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1
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79952517839
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Duodenal chemosensing and mucosal defenses
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Akiba Y, Kaunitz JD. Duodenal chemosensing and mucosal defenses. Digestion 2011;83(Suppl 1):25-31.
-
(2011)
Digestion
, vol.83
, Issue.1 SUPPL.
, pp. 25-31
-
-
Akiba, Y.1
Kaunitz, J.D.2
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2
-
-
77958470623
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Gastroduodenal mucosal defense
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This study reviewed various mechanisms involved in gastroduodenal mucosal defense
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DeFoneska A, Kaunitz JD. Gastroduodenal mucosal defense. Curr Opin Gastroenterol 2010;26:604-610. This study reviewed various mechanisms involved in gastroduodenal mucosal defense.
-
(2010)
Curr Opin Gastroenterol
, vol.26
, pp. 604-610
-
-
DeFoneska, A.1
Kaunitz, J.D.2
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3
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0035246513
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Cyclooxygenase inhibitors - Current status and future prospects
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DOI 10.1016/S0223-5234(01)01197-7, PII S0223523401011977
-
Dannhardt G, Kiefer W. Cyclooxygenase inhibitors: current status and future prospects. Eur J Med Chem 2001;36:109-126. (Pubitemid 32304514)
-
(2001)
European Journal of Medicinal Chemistry
, vol.36
, Issue.2
, pp. 109-126
-
-
Dannhardt, G.1
Kiefer, W.2
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6
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46049093776
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Gastric Mucosal Defense and Cytoprotection: Bench to Bedside
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DOI 10.1053/j.gastro.2008.05.030, PII S0016508508007944
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Laine L, Takeuchi K, Tarnawski A. Gastric mucosal defense and cytoprotection: bench to bedside. Gastroenterology 2008;135:41-60. (Pubitemid 351899027)
-
(2008)
Gastroenterology
, vol.135
, Issue.1
, pp. 41-60
-
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Laine, L.1
Takeuchi, K.2
Tarnawski, A.3
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8
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79951671436
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Microsomal prostaglandin E2 synthase-1 modulates the response to vascular injury
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Wang M, Ihida-Stansbury K, Kothapalli D, et al. Microsomal prostaglandin E2 synthase-1 modulates the response to vascular injury. Circulation 2011;123:631-639.
-
(2011)
Circulation
, vol.123
, pp. 631-639
-
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Wang, M.1
Ihida-Stansbury, K.2
Kothapalli, D.3
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9
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79951853626
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Regulatory mechanism of duodenal bicarbonate secretion: Roles of endogenous prostaglandins and nitric oxide
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Takeuchi K, Kita K, Hayashi S, Aihara E. Regulatory mechanism of duodenal bicarbonate secretion: roles of endogenous prostaglandins and nitric oxide. Pharmacol Ther 2011;130:59-70.
-
(2011)
Pharmacol Ther
, vol.130
, pp. 59-70
-
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Takeuchi, K.1
Kita, K.2
Hayashi, S.3
Aihara, E.4
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10
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78149481801
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Role of microsomal prostaglandin E synthase-1 in the facilitation of angiogenesis and the healing of gastric ulcers
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authors studied the role of mPGES-1 on the enhancement of the ulcer healing process. The authors found that mPGES-1 is upregulated in ulcer tissue and that it plays an important role in the acceleration of angiogenesis and healing
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Ae T, Ohno T, Hattori Y, et al. Role of microsomal prostaglandin E synthase-1 in the facilitation of angiogenesis and the healing of gastric ulcers. Am J Physiol Gastrointest Liver Physiol 2010;299:G1139-G1146. The authors studied the role of mPGES-1 on the enhancement of the ulcer healing process. The authors found that mPGES-1 is upregulated in ulcer tissue and that it plays an important role in the acceleration of angiogenesis and healing.
-
(2010)
Am J Physiol Gastrointest Liver Physiol
, vol.299
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Ae, T.1
Ohno, T.2
Hattori, Y.3
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11
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79952763798
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Muc-2-deficient mice display a sex-specific, COX-2-related impairment of gastric mucosal repair
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authors studied the influences of Muc-2 on the susceptibility to and healing of gastric injury in the mouse model. The authors found a significant slowing of mucosal healing in Muc-2-deficient mice, more so in males than in females
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Wallace JL, Vong L, Dharmani P, et al. Muc-2-deficient mice display a sex-specific, COX-2-related impairment of gastric mucosal repair. Am J Pathol 2011;178:1126-1133. The authors studied the influences of Muc-2 on the susceptibility to and healing of gastric injury in the mouse model. The authors found a significant slowing of mucosal healing in Muc-2-deficient mice, more so in males than in females.
-
(2011)
Am J Pathol
, vol.178
, pp. 1126-1133
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Wallace, J.L.1
Vong, L.2
Dharmani, P.3
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12
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79955596210
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Sulindac sulfide induces autophagic death in gastric epithelial cells via survivin down-regulation: A mechanism of NSAIDs-induced gastric injury
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authors studied the effects of sulindac sulfide on the autophagic death in gastric epithelium and the role of survivin on gastric damage. The authors concluded that physiological concentrations of sulindac sulfide induce autophagy in gastric epithelial cells and that the downregulation of survivin is involved in the mechanism
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Chiou SK, Hoa N, Hodges A. Sulindac sulfide induces autophagic death in gastric epithelial cells via survivin down-regulation: a mechanism of NSAIDs-induced gastric injury. Biochem Pharmacol 2011;81:1317-1323. The authors studied the effects of sulindac sulfide on the autophagic death in gastric epithelium and the role of survivin on gastric damage. The authors concluded that physiological concentrations of sulindac sulfide induce autophagy in gastric epithelial cells and that the downregulation of survivin is involved in the mechanism.
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(2011)
Biochem Pharmacol
, vol.81
, pp. 1317-1323
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Chiou, S.K.1
Hoa, N.2
Hodges, A.3
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13
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77950367134
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In vivo evidence for the role of RegI in gastric regeneration: Transgenic overexpression of RegI accelerates the healing of experimental gastric ulcers
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authors studied RegI-transgenic mice to examine the role of RegI protein in the healing of gastric ulcers. The authors concluded that RegI contributes to healing during the recovery phase by stimulating progenitor cell growth
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Fukuhara H, Kadowaki Y, Ose T, et al. In vivo evidence for the role of RegI in gastric regeneration: transgenic overexpression of RegI accelerates the healing of experimental gastric ulcers. Lab Invest 2010;90:556-565. The authors studied RegI-transgenic mice to examine the role of RegI protein in the healing of gastric ulcers. The authors concluded that RegI contributes to healing during the recovery phase by stimulating progenitor cell growth.
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(2010)
Lab Invest
, vol.90
, pp. 556-565
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Fukuhara, H.1
Kadowaki, Y.2
Ose, T.3
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14
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29344457901
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TFF (trefoil factor family) peptide-triggered signals promoting mucosal restitution
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DOI 10.1007/s00018-005-5481-9
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Hoffmann W. Trefoil factors TFF (trefoil factor family) peptide-triggered signals promoting mucosal restitution. Cell Mol Life Sci 2005;62:2932-2938. (Pubitemid 43004746)
-
(2005)
Cellular and Molecular Life Sciences
, vol.62
, Issue.24
, pp. 2932-2938
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Hoffmann, W.1
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15
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77956100430
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In vivo action of trefoil factor 2 (TFF2) to speed gastric repair is independent of cyclooxygenase
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authors studied the role of TFFs in epithelial restitution. The authors found that TFF2 is the fastest acting TFF in the stomach, taking effect through a mechanism that does not require COX activation
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Xue L, Aihara E, Podolsky DK, et al. In vivo action of trefoil factor 2 (TFF2) to speed gastric repair is independent of cyclooxygenase. Gut 2010;59:1184-1191. The authors studied the role of TFFs in epithelial restitution. The authors found that TFF2 is the fastest acting TFF in the stomach, taking effect through a mechanism that does not require COX activation.
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(2010)
Gut
, vol.59
, pp. 1184-1191
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Xue, L.1
Aihara, E.2
Podolsky, D.K.3
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16
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77956244664
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Suppression of growth arrest and DNAdamageinducible 45a expression confers resistance to sulindac and indomethacininduced gastric mucosal injury
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Chiou SK, Hodges A, Hoa N. Suppression of growth arrest and DNAdamageinducible 45a expression confers resistance to sulindac and indomethacininduced gastric mucosal injury. J Pharmacol Exp Ther 2010;334:693-702.
-
(2010)
J Pharmacol Exp Ther
, vol.334
, pp. 693-702
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Chiou, S.K.1
Hodges, A.2
Hoa, N.3
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17
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33846082831
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Oxidative stress involvement and gene expression in indomethacin-induced gastropathy
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DOI 10.1179/135100006X155021
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Naito Y, Yoshikawa T. Oxidative stress involvement and gene expression in indomethacin-induced gastropathy. Redox Rep 2006;11:243-253. (Pubitemid 46062349)
-
(2006)
Redox Report
, vol.11
, Issue.6
, pp. 243-253
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Naito, Y.1
Yoshikawa, T.2
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18
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77951447882
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Effect of indomethacin on bile acid-phospholipid interactions: Implication for small intestinal injury induced by nonsteroidal anti-inflammatory drugs
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authors studied the increased toxicity of the combination of NSAIDs and bile. The authors found that when combined, NSAIDs and bile have altered physiochemical properties that allow them to interact more vigorously with cell membranes and thus enhance their disruptive effect
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Zhou Y, Dial EJ, Doyen R, Lichtenberger LM. Effect of indomethacin on bile acid-phospholipid interactions: implication for small intestinal injury induced by nonsteroidal anti-inflammatory drugs. Am J Physiol Gastrointest Liver Physiol 2010;298:G722-G731. The authors studied the increased toxicity of the combination of NSAIDs and bile. The authors found that when combined, NSAIDs and bile have altered physiochemical properties that allow them to interact more vigorously with cell membranes and thus enhance their disruptive effect.
-
(2010)
Am J Physiol Gastrointest Liver Physiol
, vol.298
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Zhou, Y.1
Dial, E.J.2
Doyen, R.3
Lichtenberger, L.M.4
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19
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79952029153
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Effects of lansoprazole on the expression of VEGF and cellular proliferation in a rat model of acetic acidinduced gastric ulcer
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authors studied the effects of lansoprazole on epithelial restitution. The authors found that lansoprazole increased the expression of VEGF and MMP-2 at the ulcer margin in a dose-dependent manner
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Kobayashi S, Nakajima N, Ito Y, Moriyama M. Effects of lansoprazole on the expression of VEGF and cellular proliferation in a rat model of acetic acidinduced gastric ulcer. J Gastroenterol 2010;45:846-858. The authors studied the effects of lansoprazole on epithelial restitution. The authors found that lansoprazole increased the expression of VEGF and MMP-2 at the ulcer margin in a dose-dependent manner.
-
(2010)
J Gastroenterol
, vol.45
, pp. 846-858
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Kobayashi, S.1
Nakajima, N.2
Ito, Y.3
Moriyama, M.4
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20
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0029969571
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Capsaicin-sensitive sensory neurons are involved in bicarbonate secretion induced by lansoprazole, a proton pump inhibitor, in rats
-
DOI 10.1007/BF02213135
-
Inada I, Satoh H. Capsaicin-sensitive sensory neurons are involved in bicarbonate secretion induced by lansoprazole, a proton pump inhibitor, in rats. Dig Dis Sci 1996;41:785-790. (Pubitemid 26140082)
-
(1996)
Digestive Diseases and Sciences
, vol.41
, Issue.4
, pp. 785-790
-
-
Inada, I.1
Satoh, H.2
-
22
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36549085160
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Control of matrix metalloproteinase catalytic activity
-
DOI 10.1016/j.matbio.2007.07.001, PII S0945053X0700090X
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Ra HJ, Parks WC. Control of matrix metalloproteinase catalytic activity. Matrix Biol 2007;26:587-596. (Pubitemid 350180394)
-
(2007)
Matrix Biology
, vol.26
, Issue.8
, pp. 587-596
-
-
Ra, H.-J.1
Parks, W.C.2
-
23
-
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43049092274
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Metalloproteinases and their inhibitors: Regulators of wound healing
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Gill SE, Parks WC. Metalloproteinases and their inhibitors: regulators of wound healing. Int J Biochem Cell Biol 2008;40:1334-1347.
-
(2008)
Int J Biochem Cell Biol
, vol.40
, pp. 1334-1347
-
-
Gill, S.E.1
Parks, W.C.2
-
24
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33646853281
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Beyond gastric acid reduction: Proton pump inhibitors induce heme oxygenase-1 in gastric and endothelial cells
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DOI 10.1016/j.bbrc.2006.04.170, PII S0006291X0601014X
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Becker JC, Grosser N, Waltke C, et al. Beyond gastric acid reduction: proton pump inhibitors induce heme oxygenase-1 in gastric and endothelial cells. Biochem Biophys Res Commun 2006;345:1014-1021. (Pubitemid 43776514)
-
(2006)
Biochemical and Biophysical Research Communications
, vol.345
, Issue.3
, pp. 1014-1021
-
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Becker, J.C.1
Grosser, N.2
Waltke, C.3
Schulz, S.4
Erdmann, K.5
Domschke, W.6
Schroder, H.7
Pohle, T.8
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25
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0029064621
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Effects of lansoprazole on ethanolinduced injury and PG synthetic activity in rat gastric mucosa
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Fukuda T, Arakawa T, Shimizu Y, et al. Effects of lansoprazole on ethanolinduced injury and PG synthetic activity in rat gastric mucosa. J Clin Gastroenterol 1995;20(Suppl 2):S5-S7.
-
(1995)
J Clin Gastroenterol
, vol.20
, Issue.2 SUPPL.
-
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Fukuda, T.1
Arakawa, T.2
Shimizu, Y.3
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26
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79151479663
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Effects of esomeprazole on healing of nonsteroidal anti-inflammatory drug (NSAID)-induced gastric ulcers in the presence of a continued NSAID treatment: Characterization of molecular mechanisms
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This study reported that PPIs promote ulcer healing through acid-dependent and acid-independent pathways. In addition to inhibiting acid secretion, esomeprazole exhibits antioxidant and antiapoptotic effects
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Fornai M, Colucci R, Antonioli L, et al. Effects of esomeprazole on healing of nonsteroidal anti-inflammatory drug (NSAID)-induced gastric ulcers in the presence of a continued NSAID treatment: characterization of molecular mechanisms. Pharmacol Res 2011;63:59-67. This study reported that PPIs promote ulcer healing through acid-dependent and acid-independent pathways. In addition to inhibiting acid secretion, esomeprazole exhibits antioxidant and antiapoptotic effects.
-
(2011)
Pharmacol Res
, vol.63
, pp. 59-67
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Fornai, M.1
Colucci, R.2
Antonioli, L.3
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27
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77951529197
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Protective effect of roxatidine against indomethacin-induced small intestinal mucosal injury in rats
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The authors studied the influence of roxatidine on gastric mucus secretion in indomethacin-treated rats. The authors concluded that roxatidine lessens gastric injury through a mechanism involving nitric oxide, rather than prostaglandins
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Umegaki E, Yoda Y, Tokioka S, et al. Protective effect of roxatidine against indomethacin-induced small intestinal mucosal injury in rats. J Gastroenterol Hepatol 2010;25(Suppl 1):S35-S40. The authors studied the influence of roxatidine on gastric mucus secretion in indomethacin-treated rats. The authors concluded that roxatidine lessens gastric injury through a mechanism involving nitric oxide, rather than prostaglandins.
-
(2010)
J Gastroenterol Hepatol
, vol.25
, Issue.1 SUPPL.
-
-
Umegaki, E.1
Yoda, Y.2
Tokioka, S.3
-
28
-
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0036798932
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Gastrointestinal melatonin: Localization, function, and clinical relevance
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DOI 10.1023/A:1020107915919
-
Bubenik GA. Gastrointestinal melatonin: localization, function, and clinical relevance. Dig Dis Sci 2002;47:2336-2348. (Pubitemid 35205309)
-
(2002)
Digestive Diseases and Sciences
, vol.47
, Issue.10
, pp. 2336-2348
-
-
Bubenik, G.A.1
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29
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77950630864
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Role of melatonin in mucosal gastroprotection against aspirin-induced gastric lesions in humans
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The authors studied the effects of melatonin and L-tryptophan on gastric microbleeding, PGE2 secretion, plasma melatonin, and gastrin levels in aspirin-induced gastric injury in rats. The authors concluded that melatonin and tryptophan decrease gastric damage through the protective action of exogenous melatonin or that generated from tryptophan and through plasma gastrin released from the mucosa
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Konturek PC, Konturek SJ, Celinski K, et al. Role of melatonin in mucosal gastroprotection against aspirin-induced gastric lesions in humans. J Pineal Res 2010;48:318-323. The authors studied the effects of melatonin and L-tryptophan on gastric microbleeding, PGE2 secretion, plasma melatonin, and gastrin levels in aspirin-induced gastric injury in rats. The authors concluded that melatonin and tryptophan decrease gastric damage through the protective action of exogenous melatonin or that generated from tryptophan and through plasma gastrin released from the mucosa.
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(2010)
J Pineal Res
, vol.48
, pp. 318-323
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Konturek, P.C.1
Konturek, S.J.2
Celinski, K.3
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30
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77955159826
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Melatonin promotes angio-genesis during protection and healing of indomethacin-induced gastric ulcer: Role of matrix metaloproteinase-2
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The authors studied the gastroprotective role of melatonin via the upregulation of VEGF and MMP-2 expression
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Ganguly K, Sharma AV, Reiter RJ, Swarnakar S. Melatonin promotes angio-genesis during protection and healing of indomethacin-induced gastric ulcer: role of matrix metaloproteinase-2. J Pineal Res 2010;49:130-140. The authors studied the gastroprotective role of melatonin via the upregulation of VEGF and MMP-2 expression.
-
(2010)
J Pineal Res
, vol.49
, pp. 130-140
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Ganguly, K.1
Sharma, A.V.2
Reiter, R.J.3
Swarnakar, S.4
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31
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79960342631
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Upregulation of collagenase-1 and-3 in indomethacin-induced gastric ulcer in diabetic rats: Role of melatonin
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The authors studied the gastroprotective effects of melatonin. The authors concluded that melatonin protects the gastric mucosa through the strict regulation of MMP-1 and MMP-13 secretion
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Pradeepkumar SL, Vivek SA, Swarnakar S. Upregulation of collagenase-1 and-3 in indomethacin-induced gastric ulcer in diabetic rats: role of melatonin. J Pineal Res 2011;51:61-74. The authors studied the gastroprotective effects of melatonin. The authors concluded that melatonin protects the gastric mucosa through the strict regulation of MMP-1 and MMP-13 secretion.
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(2011)
J Pineal Res
, vol.51
, pp. 61-74
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Pradeepkumar, S.L.1
Vivek, S.A.2
Swarnakar, S.3
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32
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77955660458
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Reactive oxygen species-quenching and antiapoptotic effect of polaprezinc on indomethacin-induced small intestinal epithelial cell injury
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The authors studied the effects of polaprezinc seen in indomethacin-induced small intestinal mucosal damage. The authors found that polaprezinc prevents ROS production via the inhibition of protein S-oxidation and thus decreases future cell apoptosis in the mucosa
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Omatsu T, Naito Y, Handa O, et al. Reactive oxygen species-quenching and antiapoptotic effect of polaprezinc on indomethacin-induced small intestinal epithelial cell injury. J Gastroenterol 2010;45:692-702. The authors studied the effects of polaprezinc seen in indomethacin-induced small intestinal mucosal damage. The authors found that polaprezinc prevents ROS production via the inhibition of protein S-oxidation and thus decreases future cell apoptosis in the mucosa.
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(2010)
J Gastroenterol
, vol.45
, pp. 692-702
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Omatsu, T.1
Naito, Y.2
Handa, O.3
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33
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79952187542
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Lipopolysaccharide-induced intestinal motility disturbances are mediated by c-Jun. NH2-terminal kinases
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The authors studied the influence of JNKs on intestinal motility abnormalities induced by LPS. The authors concluded that JNK is involved in LPS-induced intestinal contractions, oxidative stress, and COX-2 expression
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Gonzalo S, Grasa L, Arruebo MP, et al. Lipopolysaccharide-induced intestinal motility disturbances are mediated by c-Jun. NH2-terminal kinases. Dig Liver Dis 2011;43:277-285. The authors studied the influence of JNKs on intestinal motility abnormalities induced by LPS. The authors concluded that JNK is involved in LPS-induced intestinal contractions, oxidative stress, and COX-2 expression.
-
(2011)
Dig Liver Dis
, vol.43
, pp. 277-285
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Gonzalo, S.1
Grasa, L.2
Arruebo, M.P.3
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34
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77957749746
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Intestinal effects of lipopolysaccharide in rabbit are mediated by cyclooxygenase-2 through p38 mitogen activated protein kinase
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The authors studied the effects of COX-2 and p38 MAPK on intestinal disturbances in rabbits. The authors concluded that the LPS-induced decrease in duodenal contractility is because of the increase in oxidative stress caused by the increase in COX-2 expression that is mediated by the activation of the p38 MAPK pathway
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Gonzalo S, Grasa L, Fagundes DS, et al. Intestinal effects of lipopolysaccharide in rabbit are mediated by cyclooxygenase-2 through p38 mitogen activated protein kinase. Eur J Pharmacol 2010;648:171-178. The authors studied the effects of COX-2 and p38 MAPK on intestinal disturbances in rabbits. The authors concluded that the LPS-induced decrease in duodenal contractility is because of the increase in oxidative stress caused by the increase in COX-2 expression that is mediated by the activation of the p38 MAPK pathway.
-
(2010)
Eur J Pharmacol
, vol.648
, pp. 171-178
-
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Gonzalo, S.1
Grasa, L.2
Fagundes, D.S.3
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35
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77950662155
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Inhibition of p38 MAPK improves intestinal disturbances and oxidative stress induced in a rabbit endotoxemia model
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e123, The authors studied the role of p38 MAPK on intestinal motility and oxidative damage to gastric tissue. The authors concluded that p38 MAPK mediates the decrease in intestinal contractility caused by LPS
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Gonzalo S, Grasa L, Arruebo MP, et al. Inhibition of p38 MAPK improves intestinal disturbances and oxidative stress induced in a rabbit endotoxemia model. Neurogastroenterol Motil 2010;22:564-572; e123. The authors studied the role of p38 MAPK on intestinal motility and oxidative damage to gastric tissue. The authors concluded that p38 MAPK mediates the decrease in intestinal contractility caused by LPS.
-
(2010)
Neurogastroenterol Motil
, vol.22
, pp. 564-572
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Gonzalo, S.1
Grasa, L.2
Arruebo, M.P.3
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36
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78149491656
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Effect of aging on gastric mucosal defense mechanisms: ROS, apoptosis, angiogenesis, and sensory neurons
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The authors studied the aging gastric mucosa's increased susceptibility to gastric injury. The authors concluded that the aging mucosa is replaced by connective tissue and collagen, thus lessening its intrinsic gastroprotective mechanisms
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Kang JM, Kim N, Kim JH, et al. Effect of aging on gastric mucosal defense mechanisms: ROS, apoptosis, angiogenesis, and sensory neurons. Am J Physiol Gastrointest Liver Physiol 2010;299:G1147-G1153. The authors studied the aging gastric mucosa's increased susceptibility to gastric injury. The authors concluded that the aging mucosa is replaced by connective tissue and collagen, thus lessening its intrinsic gastroprotective mechanisms.
-
(2010)
Am J Physiol Gastrointest Liver Physiol
, vol.299
-
-
Kang, J.M.1
Kim, N.2
Kim, J.H.3
-
37
-
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0037192768
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PTEN modulates vascular endothelial growth factor-mediated signaling and angiogenic effects
-
DOI 10.1074/jbc.M110219200
-
Huang J, Kontos CD. PTEN modulates vascular endothelial growth factormediated signaling and angiogenic effects. J Biol Chem 2002;277:10760-10766. (Pubitemid 34952828)
-
(2002)
Journal of Biological Chemistry
, vol.277
, Issue.13
, pp. 10760-10766
-
-
Huang, J.1
Kontos, C.D.2
-
38
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77950291572
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PTEN silencing reverses aging-related impairment of angiogenesis in microvascular endothelial cells
-
Tarnawski AS, Pai R, Tanigawa T, et al. PTEN silencing reverses aging-related impairment of angiogenesis in microvascular endothelial cells. Biochem Biophys Res Commun 2010;394:291-296.
-
(2010)
Biochem Biophys Res Commun
, vol.394
, pp. 291-296
-
-
Tarnawski, A.S.1
Pai, R.2
Tanigawa, T.3
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