Hepatic ERK activity plays a role in energy metabolism

Molecular and Cellular Endocrinology

Volumn 375, Issue 1-2, 2013, Pages 157-166

  Jiao, Ping ^a,b   Feng, Bin ^a   Li, Yujie ^a   He, Qin ^a   Xu, Haiyan ^a  

^a BROWN UNIVERSITY   (United States)

^b JILIN UNIVERSITY   (China)

Author keywords

Gluconeogenesis; MAPK; Obesity

Indexed keywords

FATTY ACID; GLUCOSE; GLYCOGEN; GREEN FLUORESCENT PROTEIN; MITOGEN ACTIVATED PROTEIN KINASE; MITOGEN ACTIVATED PROTEIN KINASE 1; TRANSCRIPTION FACTOR FKHR;

ADIPOCYTE; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; ENERGY EXPENDITURE; ENERGY METABOLISM; ENZYME ACTIVITY; ENZYME PHOSPHORYLATION; FATTY ACID BLOOD LEVEL; FATTY ACID OXIDATION; G6PASE GENE; GENE EXPRESSION; GENE OVEREXPRESSION; GENE SILENCING; GLYCOGEN LIVER LEVEL; HYPERGLYCEMIA; INSULIN RESISTANCE; LIPOLYSIS; LIVER METABOLISM; MALE; MEK CA GENE; MOUSE; NONHUMAN; OBESITY; PRIORITY JOURNAL;

ANIMALS; BLOOD GLUCOSE; DIET, HIGH-FAT; ENERGY METABOLISM; ENZYME ACTIVATION; GENE KNOCKDOWN TECHNIQUES; GLUCOSE; HEK293 CELLS; HOMEOSTASIS; HUMANS; INSULIN RESISTANCE; LIPID METABOLISM; LIVER; MALE; MAP KINASE SIGNALING SYSTEM; MICE; MICE, INBRED C57BL; MICE, OBESE; MITOGEN-ACTIVATED PROTEIN KINASE 1; MITOGEN-ACTIVATED PROTEIN KINASE 3; OBESITY; ORGAN SIZE; PHOSPHORYLATION; PROTEIN PROCESSING, POST-TRANSLATIONAL; RNA, SMALL INTERFERING; TRIGLYCERIDES;

EID: 84879559745     PISSN: 03037207     EISSN: 18728057     Source Type: Journal    
DOI: 10.1016/j.mce.2013.05.021     Document Type: Article

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