Beyond reduction of atherosclerosis: PON2 provides apoptosis resistance and stabilizes tumor cells

Cell Death and Disease

Volumn 2, Issue 1, 2011, Pages

  Witte, I ^a   Altenhofer S ^a   Wilgenbus, P ^a   Amort, J ^a   Clement, A M ^a   Pautz, A ^a   Li, H ^a   Fo Rstermann U ^a   Horke, S ^a  

^a UNIVERSITY MEDICAL CENTER MAINZ   (Germany)

Author keywords

Apoptosis; CHOP; Endoplasmic reticulum stress; Mitochondria; Oxidative stress; Paraoxonase

Indexed keywords

ARYLDIALKYLPHOSPHATASE 2; CARDIOLIPIN; CASPASE; CYTOCHROME C; DACTINOMYCIN; DOXORUBICIN; IMATINIB; STAUROSPORINE; STRESS ACTIVATED PROTEIN KINASE; SUPEROXIDE; TUMOR NECROSIS FACTOR ALPHA; TUMOR NECROSIS FACTOR RELATED APOPTOSIS INDUCING LIGAND; ARYLDIALKYLPHOSPHATASE; PON2 PROTEIN, HUMAN;

APOPTOSIS; ARTICLE; ATHEROSCLEROSIS; CANCER RESISTANCE; CARCINOGENESIS; CELL DEATH; CONTROLLED STUDY; ENZYME ACTIVATION; GENE OVEREXPRESSION; GENE SILENCING; HUMAN; HUMAN CELL; LIPID PEROXIDATION; MOLECULAR MECHANICS; OXIDATION REDUCTION REACTION; PLEIOTROPY; PRIORITY JOURNAL; PROTEIN DEFICIENCY; PROTEIN FUNCTION; SIGNAL TRANSDUCTION; TUMOR CELL; UPREGULATION; GENETICS; METABOLISM; MITOCHONDRION; OXIDATIVE STRESS; PATHOPHYSIOLOGY; TUMOR CELL LINE;

APOPTOSIS; ARYLDIALKYLPHOSPHATASE; ATHEROSCLEROSIS; CELL LINE, TUMOR; HUMANS; MITOCHONDRIA; OXIDATIVE STRESS; SUPEROXIDES;

EID: 79251528324     PISSN: None     EISSN: 20414889     Source Type: Journal    
DOI: 10.1038/cddis.2010.91     Document Type: Article

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