The genetic component of brugada syndrome

Frontiers in Physiology

Volumn 4 JUL, Issue , 2013, Pages

  Nielsen, Morten W ^a,b   Holst, Anders G ^a,b   Olesen, Søren Peter ^a   Olesen, Morten S ^a,b  

^a DANISH NATIONAL RESEARCH FOUNDATION   (Denmark)

^b UNIVERSITY OF COPENHAGEN   (Denmark)

Author keywords

Brugada syndrome; Exome sequencing project; Genetics; Mutation; Treatment

Indexed keywords

DIHYDROQUINIDINE; QUINIDINE;

BRUGADA SYNDROME; CACNA1C GENE; CACNA2D1 GENE; CACNB2B GENE; CLINICAL EFFECTIVENESS; DRUG EFFICACY; DRUG MECHANISM; DRUG RESPONSE; DRUG TOLERABILITY; DRUG WITHDRAWAL; GASTROINTESTINAL DISEASE; GENE; GENE EXPRESSION; GENE FREQUENCY; GENE FUNCTION; GENE MUTATION; GENETIC VARIABILITY; GENOTYPE; GPD1L GENE; HCN4 GENE; HEART ATRIUM FIBRILLATION; HEART VENTRICLE TACHYCARDIA; HETEROZYGOSITY; HUMAN; IMPLANTABLE CARDIOVERTER DEFIBRILLATOR; KCND3 GENE; KCNE3 GENE; KCNE5 GENE; KCNH2 GENE; KCNJ8 GENE; LOW DRUG DOSE; MISSENSE MUTATION; NONHUMAN; PATHOGENESIS; PHENOTYPE; QT PROLONGATION; RANGRF GENE; REVIEW; SCN1B GENE; SCN2B GENE; SCN3B GENE; SCN5A GENE; SLMAP GENE; TREATMENT INDICATION; TRPM4 GENE;

EID: 84884546937     PISSN: None     EISSN: 1664042X     Source Type: Journal    
DOI: 10.3389/fphys.2013.00179     Document Type: Review

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