PKA and PKC partially rescue long QT type 1 phenotype by restoring channel-PIP2 interactions

Channels

Volumn 4, Issue 1, 2010, Pages 1-9

  Matavel, Alessandra ^a   Medei, Emiliano ^a   Lopes, Coeli M B ^a  

^a UNIVERSITY OF ROCHESTER MEDICAL CENTER   (United States)

Author keywords

Arrhythmias; Cardiac arrhythmia; Channelopathies; Ion channel modulation by GPCRs; KvLQT1; MinK; Modulation of channels; PI(4,5)P2; Potassium; Potassium channels

Indexed keywords

CYCLIC AMP DEPENDENT PROTEIN KINASE; FORSKOLIN; G PROTEIN COUPLED RECEPTOR; PHOSPHATIDYLINOSITOL 4 PHOSPHATE KINASE; PHOSPHATIDYLINOSITOL 4,5 BISPHOSPHATE; POTASSIUM CHANNEL KCNE1; POTASSIUM CHANNEL KCNQ1; PROTEIN KINASE C; WORTMANNIN;

ANIMAL CELL; ARTICLE; CONTROLLED STUDY; ENZYME ACTIVATION; ENZYME REGULATION; GENE MUTATION; LONG QT SYNDROME; NONHUMAN; OOCYTE; PHENOTYPE; POTASSIUM CURRENT; PROTEIN DOMAIN; PROTEIN FUNCTION; PROTEIN PROTEIN INTERACTION; SIGNAL TRANSDUCTION;

MUSTELA;

EID: 75649100572     PISSN: 19336950     EISSN: 19336969     Source Type: Journal    
DOI: 10.4161/chan.4.1.11537     Document Type: Article

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