). Studies on the phosphorylation of the inhibitory subunit of troponin during modification of contraction in perfused rat heart. Biochem J 160, 295-304.
-
(1976)
Biochem J
, vol.160
, pp. 295-304
-
-
England, P.J.1
-
31
-
-
84867807258
-
Coordinated regulation of murine cardiomyocyte contractility by nanomolar (-)- epigallocatechin-3-gallate, the major green tea catechin
-
doi: 10.1124/mol.ll2.079707
-
Feng, W., Hwang, H.S., Kryshtal, D.O., Yang, T., Padilla, I.T., Tiwary, A.K., Puschner, B., Pessah, I.N., and Knollmann, B.C. (2012). Coordinated regulation of murine cardiomyocyte contractility by nanomolar (-)- epigallocatechin-3-gallate, the major green tea catechin. Mol Pharmacol 82, 993-1000. doi: 10.1124/mol.ll2.079707.
-
(2012)
Mol Pharmacol
, vol.82
, pp. 993-1000
-
-
Feng, W.1
Hwang, H.S.2
Kryshtal, D.O.3
Yang, T.4
Padilla, I.T.5
Tiwary, A.K.6
Puschner, B.7
Pessah, I.N.8
Knollmann, B.C.9
-
32
-
-
0033563035
-
Impaired cardiomyocyte relaxation and diastolic function in transgenic mice expressing slow skeletal troponin I in the heart
-
Fentzke, R.C., Buck, S.H., Patel, J.R., Lin, H., Wolska, B.M., Stojanovic, M.O., Martin, A.F., Solaro, R.J., Moss, R.L., and Leiden, J.M. (1999). Impaired cardiomyocyte relaxation and diastolic function in transgenic mice expressing slow skeletal troponin I in the heart. J Physiol (Lond) 517, 143-157.
-
(1999)
J Physiol (Lond)
, vol.517
, pp. 143-157
-
-
Fentzke, R.C.1
Buck, S.H.2
Patel, J.R.3
Lin, H.4
Wolska, B.M.5
Stojanovic, M.O.6
Martin, A.F.7
Solaro, R.J.8
Moss, R.L.9
Leiden, J.M.10
-
33
-
-
0343526912
-
Systematic mapping of regions of human cardiac troponin I involved in binding to cardiac troponin C: N- and Cterminal low affinity contributing regions
-
Ferrieres, G., Pugniere, M., Mani, J.C., Villard, S., Laprade, M., Doutre, P., Pau, B., and Granier, C. (2000). Systematic mapping of regions of human cardiac troponin I involved in binding to cardiac troponin C: N- and Cterminal low affinity contributing regions. FEBS Lett 479, 99-105.
-
(2000)
FEBS Lett
, vol.479
, pp. 99-105
-
-
Ferrieres, G.1
Pugniere, M.2
Mani, J.C.3
Villard, S.4
Laprade, M.5
Doutre, P.6
Pau, B.7
Granier, C.8
-
34
-
-
48849090596
-
*Altered functions and post-translational modification of contractile proteins in myectomy samples from HOCM patients
-
Gallon, C, Jacques, A., Messer, A., Tsang, V., Mckenna, W., and Marston, S. (2007). *Altered functions and post-translational modification of contractile proteins in myectomy samples from HOCM patients. J Mol Cell Cardiol 42, S167.
-
(2007)
J Mol Cell Cardiol
, vol.42
-
-
Gallon, C.1
Jacques, A.2
Messer, A.3
Tsang, V.4
Mckenna, W.5
Marston, S.6
-
35
-
-
26844461544
-
A mutation in the Nterminus of troponin I that is associated with hypertrophic cardiomyopathy affects the Ca(2+)-sensitivity, phosphorylation kinetics and proteolytic susceptibility of troponin
-
doi: 10.1016/j.yjmcc.2005.05.013
-
Gomes, A.V., Harada, K., and Potter, J.D. (2005). A mutation in the Nterminus of troponin I that is associated with hypertrophic cardiomyopathy affects the Ca(2+)-sensitivity, phosphorylation kinetics and proteolytic susceptibility of troponin. J Mol Cell Cardiol 39, 754-765. doi: 10.1016/j.yjmcc.2005.05.013.
-
(2005)
J Mol Cell Cardiol
, vol.39
, pp. 754-765
-
-
Gomes, A.V.1
Harada, K.2
Potter, J.D.3
-
36
-
-
0034059761
-
Regulation of contraction in striated muscle
-
Gordon, A.M., Homsher, E., and Regnier, M. (2000). Regulation of contraction in striated muscle. Physiol Rev 80, 853-924.
-
(2000)
Physiol Rev
, vol.80
, pp. 853-924
-
-
Gordon, A.M.1
Homsher, E.2
Regnier, M.3
-
37
-
-
84906551224
-
*Modulation of the Interaction between Troponin I N-Terminal Peptide and Troponin C by Phosphorylation Studied by Molecular Dynamics
-
doi: 10.1016/j.bpj.2013.11.1988
-
Gould, I., Messer, A.E., Papadaki, M., and Marston, S.B. (2014). *Modulation of the Interaction between Troponin I N-Terminal Peptide and Troponin C by Phosphorylation Studied by Molecular Dynamics. BiophysJ 106, 349a. doi: 10.1016/j.bpj.2013.11.1988.
-
(2014)
BiophysJ
, vol.106
-
-
Gould, I.1
Messer, A.E.2
Papadaki, M.3
Marston, S.B.4
-
38
-
-
67650738888
-
Stress-induced dilated cardiomyopathy in a knock-in mouse model mimicking human titinbased disease
-
doi: 10.1016/j.yjmcc.2009.04.014
-
Gramlich, M., Michely, B., Krohne, C, Heuser, A., Erdmann, B., Klaassen, S., Hudson, B., Magarin, M., Kirchner, F., Todiras, M., Granzier, H., Labeit, S., Thierfelder, L, and Gerull, B. (2009). Stress-induced dilated cardiomyopathy in a knock-in mouse model mimicking human titinbased disease. J Mol Cell Cardiol 47, 352-358. doi: 10.1016/j.yjmcc.2009.04.014.
-
(2009)
J Mol Cell Cardiol
, vol.47
, pp. 352-358
-
-
Gramlich, M.1
Michely, B.2
Krohne, C.3
Heuser, A.4
Erdmann, B.5
Klaassen, S.6
Hudson, B.7
Magarin, M.8
Kirchner, F.9
Todiras, M.10
Granzier, H.11
Labeit, S.12
Thierfelder, L.13
Gerull, B.14
-
39
-
-
68049115750
-
Temporal and mutation-specific alterations in Ca2 + homeostasis differentially determine the progression of cTnT-related cardiomyopathies in murine models
-
doi: 10.1152/ajpheart.01143.2008
-
Guinto, P.J., Haim, T.E., Dowell-Martino, C.C., Sibinga, N., and Tardiff, J.C. (2009). Temporal and mutation-specific alterations in Ca2 + homeostasis differentially determine the progression of cTnT-related cardiomyopathies in murine models. Am J PhysiolHeart Circ Physiol 297, H614-626. doi: 10.1152/ajpheart.01143.2008.
-
(2009)
Am J PhysiolHeart Circ Physiol
, vol.297
-
-
Guinto, P.J.1
Haim, T.E.2
Dowell-Martino, C.C.3
Sibinga, N.4
Tardiff, J.C.5
-
40
-
-
62449234647
-
Myofilament dysfunction in cardiac disease from mice to men
-
Hamdani, N., De Waard, M., Messer, A.E., Boontje, N.M., Kooij, V., Van Dijk, S., Versteilen, A., Lamberts, R., Merkus, D., Dos Remedios, C., Duncker, D.J., Borbely, A., Papp, Z., Paulus, Wv Stienen, G.J., Marston, S.B., and Van Der Velden, J. (2009). Myofilament dysfunction in cardiac disease from mice to men. J Muse Res Cell Motil 29,189-201.
-
(2009)
J Muse Res Cell Motil
, vol.29
, pp. 189-201
-
-
Hamdani, N.1
De Waard, M.2
Messer, A.E.3
Boontje, N.M.4
Kooij, V.5
Van Dijk, S.6
Versteilen, A.7
Lamberts, R.8
Merkus, D.9
Dos Remedios, C.10
Duncker, D.J.11
Borbely, A.12
Papp, Z.13
Paulus Wv Stienen, G.J.14
Marston, S.B.15
Van Der Velden, J.16
-
41
-
-
9344230851
-
Protein kinase D is a novel mediator of cardiac troponin I phosphorylation and regulates myofilament function
-
Haworth, R.S., Cuello, F., Herron, T.J., Franzen, G., Kentish, J.C., Gautel, M., and Avkiran, M. (2004). Protein kinase D is a novel mediator of cardiac troponin I phosphorylation and regulates myofilament function. Circ Res 95,1091-1099.
-
(2004)
Circ Res
, vol.95
, pp. 1091-1099
-
-
Haworth, R.S.1
Cuello, F.2
Herron, T.J.3
Franzen, G.4
Kentish, J.C.5
Gautel, M.6
Avkiran, M.7
-
42
-
-
0037423881
-
Is depressed myocyte contractility centrally involved in heart failure?
-
Houser, S.R., and Margulies, K.B. (2003). Is depressed myocyte contractility centrally involved in heart failure? Circ Res 92, 350-358.
-
(2003)
Circ Res
, vol.92
, pp. 350-358
-
-
Houser, S.R.1
Margulies, K.B.2
-
43
-
-
34748818674
-
Phosphorylation-dependent conformational transition of the cardiac specific N-extension of troponin I in cardiac troponin
-
doi: 10.1016/j.jmb.2007.08.035
-
Howarth, J.W., Meller, J., Solaro, R.J., Trewhella, J., and Rosevear, P.R. (2007). Phosphorylation-dependent conformational transition of the cardiac specific N-extension of troponin I in cardiac troponin. J Mol Biol 373, 706-722. doi: 10.1016/j.jmb.2007.08.035.
-
(2007)
J Mol Biol
, vol.373
, pp. 706-722
-
-
Howarth, J.W.1
Meller, J.2
Solaro, R.J.3
Trewhella, J.4
Rosevear, P.R.5
-
44
-
-
84884810564
-
Depressed Frank-Starling mechanism in the left ventricular muscle of the knock-in mouse model of dilated cardiomyopathy with troponin T deletion mutation AK210
-
doi: 10.1016/j.yjmcc.2013.07.001
-
Inoue, T., Kobirumaki-Shimozawa, F., Kagemoto, T., Fujii, T., Terui, T., Kusakari, Y., Hongo, K., Morimoto, S., Ohtsuki, I., Hashimoto, K., and Fukuda, N. (2013). Depressed Frank-Starling mechanism in the left ventricular muscle of the knock-in mouse model of dilated cardiomyopathy with troponin T deletion mutation AK210. J Mol Cell Cardiol 63, 69-78. doi: 10.1016/j.yjmcc.2013.07.001.
-
(2013)
J Mol Cell Cardiol
, vol.63
, pp. 69-78
-
-
Inoue, T.1
Kobirumaki-Shimozawa, F.2
Kagemoto, T.3
Fujii, T.4
Terui, T.5
Kusakari, Y.6
Hongo, K.7
Morimoto, S.8
Ohtsuki, I.9
Hashimoto, K.10
Fukuda, N.11
-
45
-
-
48749118698
-
The molecular phenotype of human cardiac myosin associated with hypertrophic obstructive cardiomyopathy
-
doi: 10.1093/cvr/cvn094
-
Jacques, A., Briceno, N., Messer, A., Gallon, C., Jalizadeh, S., Garcia, E., Kikonda-Kanda, G., Goddard, J., Harding, S., Watkins, H., Tsang, V, Mckenna, W., and Marston, S. (2008). The molecular phenotype of human cardiac myosin associated with hypertrophic obstructive cardiomyopathy. Cardiovasc Res 79, 481-491. doi: 10.1093/cvr/cvn094.
-
(2008)
Cardiovasc Res
, vol.79
, pp. 481-491
-
-
Jacques, A.1
Briceno, N.2
Messer, A.3
Gallon, C.4
Jalizadeh, S.5
Garcia, E.6
Kikonda-Kanda, G.7
Goddard, J.8
Harding, S.9
Watkins, H.10
Tsang, V.11
Mckenna, W.12
Marston, S.13
-
46
-
-
62449192447
-
From genotype to phenotype: a longitudinal study of a patient with hypertrophic cardiomyopathy due to a mutation in the MYBPC3 gene
-
doi: 10.1007/sl0974-009-9174-0
-
Jacques, A., Hoskins, A., Kentish, J., and Marston, S.B. (2009). From genotype to phenotype: a longitudinal study of a patient with hypertrophic cardiomyopathy due to a mutation in the MYBPC3 gene. J Muse Res Cell Motil 29, 239-246. doi: 10.1007/sl0974-009-9174-0.
-
(2009)
J Muse Res Cell Motil
, vol.29
, pp. 239-246
-
-
Jacques, A.1
Hoskins, A.2
Kentish, J.3
Marston, S.B.4
-
47
-
-
85047691343
-
Decreased energetics in murine hearts bearing the R92Q mutation in cardiac troponin T
-
Javadpour, M.M., Tardiff, J.C., Pinz, I., and Ingwall, J., S. (2003). Decreased energetics in murine hearts bearing the R92Q mutation in cardiac troponin T.J. Clin Invest 112, 768-775.
-
(2003)
J Clin Invest
, vol.112
, pp. 768-775
-
-
Javadpour, M.M.1
Tardiff, J.C.2
Pinz, I.3
Ingwall, J.S.4
-
48
-
-
34047134952
-
The use of human cardiac tissue in biophysical research: the risks of translation
-
Jweied, E., Detombe, P., and Buttrick, P.M. (2007). The use of human cardiac tissue in biophysical research: the risks of translation. J Mol Cell Cardiol 42, 722-726.
-
(2007)
J Mol Cell Cardiol
, vol.42
, pp. 722-726
-
-
Jweied, E.1
Detombe, P.2
Buttrick, P.M.3
-
49
-
-
34748913759
-
Mutations in the cardiac Troponin C gene are a cause of idiopathic dilated cardiomyopathy in childhood
-
Kaski, J.P., Burch, M., and Elliott, P.M. (2007). Mutations in the cardiac Troponin C gene are a cause of idiopathic dilated cardiomyopathy in childhood. Cardiol Young 17, 675-677.
-
(2007)
Cardiol Young
, vol.17
, pp. 675-677
-
-
Kaski, J.P.1
Burch, M.2
Elliott, P.M.3
-
50
-
-
0025260746
-
Peptide mimetics of an actin-binding site on myosin span two functional domains on actin
-
Keane, A.M., Trayer, I.P., Levine, B.A., Zeugner, C, and Ruegg, C.J. (1990). Peptide mimetics of an actin-binding site on myosin span two functional domains on actin. Nature 344, 265-268.
-
(1990)
Nature
, vol.344
, pp. 265-268
-
-
Keane, A.M.1
Trayer, I.P.2
Levine, B.A.3
Zeugner, C.4
Ruegg, C.J.5
-
51
-
-
0035947749
-
Phosphorylation of troponin I by protein kinase A accelerates relaxation and crossbridge cycle kinetics in mouse ventricular muscle
-
Kentish, J.C., Mccloskey, D.T., Layland, J., Palmer, S., Leiden, J.M., Martin, A.F., and Solaro, R.J. (2001). Phosphorylation of troponin I by protein kinase A accelerates relaxation and crossbridge cycle kinetics in mouse ventricular muscle. Circ Res 88,1059-1065.
-
(2001)
Circ Res
, vol.88
, pp. 1059-1065
-
-
Kentish, J.C.1
Mccloskey, D.T.2
Layland, J.3
Palmer, S.4
Leiden, J.M.5
Martin, A.F.6
Solaro, R.J.7
-
52
-
-
33645714857
-
Phosphate-binding tag, a new tool to visualize phosphorylated proteins
-
Kinoshita, E., Kinoshita-Kikuta, E., Takiyama, K., and Koike, T. (2006). Phosphate-binding tag, a new tool to visualize phosphorylated proteins. Mol Cell Proteomics 5, 749-757.
-
(2006)
Mol Cell Proteomics
, vol.5
, pp. 749-757
-
-
Kinoshita, E.1
Kinoshita-Kikuta, E.2
Takiyama, K.3
Koike, T.4
-
53
-
-
77949379473
-
A common MLP (muscle LIM protein) variant is associated with cardiomyopathy
-
doi: 10.1161/CIRCRESAHA.109.206243
-
Knoell, R., Kostin, S., Klede, S., Savvatis, K., Klinge, L, Stehle, I., Gunkel, S., Koetter, S., Babicz, K., Sohns, M., Miocic, S., Didier, M., Knoell, G., Zimmermann, W.H., Thelen, P., Bickeboeller, H., Maier, L.S., Schaper, W., Schaper, J., Kraft, T., Tschoepe, C, Linke, W.A., and Chien, K.R. (2010). A common MLP (muscle LIM protein) variant is associated with cardiomyopathy. Circ Res 106, 695-704. doi: 10.1161/CIRCRESAHA.109.206243.
-
(2010)
Circ Res
, vol.106
, pp. 695-704
-
-
Knoell, R.1
Kostin, S.2
Klede, S.3
Savvatis, K.4
Klinge, L.5
Stehle, I.6
Gunkel, S.7
Koetter, S.8
Babicz, K.9
Sohns, M.10
Miocic, S.11
Didier, M.12
Knoell, G.13
Zimmermann, W.H.14
Thelen, P.15
Bickeboeller, H.16
Maier, L.S.17
Schaper, W.18
Schaper, J.19
Kraft, T.20
Tschoepe, C.21
Linke, W.A.22
Chien, K.R.23
more..
-
54
-
-
80053046816
-
Telethonin Deficiency Is Associated With Maladaptation to Biomechanical Stress in the Mammalian Heart
-
doi: 10.1161/CIRCRESAHA.lll.245787.
-
Knoell, R., Linke, W.A., Zou, P., Miocic, S., Kostin, S., Buyandelger, B., Ku, C.H., Neef, S., Bug, M., Sch √§Fer, K., Knoell, G., Felkin, L.E., Wessels, J., Toischer, K., Hagn, F., Kessler, H., Didi√. ©, M., Quentin, T., Maier, L.S., Teucher, N., Uns √∂Ld, B., Schmidt, A., Birks, E.J., Gunkel, S., Lang, P., Granzier, H., Zimmermann, W.H., Field, L.J., Faulkner, G., Dobbelstein, M., Barton, P.J., Sattler, M., Wilmanns, M., and Chien, K.R. (2011). Telethonin Deficiency Is Associated With Maladaptation to Biomechanical Stress in the Mammalian Heart. Circ Res, 758-769. doi: 10.1161/CIRCRESAHA.lll.245787.
-
(2011)
Circ Res
, pp. 758-769
-
-
Knoell, R.1
Linke, W.A.2
Zou, P.3
Miocic, S.4
Kostin, S.5
Buyandelger, B.6
Ku, C.H.7
Neef, S.8
Bug, M.9
Sch Fer, K.10
Knoell, G.11
Felkin, L.E.12
Wessels, J.13
Toischer, K.14
Hagn, F.15
Kessler, H.16
Didi, M.17
Quentin, T.18
Maier, L.S.19
Teucher, N.20
Uns Ld, B.21
Schmidt, A.22
Birks, E.J.23
Gunkel, S.24
Lang, P.25
Granzier, H.26
Zimmermann, W.H.27
Field, L.J.28
Faulkner, G.29
Dobbelstein, M.30
Barton, P.J.31
Sattler, M.32
Wilmanns, M.33
Chien, K.R.34
more..
-
55
-
-
0035971216
-
Inotropic stimulation induces cardiac dysfunction in transgenic mice expressing a troponin T (I79N) mutation linked to familial hypertrophic cardiomyopathy
-
Knollmann, B.C., Blatt, S.A., Horton, K., De Freitas, F., Miller, T., Bell, M., Housmans, P.R., Weissman, N.J., Morad, M., and Potter, J.D. (2001). Inotropic stimulation induces cardiac dysfunction in transgenic mice expressing a troponin T (I79N) mutation linked to familial hypertrophic cardiomyopathy. J Biol Chem 276,10039-10048.
-
(2001)
J Biol Chem
, vol.276
, pp. 10039-10048
-
-
Knollmann, B.C.1
Blatt, S.A.2
Horton, K.3
De Freitas, F.4
Miller, T.5
Bell, M.6
Housmans, P.R.7
Weissman, N.J.8
Morad, M.9
Potter, J.D.10
-
56
-
-
11144312378
-
A non-equilibrium isoelectric focusing method to determine states of phosphorylation of cardiac troponin I: identification of Ser- 23 and Ser-24 as significant sites of phosphorylation by protein kinase C
-
Kobayashi, T., Yang, X., Walker, L.A., Van Breemen, R.B., and Solaro, R.J. (2005). A non-equilibrium isoelectric focusing method to determine states of phosphorylation of cardiac troponin I: identification of Ser- 23 and Ser-24 as significant sites of phosphorylation by protein kinase C.J Mol Cell Cardiol 38, 213-218.
-
(2005)
J Mol Cell Cardiol
, vol.38
, pp. 213-218
-
-
Kobayashi, T.1
Yang, X.2
Walker, L.A.3
Van Breemen, R.B.4
Solaro, R.J.5
-
57
-
-
77951623074
-
Effect of troponin I Ser23/24 phosphorylation on Ca2+-sensitivity in human myocardium depends on the phosphorylation background
-
doi: 10.1016/j.yjmcc.2010.01.002.
-
Kooij, V., Saes, M., Jaquet, K., Zaremba, R., Foster, D.B., Murphy, A., Remedios, C, Velden, J., and Stienen, GJ. (2010). Effect of troponin I Ser23/24 phosphorylation on Ca2+-sensitivity in human myocardium depends on the phosphorylation background. J Mol Cell Cardiol 48, 954-963. doi: 10.1016/j.yjmcc.2010.01.002.
-
(2010)
J Mol Cell Cardiol
, vol.48
, pp. 954-963
-
-
Kooij, V.1
Saes, M.2
Jaquet, K.3
Zaremba, R.4
Foster, D.B.5
Murphy, A.6
Remedios, C.7
Velden, J.8
Stienen, G.J.9
-
58
-
-
14844344027
-
Regulation of cardiac contractile function by troponin I phosphorylation
-
Layland, J., Solaro, R.J., and Shah, A.M. (2005). Regulation of cardiac contractile function by troponin I phosphorylation. Cardiovasc Res 66, 12-21.
-
(2005)
Cardiovasc Res
, vol.66
, pp. 12-21
-
-
Layland, J.1
Solaro, R.J.2
Shah, A.M.3
-
59
-
-
84894137070
-
Familial hypertrophic cardiomyopathy related cardiac troponin C L29Q mutation alters length-dependent activation and functional effects of phosphomimetic troponin I*
-
doi: 10.1371/journal.pone.0079363
-
Li, A.Y., Stevens, CM., Liang, B., Rayani, K., Little, S., Davis, J., and Tibbits, G.F. (2013). Familial hypertrophic cardiomyopathy related cardiac troponin C L29Q mutation alters length-dependent activation and functional effects of phosphomimetic troponin I*. PLoS ONE 8, e79363. doi: 10.1371/journal.pone.0079363.
-
(2013)
PLoS ONE
, vol.8
-
-
Li, A.Y.1
Stevens, C.M.2
Liang, B.3
Rayani, K.4
Little, S.5
Davis, J.6
Tibbits, G.F.7
-
60
-
-
40849131184
-
Interaction of cardiac troponin with cardiotonic drugs: a structural perspective
-
doi: 10.1016/j.bbrc.2007.12.108
-
Li, M.X., Robertson, I.M., and Sykes, B.D. (2008). Interaction of cardiac troponin with cardiotonic drugs: a structural perspective. Biochem Biophys Res Commun 369, 88-99. doi: 10.1016/j.bbrc.2007.12.108.
-
(2008)
Biochem Biophys Res Commun
, vol.369
, pp. 88-99
-
-
Li, M.X.1
Robertson, I.M.2
Sykes, B.D.3
-
61
-
-
84861880853
-
Disease-related cardiac troponins alter thin filament Ca2+ association and dissociation rates
-
doi: 10.1371/journal.pone.0038259
-
Liu, B., Tikunova, S.B., Kline, K.P., Siddiqui, J.K., and Davis, J.P. (2012). Disease-related cardiac troponins alter thin filament Ca2+ association and dissociation rates. PLoS ONE 7, e38259. doi: 10.1371/journal.pone.0038259.
-
(2012)
PLoS ONE
, vol.7
-
-
Liu, B.1
Tikunova, S.B.2
Kline, K.P.3
Siddiqui, J.K.4
Davis, J.P.5
-
62
-
-
85044827827
-
Cardiac myocytes and the cardiac action potential
-
eds. D.A. Warrel, T.M. Cox & J.D. Firth. 5th ed (Oxford: Oxford Textbook of Medicine)
-
Macleod, K.T., Marston, S.B., Poole-Wilson, P.A., Severs, N.J., and Sugden, P.H. (2010). "Cardiac myocytes and the cardiac action potential," eds. D.A. Warrel, T.M. Cox & J.D. Firth. 5th ed (Oxford: Oxford Textbook of Medicine), 2603-2617.
-
(2010)
, pp. 2603-2617
-
-
Macleod, K.T.1
Marston, S.B.2
Poole-Wilson, P.A.3
Severs, N.J.4
Sugden, P.H.5
-
63
-
-
84864216481
-
Alanine or aspartic acid substitutions atserine23/24 of cardiac troponin I decrease thin filament activation, with no effect on crossbridge detachment kinetics
-
doi: 10.1016/j.abb.2012.05.024
-
Mamidi, R., Gollapudi, S.K., Mallampalli, S.L., and Chandra, M. (2012). Alanine or aspartic acid substitutions atserine23/24 of cardiac troponin I decrease thin filament activation, with no effect on crossbridge detachment kinetics. Arch Biochem Biophys 525,1-8. doi: 10.1016/j.abb.2012.05.024.
-
(2012)
Arch Biochem Biophys
, vol.525
, pp. 1-8
-
-
Mamidi, R.1
Gollapudi, S.K.2
Mallampalli, S.L.3
Chandra, M.4
-
64
-
-
80051983795
-
A Model of Calcium Activation of the Cardiac Thin Filament
-
doi: 10.1021/bi200506k
-
Manning, E.P., Tardiff, J.C., and Schwartz, S.D. (2011). A Model of Calcium Activation of the Cardiac Thin Filament. Biochemistry 50, 7405-7413. doi: 10.1021/bi200506k.
-
(2011)
Biochemistry
, vol.50
, pp. 7405-7413
-
-
Manning, E.P.1
Tardiff, J.C.2
Schwartz, S.D.3
-
65
-
-
55649083104
-
Point/Counterpoint. Troponin phosphorylation and myofilament Ca2+-sensitivity in heart failure: increased or decreased?
-
doi: 10.1016/j.yjmcc.2008.07.004
-
Marston, S., and Detombe, P. (2008). Point/Counterpoint. Troponin phosphorylation and myofilament Ca2+-sensitivity in heart failure: increased or decreased? JMol Cell Cardiol 45, 603-607. doi: 10.1016/j.yjmcc.2008.07.004.
-
(2008)
JMol Cell Cardiol
, vol.45
, pp. 603-607
-
-
Marston, S.1
Detombe, P.2
-
66
-
-
79960564849
-
How Do Mutations in Contractile Proteins Cause the Primary Familial Cardiomyopathies?
-
doi: 10.1007/sl2265-011-9266-2
-
Marston, S.B. (2011). How Do Mutations in Contractile Proteins Cause the Primary Familial Cardiomyopathies? J Cardiovasc Transl Res 4, 245-255. doi: 10.1007/sl2265-011-9266-2.
-
(2011)
J Cardiovasc Transl Res
, vol.4
, pp. 245-255
-
-
Marston, S.B.1
-
67
-
-
84906567311
-
Pressure Overload is Associated with Low Levels of Troponin I and Myosin Binding Protein C Phosphorylation in the Hearts of Patients with Aortic Stenosis
-
Marston, S.B., Copeland, O.a. N., and Messer, A. (2012). Pressure Overload is Associated with Low Levels of Troponin I and Myosin Binding Protein C Phosphorylation in the Hearts of Patients with Aortic Stenosis. Circulation 126, A14155.
-
(2012)
Circulation
, vol.126
-
-
Marston, S.B.1
Copeland O.a., N.2
Messer, A.3
-
68
-
-
84906534432
-
DCMcausing mutation ACTC E361G blunts responses to adrener- gic agonists, reduces cardiac reserve and predispose to heart failure under chronic stress in a transgenic mouse model
-
Marston, S.B., Song, W., Wilkinson, R., and Vikhorev, P.G. (2013). DCMcausing mutation ACTC E361G blunts responses to adrener- gic agonists, reduces cardiac reserve and predispose to heart failure under chronic stress in a transgenic mouse model. J Mol Cell Cardiol 65,slO8.
-
(2013)
J Mol Cell Cardiol
, vol.65
-
-
Marston, S.B.1
Song, W.2
Wilkinson, R.3
Vikhorev, P.G.4
-
69
-
-
69549109879
-
Back to the future: new techniques show that forgotten phosphorylation sites are present in contractile proteins of the heart whilst intensively studied sites appear to be absent
-
doi: 10.1007/sl0974-009-9184-y
-
Marston, S.B., and Walker, J.W. (2009). Back to the future: new techniques show that forgotten phosphorylation sites are present in contractile proteins of the heart whilst intensively studied sites appear to be absent. J Muscle Res Cell Motil 30,93-93. doi: 10.1007/sl0974-009-9184-y.
-
(2009)
J Muscle Res Cell Motil
, vol.30
, pp. 93-93
-
-
Marston, S.B.1
Walker, J.W.2
-
70
-
-
84879390656
-
Mutations in thin Filament Proteins that Cause Familial Dilated Cardiomyopathy Uncouple Troponin I Phosphorylation from Changes in Myofibrillar Ca2+-Sensitivity
-
doi: 10.1093/cvr/cvt071
-
Memo, M., Leung, M.-C, Ward, D.G., Dos Remedios, C, Morimoto, S., Zhang, L, Ravenscroft, G., Mcnamara, E., Nowak, K.J., Marston, S.B., and Messer, A.E. (2013). Mutations in thin Filament Proteins that Cause Familial Dilated Cardiomyopathy Uncouple Troponin I Phosphorylation from Changes in Myofibrillar Ca2+-Sensitivity. Cardiovasc Res 99, 65-73. doi: 10.1093/cvr/cvt071.
-
(2013)
Cardiovasc Res
, vol.99
, pp. 65-73
-
-
Memo, M.1
Leung, M.-C.2
Ward, D.G.3
Dos Remedios, C.4
Morimoto, S.5
Zhang, L.6
Ravenscroft, G.7
Mcnamara, E.8
Nowak, K.J.9
Marston, S.B.10
Messer, A.E.11
-
71
-
-
77951074751
-
The use of phosphate-affinity SDS-PAGE to measure the troponin I phosphorylation site distribution in human heart muscle
-
doi: 10.1002/prca.200900071.
-
Messer, A., Gallon, C, Mckenna, W., Elliott, P., Dos Remedios, C, and Marston, S. (2009). The use of phosphate-affinity SDS-PAGE to measure the troponin I phosphorylation site distribution in human heart muscle. ProteomicsClinAppl3,1371-1382. doi: 10.1002/prca.200900071.
-
(2009)
Proteomics Clin Appl
, vol.3
, pp. 1371-1382
-
-
Messer, A.1
Gallon, C.2
Mckenna, W.3
Elliott, P.4
Dos Remedios, C.5
Marston, S.6
-
72
-
-
33845778948
-
Troponin phosphorylation and regulatory function in human heart muscle: Dephosphorylation of Ser23/24 on troponin I could account for the contractile defect in end-stage heart failure.
-
doi: 10.1016/j.yjmcc.2006.08.017
-
Messer, A.E., Jacques, A.M., and Marston, S.B. (2007). Troponin phosphorylation and regulatory function in human heart muscle: Dephosphorylation of Ser23/24 on troponin I could account for the contractile defect in end-stage heart failure. J Mol Cell Cardiol 42, 247-259. doi: 10.1016/j.yjmcc.2006.08.017
-
(2007)
J Mol Cell Cardiol
, vol.42
, pp. 247-259
-
-
Messer, A.E.1
Jacques, A.M.2
Marston, S.B.3
-
73
-
-
84906540645
-
*Does Uncoupling of Troponin I Phosphorylation from Changes in Myofibrillar Ca2+-Sensitivity Play a Role in the Pathogenesis of Cardiomyopathy?
-
doi: 10.1016/j.bpj. 2011.11.3033
-
Messer, A.E., Memo, M., Bayliss, C.R., Leung, M.-C, and Marston, S.B. (2012). *Does Uncoupling of Troponin I Phosphorylation from Changes in Myofibrillar Ca2+-Sensitivity Play a Role in the Pathogenesis of Cardiomyopathy? BiophysJ 102, 556a. doi: 10.1016/j.bpj. 2011.11.3033.
-
(2012)
BiophysJ
, vol.102
-
-
Messer, A.E.1
Memo, M.2
Bayliss, C.R.3
Leung, M.-C.4
Marston, S.B.5
-
74
-
-
84906567297
-
*Effects of EMD57033 and EGCG on Modulation of Ca2+-Sensitivity by Pka Phosphorylation
-
doi: 10.1016/j.bpj.2013.11.4010
-
Messer, A.E., Papadaki, M., and Marston, S.B. (2014). *Effects of EMD57033 and EGCG on Modulation of Ca2+-Sensitivity by Pka Phosphorylation. BiophysJ 106, 726a-727a. doi: 10.1016/j.bpj.2013.11.4010.
-
(2014)
BiophysJ
, vol.106
-
-
Messer, A.E.1
Papadaki, M.2
Marston, S.B.3
-
75
-
-
23344452467
-
Dilated cardiomyopathy mutations in three thin filament regulatory proteins result in a common functional phenotype
-
Mirza, M., Marston, S., Willott, R., Ashley, C, Mogensen, J., Mckenna, W., Robinson, P., Redwood, C, and Watkins, H. (2005). Dilated cardiomyopathy mutations in three thin filament regulatory proteins result in a common functional phenotype. J Mol Chem 280, 28498-28506.
-
(2005)
J Mol Chem
, vol.280
, pp. 28498-28506
-
-
Mirza, M.1
Marston, S.2
Willott, R.3
Ashley, C.4
Mogensen, J.5
Mckenna, W.6
Robinson, P.7
Redwood, C.8
Watkins, H.9
-
76
-
-
0025107563
-
A common motif of two adjacent phosphoserines in bovine, rabbit and human cardiac troponin I
-
Mittmann, K., Jaquet, K., and Heilmeyer, L.M., Jr. (1990). A common motif of two adjacent phosphoserines in bovine, rabbit and human cardiac troponin I. FEBS Lett 273, 41-45.
-
(1990)
FEBS Lett
, vol.273
, pp. 41-45
-
-
Mittmann, K.1
Jaquet, K.2
Heilmeyer Jr., L.M.3
-
77
-
-
8144224216
-
Severe disease expression of cardiac troponin C and T mutations in patients with idiopathic dilated cardiomyopathy
-
Mogensen, J., Murphy, R.T., Shaw, T., Bahl, A., Redwood, C, Watkins, H., Burke, M., Elliott, P.M., and Mckenna, W.J. (2004). Severe disease expression of cardiac troponin C and T mutations in patients with idiopathic dilated cardiomyopathy. J Am Coll Cardiol. 44, 2033-2040.
-
(2004)
J Am Coll Cardiol
, vol.44
, pp. 2033-2040
-
-
Mogensen, J.1
Murphy, R.T.2
Shaw, T.3
Bahl, A.4
Redwood, C.5
Watkins, H.6
Burke, M.7
Elliott, P.M.8
Mckenna, W.J.9
-
78
-
-
84876321824
-
HCM-linked A160E cardiac troponin T mutation causes unique progressive structural and molecular ventricular remodeling in transgenic mice
-
doi: 10.1016/j.yjmcc.2013.02.004
-
Moore, R.K., Grinspan, L.T., Jimenez, J., Guinto, P.J., Ertz-Berger, B., and Tardiff, J.C. (2013). HCM-linked A160E cardiac troponin T mutation causes unique progressive structural and molecular ventricular remodeling in transgenic mice. J Mol Cell Cardiol 58,188-198. doi: 10.1016/j.yjmcc.2013.02.004.
-
(2013)
J Mol Cell Cardiol
, vol.58
, pp. 188-198
-
-
Moore, R.K.1
Grinspan, L.T.2
Jimenez, J.3
Guinto, P.J.4
Ertz-Berger, B.5
Tardiff, J.C.6
-
79
-
-
0019190218
-
Calcium sensitivity of the contractile system and phosphorylation of troponin in hyperpermeable cardiac cells
-
Mope, L, Mcclellan, G.B., and Winegrad, S. (1980). Calcium sensitivity of the contractile system and phosphorylation of troponin in hyperpermeable cardiac cells. J Gen Physiol 75, 271-282.
-
(1980)
J Gen Physiol
, vol.75
, pp. 271-282
-
-
Mope, L.1
Mcclellan, G.B.2
Winegrad, S.3
-
80
-
-
39749103537
-
Sarcomeric proteins and inherited cardiomyopathies
-
doi: 10.1093/cvr/cvm084
-
Morimoto, S. (2008). Sarcomeric proteins and inherited cardiomyopathies. Cardiovasc Res 77, 659-666. doi: 10.1093/cvr/cvm084.
-
(2008)
Cardiovasc Res
, vol.77
, pp. 659-666
-
-
Morimoto, S.1
-
81
-
-
0033230078
-
Myocardial contractile reserve on dobutamine echocardiography predicts late spontaneous improvement in cardiac function in patients with recent onset idiopathic dilated cardiomyopathy
-
Naqvi, T.Z., Goel, R.K., Forrester, J.S., and Siegel, R.J. (1999). Myocardial contractile reserve on dobutamine echocardiography predicts late spontaneous improvement in cardiac function in patients with recent onset idiopathic dilated cardiomyopathy. J Am Coll Cardiol. 34,1537-1544.
-
(1999)
J Am Coll Cardiol
, vol.34
, pp. 1537-1544
-
-
Naqvi, T.Z.1
Goel, R.K.2
Forrester, J.S.3
Siegel, R.J.4
-
82
-
-
34249274852
-
Abnormal cardiac response to exercise in a murine model of familial hypertrophic cardiomyopathy
-
doi: 10.1016/j.ijcard.2006.09.001
-
Nguyen, L, Chung, J., Lam, L, Tsoutsman, T., and Semsarian, C. (2007). Abnormal cardiac response to exercise in a murine model of familial hypertrophic cardiomyopathy. IntJ Cardiol 119, 245-248. doi: 10.1016/j.ijcard.2006.09.001.
-
(2007)
IntJ Cardiol
, vol.119
, pp. 245-248
-
-
Nguyen, L.1
Chung, J.2
Lam, L.3
Tsoutsman, T.4
Semsarian, C.5
-
83
-
-
84861740319
-
AMP-activated protein kinase phosphorylates cardiac troponin I at Ser-150 to increase myofilament calcium sensitivity and blunt PKA-dependent function
-
doi: 10.1074/jbc. M111.323048
-
Nixon, B.R., Thawornkaiwong, A., Jin, J., Brundage, E.A., Little, S.C., Davis, J.P., Solaro, R.J., and Biesiadecki, B.J. (2012). AMP-activated protein kinase phosphorylates cardiac troponin I at Ser-150 to increase myofilament calcium sensitivity and blunt PKA-dependent function. J Biol Chem 287,19136-19147. doi: 10.1074/jbc. M111.323048.
-
(2012)
J Biol Chem
, vol.287
, pp. 19136-19147
-
-
Nixon, B.R.1
Thawornkaiwong, A.2
Jin, J.3
Brundage, E.A.4
Little, S.C.5
Davis, J.P.6
Solaro, R.J.7
Biesiadecki, B.J.8
-
84
-
-
0037023633
-
Phosphorylation of troponin I controls cardiac twitch dynamics. Evidence from phosphorylation site mutants expressed on a troponin I-null background in mice
-
Pi, Y.-Q., Kemnitz, K.R., Zhang, D., Kranias, E.G., and Walker, J.W. (2002). Phosphorylation of troponin I controls cardiac twitch dynamics. Evidence from phosphorylation site mutants expressed on a troponin I-null background in mice. Circ Res 90, 649-656.
-
(2002)
Circ Res
, vol.90
, pp. 649-656
-
-
Pi, Y.-Q.1
Kemnitz, K.R.2
Zhang, D.3
Kranias, E.G.4
Walker, J.W.5
-
85
-
-
80053195075
-
Functional Characterization of TNNC1 Rare Variants Identified in Dilated Cardiomyopathy
-
doi: 10.1074/jbc. M111.267211
-
Pinto, J.R., Siegfried, J.D., Parvatiyar, M.S., Li, D., Norton, N., Jones, M.A., Liang, J., Potter, J.D., and Hershberger, R.E. (2011). Functional Characterization of TNNC1 Rare Variants Identified in Dilated Cardiomyopathy. J Biol Chem 286, 34404-34412. doi: 10.1074/jbc. M111.267211.
-
(2011)
J Biol Chem
, vol.286
, pp. 34404-34412
-
-
Pinto, J.R.1
Siegfried, J.D.2
Parvatiyar, M.S.3
Li, D.4
Norton, N.5
Jones, M.A.6
Liang, J.7
Potter, J.D.8
Hershberger, R.E.9
-
86
-
-
34547605959
-
Dilated cardiomyopathy mutant tropomyosin mice develop cardiac dysfunction with significantly decreased fractional shortening and myofilament calcium sensitivity
-
Rajan, S., Ahmed, R.P., Jagatheesan, G., Petrashevskaya, N., Boivin, G.P., Urboniene, D., Arteaga, G.M., Wolska, B.M., Solaro, R.J., Liggett, S.B., and Wieczorek, D.F. (2007). Dilated cardiomyopathy mutant tropomyosin mice develop cardiac dysfunction with significantly decreased fractional shortening and myofilament calcium sensitivity. CircRes 101, 205-214.
-
(2007)
CircRes
, vol.101
, pp. 205-214
-
-
Rajan, S.1
Ahmed, R.P.2
Jagatheesan, G.3
Petrashevskaya, N.4
Boivin, G.P.5
Urboniene, D.6
Arteaga, G.M.7
Wolska, B.M.8
Solaro, R.J.9
Liggett, S.B.10
Wieczorek, D.F.11
-
87
-
-
0017176627
-
Phosphorylation of the inhibitory subunit of troponin and its effect on the calcium dependence of cardiac myofibril adenosine triphosphatase
-
Ray, K.P., and England, P.J. (1976). Phosphorylation of the inhibitory subunit of troponin and its effect on the calcium dependence of cardiac myofibril adenosine triphosphatase. FEBS Lett 70,11-16.
-
(1976)
FEBS Lett
, vol.70
, pp. 11-16
-
-
Ray, K.P.1
England, P.J.2
-
88
-
-
69249100097
-
Solution structure of human cardiac troponin C in complex with the green tea polyphenol, (-)- epigallocatechin 3-gallate
-
doi: 10.1074/jbc. M109.021352
-
Robertson, I.M., Li, M.X., and Sykes, B.D. (2009). Solution structure of human cardiac troponin C in complex with the green tea polyphenol, (-)- epigallocatechin 3-gallate. J Biol chem. 284, 23012-23023. doi: 10.1074/jbc. M109.021352.
-
(2009)
J Biol chem.
, vol.284
, pp. 23012-23023
-
-
Robertson, I.M.1
Li, M.X.2
Sykes, B.D.3
-
89
-
-
0020069347
-
The effect of troponin I phosphorylation on the Ca2+-binding properties of the Ca2+-regulatory site of bovine cardiac troponin
-
Robertson, S.P., Johnson, J.D., Holroyde, M.J., Kranias, E.G., Potter, J.D., and Solaro, R.J. (1982). The effect of troponin I phosphorylation on the Ca2+-binding properties of the Ca2+-regulatory site of bovine cardiac troponin. J BiolChem 257, 260-263.
-
(1982)
J BiolChem
, vol.257
, pp. 260-263
-
-
Robertson, S.P.1
Johnson, J.D.2
Holroyde, M.J.3
Kranias, E.G.4
Potter, J.D.5
Solaro, R.J.6
-
90
-
-
62449191449
-
Single amino acid sequence polymorphisms in rat cardiac troponin revealed by top-down tandem mass spectrometry
-
Sancho Solis, R., Ge, Y., and Walker, J.W. (2009). Single amino acid sequence polymorphisms in rat cardiac troponin revealed by top-down tandem mass spectrometry. J Muscle Res Cell Motil 29, 203-212.
-
(2009)
J Muscle Res Cell Motil
, vol.29
, pp. 203-212
-
-
Sancho Solis, R.1
Ge, Y.2
Walker, J.W.3
-
91
-
-
28244469709
-
Cardiac troponin C-L29Q, related to hypertrophic cardiomyopathy, hinders the transduction of the protein kinase A dependent phosphorylation signal from cardiac troponin I to C
-
doi: lO.llll/j.1742-4658.2005.05001.x
-
Schmidtmann, A., Lindow, C., Villard, S., Heuser, A., Miigge, A., Gessner, R., Granier, C., and Jaquet, K. (2005). Cardiac troponin C-L29Q, related to hypertrophic cardiomyopathy, hinders the transduction of the protein kinase A dependent phosphorylation signal from cardiac troponin I to C. FEBSJ 111, 6087-6097. doi: lO.llll/j.1742-4658.2005.05001.x.
-
(2005)
FEBSJ
, vol.111
, pp. 6087-6097
-
-
Schmidtmann, A.1
Lindow, C.2
Villard, S.3
Heuser, A.4
Miigge, A.5
Gessner, R.6
Granier, C.7
Jaquet, K.8
-
92
-
-
84878624242
-
Perturbed Length-Dependent Activation in Human Hypertrophic Cardiomyopathy With Missense Sarcomeric Gene Mutations
-
doi: 10.1161/CIRCRESAHA.111.300436
-
Sequeira, V., Wijnker, P.J.M., Nijenkamp, L.L.M., Kuster, D.W.D., Najafi, A., Witjas-Paalberends, E.R., Regan, J.A., Boontje, N., Ten Cate, F.J., Germans, T., Carrier, L, Sadayappan, S., Van Slegtenhorst, M.A., Zaremba, R., Foster, D.B., Murphy, A.M., Poggesi, C, Dos Remedios, C, Stienen, G.J.M., Ho, C.Y., Michels, M., and Van Der Velden, J. (2013). Perturbed Length-Dependent Activation in Human Hypertrophic Cardiomyopathy With Missense Sarcomeric Gene Mutations. Circ Res 112, 1491-1505. doi: 10.1161/CIRCRESAHA.111.300436.
-
(2013)
Circ Res
, vol.112
, pp. 1491-1505
-
-
Sequeira, V.1
Wijnker, P.J.M.2
Nijenkamp, L.L.M.3
Kuster, D.W.D.4
Najafi, A.5
Witjas-Paalberends, E.R.6
Regan, J.A.7
Boontje, N.8
Ten Cate, F.J.9
Germans, T.10
Carrier, L.11
Sadayappan, S.12
Van Slegtenhorst, M.A.13
Zaremba, R.14
Foster, D.B.15
Murphy, A.M.16
Poggesi, C.17
Dos Remedios, C.18
Stienen, G.J.M.19
Ho, C.Y.20
Michels, M.21
Van Der Velden, J.22
more..
-
93
-
-
0017143827
-
Phosphorylation of troponin I and the inotropic effect of adrenaline in the perfused rabbit heart
-
Solaro, R.J., Moir, A.G.J., and Perry, S.V. (1976). Phosphorylation of troponin I and the inotropic effect of adrenaline in the perfused rabbit heart. Nature 262, 615-616.
-
(1976)
Nature
, vol.262
, pp. 615-616
-
-
Solaro, R.J.1
Moir, A.G.J.2
Perry, S.V.3
-
94
-
-
40849096222
-
The unique functions of cardiac troponin I in the control of cardiac muscle contraction and relaxation
-
doi: 10.1016/j.bbrc.2007.12.114
-
Solaro, R.J., Rosevear, P., and Kobayashi, T. (2008). The unique functions of cardiac troponin I in the control of cardiac muscle contraction and relaxation. Biochem Biophys Res Commun 369, 82-87. doi: 10.1016/j.bbrc.2007.12.114.
-
(2008)
Biochem Biophys Res Commun
, vol.369
, pp. 82-87
-
-
Solaro, R.J.1
Rosevear, P.2
Kobayashi, T.3
-
95
-
-
79960977475
-
Molecular mechanism of the Glu991ys mutation in cardiac actin (ACTC gene) that causes apical hypertrophy in man and mouse
-
doi: 10.1074/jbc. M111.252320
-
Song, W., Dyer, E., Stuckey, D., Copeland, 0., Leung, M., Bayliss, C, Messer, A.E., Wilkinson, R., Tremoleda, J., Schneider, M., Harding, S., Redwood, C, Clarke, K., Nowak, K., Monserrat, L., Wells, D., and Marston, S. (2011). Molecular mechanism of the Glu991ys mutation in cardiac actin (ACTC gene) that causes apical hypertrophy in man and mouse. Biol Chem 286, 27582-27593. doi: 10.1074/jbc. M111.252320.
-
(2011)
Biol Chem
, vol.286
, pp. 27582-27593
-
-
Song, W.1
Dyer, E.2
Stuckey, D.3
Copeland, 0.4
Leung, M.5
Bayliss, C.6
Messer, A.E.7
Wilkinson, R.8
Tremoleda, J.9
Schneider, M.10
Harding, S.11
Redwood, C.12
Clarke, K.13
Nowak, K.14
Monserrat, L.15
Wells, D.16
Marston, S.17
-
96
-
-
77955308896
-
Investigation of a transgenic mouse model of familial dilated cardiomyopathy
-
doi: 10.1016/j.yjmcc.2010.05.009
-
Song, W., Dyer, E., Stuckey, D., Leung, M.-C, Memo, M., Mansfield, C, Ferenczi, M., Liu, K., Redwood, C, Nowak, K., Harding, S., Clarke, K., Wells, D., and Marston, S. (2010). Investigation of a transgenic mouse model of familial dilated cardiomyopathy. J Mol Cell Cardiol 49, 380-389. doi: 10.1016/j.yjmcc.2010.05.009.
-
(2010)
J Mol Cell Cardiol
, vol.49
, pp. 380-389
-
-
Song, W.1
Dyer, E.2
Stuckey, D.3
Leung, M.-C.4
Memo, M.5
Mansfield, C.6
Ferenczi, M.7
Liu, K.8
Redwood, C.9
Nowak, K.10
Harding, S.11
Clarke, K.12
Wells, D.13
Marston, S.14
-
97
-
-
12444315072
-
Global quantitative phosphoprotein analysis using Multiplexed Proteomics technology
-
Steinberg, T.H., Agnew, B.J., Gee, K.R., Leung, W.Y., Goodman, T., Schulenberg, B., Hendrickson, J., Beechem, J.M., Haugland, R.P., and Patton, W.F. (2003). Global quantitative phosphoprotein analysis using Multiplexed Proteomics technology. Proteomics 3,1128-1144.
-
(2003)
Proteomics
, vol.3
, pp. 1128-1144
-
-
Steinberg, T.H.1
Agnew, B.J.2
Gee, K.R.3
Leung, W.Y.4
Goodman, T.5
Schulenberg, B.6
Hendrickson, J.7
Beechem, J.M.8
Haugland, R.P.9
Patton, W.F.10
-
98
-
-
84883277066
-
Contractile abnormalities and altered drug response in engineered heart tissue from Mybpc3-targeted knock-in mice
-
doi: 10.1016/j.yjmcc.2013.07.011
-
Stohr, A., Friedrich, F.W., Flenner, F., Geertz, B., Eder, A., Schaaf, S., Hirt, M.N., Uebeler, J., Schlossarek, S., Carrier, L, Hansen, A., and Eschenhagen, T. (2013). Contractile abnormalities and altered drug response in engineered heart tissue from Mybpc3-targeted knock-in mice. J Mol Cell Cardiol 63,189-198. doi: 10.1016/j.yjmcc.2013.07.011.
-
(2013)
J Mol Cell Cardiol
, vol.63
, pp. 189-198
-
-
Stohr, A.1
Friedrich, F.W.2
Flenner, F.3
Geertz, B.4
Eder, A.5
Schaaf, S.6
Hirt, M.N.7
Uebeler, J.8
Schlossarek, S.9
Carrier, L.10
Hansen, A.11
Eschenhagen, T.12
-
99
-
-
84860156650
-
Patient-specific induced pluripotent stem cells as a model for familial dilated cardiomyopathy
-
doi: 10.1126/scitranslmed.3003552.
-
Sun, N., Yazawa, M., Liu, J., Han, L, Sanchez-Freire, V., Abilez, O.J., Navarrete, E.G., Hu, S., Wang, L., Lee, A., Pavlovic, A., Lin, S., Chen, R., Hajjar, R.J., Snyder, M.P., Dolmetsch, R.E., Butte, M.J., Ashley, E.A., Longaker, M.T., Robbins, R.C., and Wu, J.C. (2012). Patient-specific induced pluripotent stem cells as a model for familial dilated cardiomyopathy. Sci TranslMed4,130ral47. doi: 10.1126/scitranslmed.3003552.
-
(2012)
Sci TranslMed
, vol.4
, pp. 130-147
-
-
Sun, N.1
Yazawa, M.2
Liu, J.3
Han, L.4
Sanchez-Freire, V.5
Abilez, O.J.6
Navarrete, E.G.7
Hu, S.8
Wang, L.9
Lee, A.10
Pavlovic, A.11
Lin, S.12
Chen, R.13
Hajjar, R.J.14
Snyder, M.P.15
Dolmetsch, R.E.16
Butte, M.J.17
Ashley, E.A.18
Longaker, M.T.19
Robbins, R.C.20
Wu, J.C.21
more..
-
100
-
-
77958524192
-
Biological actions of green tea catechins on cardiac troponin C
-
doi: 10.1111/j.1476-5381.2010.00942.x
-
Tadano, N., Du, C, Yumoto, F., Morimoto, S., Ohta, M., Xie, M., Nagata, K., Zhan, D., Lu, Q., Miwa, Y., Takahashi-Yanaga, F., Tanokura, M., Ohtsuki, I., and Sasaguri, T. (2010). Biological actions of green tea catechins on cardiac troponin C. Brit J Pharmacol 161,1034-1043. doi: 10.1111/j.1476-5381.2010.00942.x.
-
(2010)
Brit J Pharmacol
, vol.161
, pp. 1034-1043
-
-
Tadano, N.1
Du, C.2
Yumoto, F.3
Morimoto, S.4
Ohta, M.5
Xie, M.6
Nagata, K.7
Zhan, D.8
Lu, Q.9
Miwa, Y.10
Takahashi-Yanaga, F.11
Tanokura, M.12
Ohtsuki, I.13
Sasaguri, T.14
-
101
-
-
0037588762
-
Structure of the core domain of human cardiac troponin in the Ca2+-saturated form
-
Takeda, N., Yamashita, A., Maeda, K., and Maeda, Y. (2003). Structure of the core domain of human cardiac troponin in the Ca2+-saturated form. Nature 424, 35-41.
-
(2003)
Nature
, vol.424
, pp. 35-41
-
-
Takeda, N.1
Yamashita, A.2
Maeda, K.3
Maeda, Y.4
-
102
-
-
0037216694
-
Increased Ca2+-sensitivity of the contractile apparatus in end-stage human heart failure results from altered phosphorylation of contractile proteins
-
Van Der Velden, J., Papp, Z., Zaremba, R., Boontje, N.M., De Jong, J.W., Owen, V.J., Burton, P.B., Goldmann, P., Jaquet, K., and Stienen, G.J. (2003). Increased Ca2+-sensitivity of the contractile apparatus in end-stage human heart failure results from altered phosphorylation of contractile proteins. Cardiovasc Res 57, 37-47.
-
(2003)
Cardiovasc Res
, vol.57
, pp. 37-47
-
-
Van Der Velden, J.1
Papp, Z.2
Zaremba, R.3
Boontje, N.M.4
De Jong, J.W.5
Owen, V.J.6
Burton, P.B.7
Goldmann, P.8
Jaquet, K.9
Stienen, G.J.10
-
103
-
-
84906535306
-
Modulation of cardiac myofibril Ca2+- sensitivity by phosphorylation, sarcomere length and a DCM-causing mutation
-
in review.
-
Vikhorev, Song, W., Wilkinson, R., Copeland, O.A.N., Messer, A.E., Ferenczi, M.A., and Marston, S.B. (2013). Modulation of cardiac myofibril Ca2+- sensitivity by phosphorylation, sarcomere length and a DCM-causing mutation. BiophysJ in review.
-
(2013)
Biophys J
-
-
Vikhorev Song, W.1
Wilkinson, R.2
Copeland, O.A.N.3
Messer, A.E.4
Ferenczi, M.A.5
Marston, S.B.6
-
104
-
-
84862910102
-
The generation and functional characterization of knock in mice harboring the cardiac Troponin IR21C mutation associated with hypertrophic cardiomyopathy
-
doi: 10.1074/jbc. M111.294306.
-
Wang, Y., Pinto, J., Sancho Solis, R., Dweck, D., Liang, J., Diaz-Perez, Z., Ge, Y., Walker, J., and Potter, J. (2011). The generation and functional characterization of knock in mice harboring the cardiac Troponin IR21C mutation associated with hypertrophic cardiomyopathy. J Mol Chem 287, 2156-2167. doi: 10.1074/jbc. M111.294306.
-
(2011)
J Mol Chem
, vol.287
, pp. 2156-2167
-
-
Wang, Y.1
Pinto, J.2
Sancho Solis, R.3
Dweck, D.4
Liang, J.5
Diaz-Perez, Z.6
Ge, Y.7
Walker, J.8
Potter, J.9
-
105
-
-
84862910102
-
Generation and functional characterization of knock-in mice harboring the cardiac troponin IR21C mutation associated with hypertrophic cardiomyopathy
-
doi: 10.1074/jbc. M111.294306.
-
Wang, Y., Pinto, J.R., Solis, R.S., Dweck, D., Liang, J., Diaz-Perez, Z., Ge, Y., Walker, J.W., and Potter, J.D. (2012). Generation and functional characterization of knock-in mice harboring the cardiac troponin IR21C mutation associated with hypertrophic cardiomyopathy. J Mol Chem 287, 2156-2167. doi: 10.1074/jbc. M111.294306.
-
(2012)
J Mol Chem
, vol.287
, pp. 2156-2167
-
-
Wang, Y.1
Pinto, J.R.2
Solis, R.S.3
Dweck, D.4
Liang, J.5
Diaz-Perez, Z.6
Ge, Y.7
Walker, J.W.8
Potter, J.D.9
-
106
-
-
1842434499
-
Characterization of the interaction between the N-terminal extension of human cardiac troponin I and troponin C
-
doi: 10.1021/bi0361281
-
Ward, D.G., Brewer, S.M., Calvert, M.J., Gallon, C.E., Gao, Y., and Trayer, I.P. (2004a). Characterization of the interaction between the N-terminal extension of human cardiac troponin I and troponin C. Biochemistry 43, 4020-4027. doi: 10.1021/bi0361281.
-
(2004)
Biochemistry
, vol.43
, pp. 4020-4027
-
-
Ward, D.G.1
Brewer, S.M.2
Calvert, M.J.3
Gallon, C.E.4
Gao, Y.5
Trayer, I.P.6
-
107
-
-
2442429819
-
NMR and mutagenesis studies on the phosphorylation region of human cardiac troponin I
-
doi: 10.1021/bi036310m
-
Ward, D.G., Brewer, S.M., Gallon, C.E., Gao, Y., Levine, B.A., and Trayer, I.P. (2004b). NMR and mutagenesis studies on the phosphorylation region of human cardiac troponin I. Biochemistry 43, 5772-5781. doi: 10.1021/bi036310m.
-
(2004)
Biochemistry
, vol.43
, pp. 5772-5781
-
-
Ward, D.G.1
Brewer, S.M.2
Gallon, C.E.3
Gao, Y.4
Levine, B.A.5
Trayer, I.P.6
-
108
-
-
84906567299
-
An investigation into the disease causing mechanism in familial dilated cardiomyopathy
-
PhD, Imperial College.
-
Wilkinson, R. (2014). An investigation into the disease causing mechanism in familial dilated cardiomyopathy. PhD, Imperial College.
-
(2014)
-
-
Wilkinson, R.1
-
109
-
-
0028929818
-
Calcium sensitivity of isometric tension is increased in canine experimental heart failure
-
Wolff, M.R., Whitesell, L.F., and Moss, R.L. (1995). Calcium sensitivity of isometric tension is increased in canine experimental heart failure. CircRes 76, 781-789.
-
(1995)
CircRes
, vol.76
, pp. 781-789
-
-
Wolff, M.R.1
Whitesell, L.F.2
Moss, R.L.3
-
110
-
-
34547951704
-
Cardiac transgenic and gene transfer strategies converge to support an important role for troponin I in regulating relaxation in cardiac myocytes
-
doi: 10.1161/CIRCRESAHA.106.145557
-
Yasuda, S., Coutu, P., Sadayappan, S., Robbins, J., and Metzger, J.M. (2007). Cardiac transgenic and gene transfer strategies converge to support an important role for troponin I in regulating relaxation in cardiac myocytes. CircRes 101, 377-386. doi: 10.1161/CIRCRESAHA.106.145557.
-
(2007)
CircRes
, vol.101
, pp. 377-386
-
-
Yasuda, S.1
Coutu, P.2
Sadayappan, S.3
Robbins, J.4
Metzger, J.M.5
-
111
-
-
55049123817
-
Unraveling molecular complexity of phosphorylated human cardiac troponin I by top down electron capture dissociation/electron transfer dissociation mass spectrometry
-
Zabrouskov, V., Ge, Y., Schwartz, J., and Walker, J.W. (2008). Unraveling molecular complexity of phosphorylated human cardiac troponin I by top down electron capture dissociation/electron transfer dissociation mass spectrometry. Mol Cell Proteomics 7,1838-1849.
-
(2008)
Mol Cell Proteomics
, vol.7
, pp. 1838-1849
-
-
Zabrouskov, V.1
Ge, Y.2
Schwartz, J.3
Walker, J.W.4
-
112
-
-
38349188639
-
Quantitative analysis of myofilament protein phosphorylation in small cardiac biopsies
-
Zaremba, R., Merkus, D., Hamdani, N., Lamers, J., Paulus, W., Dos Remedios, C, Duncker, D., Stienen, G., and Van Der Velden, J. (2007). Quantitative analysis of myofilament protein phosphorylation in small cardiac biopsies. Proteomics ClinAppl 1,1285-1290.
-
(2007)
Proteomics ClinAppl
, vol.1
, pp. 1285-1290
-
-
Zaremba, R.1
Merkus, D.2
Hamdani, N.3
Lamers, J.4
Paulus, W.5
Dos Remedios, C.6
Duncker, D.7
Stienen, G.8
Van Der Velden, J.9
-
113
-
-
0029037870
-
Cardiac troponin I phosphorylation increases the rate of cardiac muscle relaxation
-
Zhang, R., Zhao, J., Mandveno, A., and Potter, J.D. (1995). Cardiac troponin I phosphorylation increases the rate of cardiac muscle relaxation. BiophysJ 76,1028-1035.
-
(1995)
Biophys J
, vol.76
, pp. 1028-1035
-
-
Zhang, R.1
Zhao, J.2
Mandveno, A.3
Potter, J.D.4
|