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Volumn 119, Issue 2, 2007, Pages 245-248

Abnormal cardiac response to exercise in a murine model of familial hypertrophic cardiomyopathy

Author keywords

Cardiomyopathy; Exercise; Hypertrophy; Troponin I gene

Indexed keywords

GLYCINE; SERINE;

EID: 34249274852     PISSN: 01675273     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.ijcard.2006.09.001     Document Type: Article
Times cited : (8)

References (10)
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    • Genetic causes of human heart failure
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  • 5
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    • Lehnart S.E., Wehrens X.H., and Marks A.R. Calstabin deficiency, ryanodine receptors, and sudden cardiac death. Biochem Biophys Res Commun 322 (2004) 1267-1279
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  • 7
    • 20844448376 scopus 로고    scopus 로고
    • Cardiac troponin I mutations in Australian families with hypertrophic cardiomyopathy: clinical, genetic and functional consequences
    • Doolan A., Tebo M., Ingles J., et al. Cardiac troponin I mutations in Australian families with hypertrophic cardiomyopathy: clinical, genetic and functional consequences. J Mol Cell Cardiol 38 (2005) 387-393
    • (2005) J Mol Cell Cardiol , vol.38 , pp. 387-393
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  • 8
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    • Molecular insights from a novel cardiac troponin I mouse model of familial hypertrophic cardiomyopathy
    • Tsoutsman T., Chung J., Doolan A., et al. Molecular insights from a novel cardiac troponin I mouse model of familial hypertrophic cardiomyopathy. J Mol Cell Cardiol 41 (2006) 623-632
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    • The L-type calcium-channel inhibitor diltiazem prevents cardiomyopathy in a mouse model
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    • Consequences of pressure overload in a murine model of hypertrophic cardiomyopathy
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* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.