SET translocation is associated with increase in caspase cleaved amyloid precursor protein in CA1 of Alzheimer and Down syndrome patients

Neurobiology of Aging

Volumn 35, Issue 5, 2014, Pages 958-968

  Facchinetti, Patricia ^a   Dorard, Emilie ^a   Contremoulins, Vincent ^b   Gaillard, Marie Claude ^c   Déglon, Nicole ^c   Sazdovitch, Véronique ^d   Guihenneuc Jouyaux, Chantal ^e   Brouillet, Emmanuel ^c   Duyckaerts, Charles ^d   Allinquant, Bernadette ^a  

^a INSERM   (France)

^b INSTITUT JACQUES MONOD   (France)

^c INSTITUT DE RADIOPROTECTION ET DE SÛRETÉ NUCLÉAIRE   (France)

^d PITIÉ SALPÊTRIÈRE HOSPITAL   (France)

^e AP HP   (France)

Author keywords

Alzheimer disease; Caspase cleaved APP; Down syndrome; Hippocampus; SET translocation; Tau hyperphosphorylation

Indexed keywords

ALZHEIMER DISEASE; CASPASE CLEAVED APP; DOWN SYNDROME; HIPPOCAMPUS; SET TRANSLOCATION; TAU HYPERPHOSPHORYLATION;

AGED; AGED, 80 AND OVER; ALZHEIMER DISEASE; AMYLOID BETA-PROTEIN PRECURSOR; ANIMALS; CA1 REGION, HIPPOCAMPAL; CASPASES; CYTOPLASM; DENTATE GYRUS; DISEASE PROGRESSION; DOWN SYNDROME; FEMALE; HISTONE CHAPERONES; HUMANS; MALE; MICE; MICE, INBRED C57BL; MIDDLE AGED; NEURONS; PHOSPHORYLATION; TAU PROTEINS; TRANSCRIPTION FACTORS;

EID: 84893772344     PISSN: 01974580     EISSN: 15581497     Source Type: Journal    
DOI: 10.1016/j.neurobiolaging.2013.08.039     Document Type: Article

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