Baicalin ameliorates H2O2 induced cytotoxicity in HK-2 cells through the inhibition of ER stress and the activation of Nrf2 signaling

International Journal of Molecular Sciences

Volumn 15, Issue 7, 2014, Pages 12507-12522

  Lin, Miao ^a   Li, Long ^b   Zhang, Yi ^b   Zheng, Long ^b   Xu, Ming ^a   Rong, Ruiming ^a   Zhu, Tongyu ^b  

^a ZHONGSHAN HOSPITAL OF FUDAN UNIVERSITY   (China)

^b Shanghai Key Laboratory of Organ Transplantation   (China)

Author keywords

Baicalin; ER stress; Ischemia reperfusion injury; Kidney; Nrf2; Tubular epithelial cells

Indexed keywords

BAICALIN; CASPASE 3; GLUTATHIONE; HYDROGEN PEROXIDE; REACTIVE OXYGEN METABOLITE; TRANSCRIPTION FACTOR NRF2; ANTIOXIDANT; FLAVONOID; NFE2L2 PROTEIN, HUMAN;

APOPTOSIS; ARTICLE; CELL VIABILITY; CONTROLLED STUDY; CYTOTOXICITY; ENDOPLASMIC RETICULUM STRESS; HUMAN; HUMAN CELL; IMMUNOCYTOCHEMISTRY; NICK END LABELING; OXIDATIVE STRESS; PROTEIN EXPRESSION; REAL TIME POLYMERASE CHAIN REACTION; RECEPTOR UPREGULATION; SIGNAL TRANSDUCTION; WESTERN BLOTTING; CELL LINE; DRUG EFFECTS; EPITHELIUM CELL; GENETICS; METABOLISM;

ANTIOXIDANTS; CELL LINE; ENDOPLASMIC RETICULUM STRESS; EPITHELIAL CELLS; FLAVONOIDS; HUMANS; HYDROGEN PEROXIDE; NF-E2-RELATED FACTOR 2; OXIDATIVE STRESS; REACTIVE OXYGEN SPECIES; SIGNAL TRANSDUCTION;

EID: 84904431236     PISSN: 16616596     EISSN: 14220067     Source Type: Journal    
DOI: 10.3390/ijms150712507     Document Type: Article

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