-
1
-
-
0003443746
-
-
3rd, edition, Sunderland, MA, Sinauer Associates, Inc
-
Hille B. Ion Channels of Excitable Membranes 2001, 3rd, edition, Sunderland, MA, Sinauer Associates, Inc
-
(2001)
Ion Channels of Excitable Membranes
-
-
Hille, B.1
-
2
-
-
0033694833
-
From ionic currents to molecular mechanisms: The structure and function of voltage-gated sodium channels
-
Catterall WA. From ionic currents to molecular mechanisms: The structure and function of voltage-gated sodium channels. Neuron 2000, 26:13-25.
-
(2000)
Neuron
, vol.26
, pp. 13-25
-
-
Catterall, W.A.1
-
3
-
-
29844438166
-
International Union of Pharmacology. XLVII. Nomenclature and structure-function relationships of voltage-gated sodium channels
-
Catterall WA, Goldin AL, Waxman SG. International Union of Pharmacology. XLVII. Nomenclature and structure-function relationships of voltage-gated sodium channels. Pharmacol Rev 2005, 57:397-409.
-
(2005)
Pharmacol Rev
, vol.57
, pp. 397-409
-
-
Catterall, W.A.1
Goldin, A.L.2
Waxman, S.G.3
-
4
-
-
0026727999
-
An experimental model for peripheral neuropathy produced by segmental spinal nerve ligation in the rat
-
Kim SH, Chung JM. An experimental model for peripheral neuropathy produced by segmental spinal nerve ligation in the rat. Pain 1992, 50:355-63.
-
(1992)
Pain
, vol.50
, pp. 355-363
-
-
Kim, S.H.1
Chung, J.M.2
-
5
-
-
0034255439
-
Spared nerve injury: An animal model of persistent peripheral neuropathic pain
-
Decosterd I, Woolf CJ. Spared nerve injury: An animal model of persistent peripheral neuropathic pain. Pain 2000, 87:149-58.
-
(2000)
Pain
, vol.87
, pp. 149-158
-
-
Decosterd, I.1
Woolf, C.J.2
-
6
-
-
0023895875
-
A peripheral mononeuropathy in rat that produces disorders of pain sensation like those seen in man
-
Bennett GJ, Xie YK. A peripheral mononeuropathy in rat that produces disorders of pain sensation like those seen in man. Pain 1988, 33:87-107.
-
(1988)
Pain
, vol.33
, pp. 87-107
-
-
Bennett, G.J.1
Xie, Y.K.2
-
7
-
-
0032746351
-
The molecular pathophysiology of pain: Abnormal expression of sodium channel genes and its contributions to hyperexcitability of primary sensory neurons
-
Waxman SG. The molecular pathophysiology of pain: Abnormal expression of sodium channel genes and its contributions to hyperexcitability of primary sensory neurons. Pain 1999, Suppl 6:S133-40.
-
(1999)
Pain
, vol.SUPPL. 6
-
-
Waxman, S.G.1
-
8
-
-
0035462457
-
Transcriptional channelopathies: An emerging class of disorders
-
Waxman SG. Transcriptional channelopathies: An emerging class of disorders. Nat Rev Neurosci 2001, 2:652-9.
-
(2001)
Nat Rev Neurosci
, vol.2
, pp. 652-659
-
-
Waxman, S.G.1
-
9
-
-
0027933696
-
Type III sodium channel mRNA is expressed in embryonic but not adult spinal sensory neurons, and is reexpressed following axotomy
-
Waxman SG, Kocsis JD, Black JA. Type III sodium channel mRNA is expressed in embryonic but not adult spinal sensory neurons, and is reexpressed following axotomy. J Neurophysiol 1994, 72:466-70.
-
(1994)
J Neurophysiol
, vol.72
, pp. 466-470
-
-
Waxman, S.G.1
Kocsis, J.D.2
Black, J.A.3
-
10
-
-
0032743734
-
Upregulation of a silent sodium channel after peripheral, but not central, nerve injury in DRG neurons
-
Black JA, Cummins TR, Plumpton C. Upregulation of a silent sodium channel after peripheral, but not central, nerve injury in DRG neurons. J Neurophysiol 1999, 82:2776-85.
-
(1999)
J Neurophysiol
, vol.82
, pp. 2776-2785
-
-
Black, J.A.1
Cummins, T.R.2
Plumpton, C.3
-
11
-
-
0029907091
-
Down-regulation of transcripts for Na channel alpha-SNS in spinal sensory neurons following axotomy
-
Dib-Hajj S, Black JA, Felts P, Waxman SG. Down-regulation of transcripts for Na channel alpha-SNS in spinal sensory neurons following axotomy. Proc Natl Acad Sci USA 1996, 93:14950-4.
-
(1996)
Proc Natl Acad Sci USA
, vol.93
, pp. 14950-14954
-
-
Dib-Hajj, S.1
Black, J.A.2
Felts, P.3
Waxman, S.G.4
-
12
-
-
0032555278
-
NaN, a novel voltage-gated Na channel, is expressed preferentially in peripheral sensory neurons and down-regulated after axotomy
-
Dib-Hajj SD, Tyrrell L, Black JA, Waxman SG. NaN, a novel voltage-gated Na channel, is expressed preferentially in peripheral sensory neurons and down-regulated after axotomy. Proc Natl Acad Sci USA 1998, 95:8963-8.
-
(1998)
Proc Natl Acad Sci USA
, vol.95
, pp. 8963-8968
-
-
Dib-Hajj, S.D.1
Tyrrell, L.2
Black, J.A.3
Waxman, S.G.4
-
13
-
-
0034307559
-
Changes in expression of two tetrodotoxin-resistant sodium channels and their currents in dorsal root ganglion neurons after sciatic nerve injury but not rhizotomy
-
Sleeper AA, Cummins TR, Dib-Hajj SD. Changes in expression of two tetrodotoxin-resistant sodium channels and their currents in dorsal root ganglion neurons after sciatic nerve injury but not rhizotomy. J Neurosci 2000, 20:7279-89.
-
(2000)
J Neurosci
, vol.20
, pp. 7279-7289
-
-
Sleeper, A.A.1
Cummins, T.R.2
Dib-Hajj, S.D.3
-
14
-
-
0036221972
-
The pattern of expression of the voltage-gated sodium channels Na(v)1.8 and Na(v)1.9 does not change in uninjured primary sensory neurons in experimental neuropathic pain models
-
Decosterd I, Ji RR, Abdi S, Tate S, Woolf CJ. The pattern of expression of the voltage-gated sodium channels Na(v)1.8 and Na(v)1.9 does not change in uninjured primary sensory neurons in experimental neuropathic pain models. Pain 2002, 96:269-77.
-
(2002)
Pain
, vol.96
, pp. 269-277
-
-
Decosterd, I.1
Ji, R.R.2
Abdi, S.3
Tate, S.4
Woolf, C.J.5
-
15
-
-
0035500407
-
The changes in expression of three subtypes of TTX sensitive sodium channels in sensory neurons after spinal nerve ligation
-
Kim CH, Oh Y, Chung JM, Chung K. The changes in expression of three subtypes of TTX sensitive sodium channels in sensory neurons after spinal nerve ligation. Mol Brain Res 2001, 95:153-61.
-
(2001)
Mol Brain Res
, vol.95
, pp. 153-161
-
-
Kim, C.H.1
Oh, Y.2
Chung, J.M.3
Chung, K.4
-
16
-
-
0037177482
-
Changes in three subtypes of tetrodotoxin sensitive sodium channel expression in the axotomized dorsal root ganglion in the rat
-
Kim CH, Oh Y, Chung JM, Chung K. Changes in three subtypes of tetrodotoxin sensitive sodium channel expression in the axotomized dorsal root ganglion in the rat. Neurosci Lett 2002, 323:125-8.
-
(2002)
Neurosci Lett
, vol.323
, pp. 125-128
-
-
Kim, C.H.1
Oh, Y.2
Chung, J.M.3
Chung, K.4
-
17
-
-
0030997916
-
Downregulation of tetrodotoxin-resistant sodium currents and upregulation of a rapidly repriming tetrodotoxin-sensitive sodium current in small spinal sensory neurons after nerve injury
-
Cummins TR, Waxman SG. Downregulation of tetrodotoxin-resistant sodium currents and upregulation of a rapidly repriming tetrodotoxin-sensitive sodium current in small spinal sensory neurons after nerve injury. J Neurosci 1997, 17:3503-14.
-
(1997)
J Neurosci
, vol.17
, pp. 3503-3514
-
-
Cummins, T.R.1
Waxman, S.G.2
-
18
-
-
0029279185
-
Selective loss of slow and enhancement of fast Na+ currents in cutaneous afferent dorsal root ganglion neurones following axotomy
-
Rizzo MA, Kocsis JD, Waxman SG. Selective loss of slow and enhancement of fast Na+ currents in cutaneous afferent dorsal root ganglion neurones following axotomy. Neurobiol Dis 1995, 2:87-96.
-
(1995)
Neurobiol Dis
, vol.2
, pp. 87-96
-
-
Rizzo, M.A.1
Kocsis, J.D.2
Waxman, S.G.3
-
19
-
-
0034554778
-
Glial-derived neurotrophic factor upregulates expression of functional SNS and NaN sodium channels and their currents in axotomized dorsal root ganglion neurons
-
Cummins TR, Black JA, Dib-Hajj SD, Waxman SG. Glial-derived neurotrophic factor upregulates expression of functional SNS and NaN sodium channels and their currents in axotomized dorsal root ganglion neurons. J Neurosci 2000, 20:8754-61.
-
(2000)
J Neurosci
, vol.20
, pp. 8754-8761
-
-
Cummins, T.R.1
Black, J.A.2
Dib-Hajj, S.D.3
Waxman, S.G.4
-
20
-
-
0037218619
-
Redistribution of Na(V)1.8 in uninjured axons enables neuropathic pain
-
Gold MS, Weinreich D, Kim CS. Redistribution of Na(V)1.8 in uninjured axons enables neuropathic pain. J Neurosci 2003, 23:158-66.
-
(2003)
J Neurosci
, vol.23
, pp. 158-166
-
-
Gold, M.S.1
Weinreich, D.2
Kim, C.S.3
-
21
-
-
0035882277
-
Nav1.3 sodium channels: Rapid repriming and slow closed-state inactivation display quantitative differences after expression in a mammalian cell line and in spinal sensory neurons
-
Cummins TR, Aglieco F, Renganathan M. Nav1.3 sodium channels: Rapid repriming and slow closed-state inactivation display quantitative differences after expression in a mammalian cell line and in spinal sensory neurons. J Neurosci 2001, 21:5952-61.
-
(2001)
J Neurosci
, vol.21
, pp. 5952-5961
-
-
Cummins, T.R.1
Aglieco, F.2
Renganathan, M.3
-
22
-
-
33749326179
-
Upregulation of persistent and ramp sodium current in dorsal horn neurons after spinal cord injury
-
Lampert A, Hains BC, Waxman SG. Upregulation of persistent and ramp sodium current in dorsal horn neurons after spinal cord injury. Exp Brain Res 2006, 174:660-6.
-
(2006)
Exp Brain Res
, vol.174
, pp. 660-666
-
-
Lampert, A.1
Hains, B.C.2
Waxman, S.G.3
-
23
-
-
0033955244
-
Immunolocalization of SNS/PN3 and NaN/SNS2 sodium channels in human pain states
-
Coward K, Plumpton C, Facer P. Immunolocalization of SNS/PN3 and NaN/SNS2 sodium channels in human pain states. Pain 2000, 85:41-50.
-
(2000)
Pain
, vol.85
, pp. 41-50
-
-
Coward, K.1
Plumpton, C.2
Facer, P.3
-
24
-
-
0032723079
-
Plasticity of sodium channel expression in DRG neurons in the chronic constriction injury model of neuropathic pain
-
Dib-Hajj SD, Fjell J, Cummins TR. Plasticity of sodium channel expression in DRG neurons in the chronic constriction injury model of neuropathic pain. Pain 1999, 83:591-600.
-
(1999)
Pain
, vol.83
, pp. 591-600
-
-
Dib-Hajj, S.D.1
Fjell, J.2
Cummins, T.R.3
-
25
-
-
2442651551
-
Altered sodium channel expression in second-order spinal sensory neurons contributes to pain after peripheral nerve injury
-
Hains BC, Saab CY, Klein JP, Craner MJ, Waxman SG. Altered sodium channel expression in second-order spinal sensory neurons contributes to pain after peripheral nerve injury. J Neurosci 2004, 24:4832-9.
-
(2004)
J Neurosci
, vol.24
, pp. 4832-4839
-
-
Hains, B.C.1
Saab, C.Y.2
Klein, J.P.3
Craner, M.J.4
Waxman, S.G.5
-
26
-
-
30844469806
-
Sodium channels and mechanisms of neuropathic pain
-
Devor M. Sodium channels and mechanisms of neuropathic pain. J Pain 2006, 7(1):S3-12. suppl
-
(2006)
J Pain
, vol.7
, Issue.1 SUPPL
-
-
Devor, M.1
-
27
-
-
0036366983
-
Clinical article accumulation of PN1 and PN3 sodium channels in painful human neuroma-evidence from immunocytochemistry
-
Kretschmer T, Happel LT, England JD. Clinical article accumulation of PN1 and PN3 sodium channels in painful human neuroma-evidence from immunocytochemistry. Acta Neurochir (Wien) 2002, 144:803-10.
-
(2002)
Acta Neurochir (Wien)
, vol.144
, pp. 803-810
-
-
Kretschmer, T.1
Happel, L.T.2
England, J.D.3
-
28
-
-
58149232445
-
Multiple sodium channel isoforms and mitogen-activated protein kinases are present in painful human neuromas
-
Black JA, Nikolajsen L, Kroner K, Jensen TS, Waxman SG. Multiple sodium channel isoforms and mitogen-activated protein kinases are present in painful human neuromas. Ann Neurol 2008, 64:644-53.
-
(2008)
Ann Neurol
, vol.64
, pp. 644-653
-
-
Black, J.A.1
Nikolajsen, L.2
Kroner, K.3
Jensen, T.S.4
Waxman, S.G.5
-
29
-
-
33947124182
-
Na(v)1.7 sodium channel expression in human lingual nerve neuromas
-
Bird EV, Robinson PP, Boissonade FM. Na(v)1.7 sodium channel expression in human lingual nerve neuromas. Arch Oral Biol 2007, 52(5):494-502.
-
(2007)
Arch Oral Biol
, vol.52
, Issue.5
, pp. 494-502
-
-
Bird, E.V.1
Robinson, P.P.2
Boissonade, F.M.3
-
30
-
-
41149153802
-
Phosphorylation of sodium channel Nav1.8 by p38 mitogen-activated protein kinase increases current density in dorsal root ganglion neurons
-
Hudmon A, Choi JS, Tyrrell L. Phosphorylation of sodium channel Nav1.8 by p38 mitogen-activated protein kinase increases current density in dorsal root ganglion neurons. J Neurosci 2008, 28:3190-201.
-
(2008)
J Neurosci
, vol.28
, pp. 3190-3201
-
-
Hudmon, A.1
Choi, J.S.2
Tyrrell, L.3
-
31
-
-
51549119311
-
The sodium channel Nav1.7 is a substrate and is modulated by the MAP kinase ERK
-
Abstr #46620
-
Stamboulian S, Choi J-S, Tyrrell L, Waxman S, Dib-Hajj S. The sodium channel Nav1.7 is a substrate and is modulated by the MAP kinase ERK. Soc Neurosci 2007, Abstr #46620
-
(2007)
Soc Neurosci
-
-
Stamboulian, S.1
Choi, J.-S.2
Tyrrell, L.3
Waxman, S.4
Dib-Hajj, S.5
-
32
-
-
0034807327
-
Protein kinase A modulates spontaneous activity in chronically compressed dorsal root ganglion neurons in the rat
-
Hu SJ, Song XJ, Greenquist KW, Zhang JM, LaMotte RH. Protein kinase A modulates spontaneous activity in chronically compressed dorsal root ganglion neurons in the rat. Pain 2001, 94:39-46.
-
(2001)
Pain
, vol.94
, pp. 39-46
-
-
Hu, S.J.1
Song, X.J.2
Greenquist, K.W.3
Zhang, J.M.4
LaMotte, R.H.5
-
33
-
-
0141955927
-
Somata of nerve-injured sensory neurons exhibit enhanced responses to inflammatory mediators
-
Song XJ, Zhang JM, Hu SJ, LaMotte RH. Somata of nerve-injured sensory neurons exhibit enhanced responses to inflammatory mediators. Pain 2003, 104:701-9.
-
(2003)
Pain
, vol.104
, pp. 701-709
-
-
Song, X.J.1
Zhang, J.M.2
Hu, S.J.3
LaMotte, R.H.4
-
34
-
-
33751220188
-
MCP-1 enhances excitability of nociceptive neurons in chronically compressed dorsal root ganglia
-
Sun JH, Yang B, Donnelly DF, Ma C, LaMotte RH. MCP-1 enhances excitability of nociceptive neurons in chronically compressed dorsal root ganglia. J Neurophysiol 2006, 96:2189-99.
-
(2006)
J Neurophysiol
, vol.96
, pp. 2189-2199
-
-
Sun, J.H.1
Yang, B.2
Donnelly, D.F.3
Ma, C.4
LaMotte, R.H.5
-
35
-
-
0037099592
-
Changes in expression of voltage-dependent ion channel subunits in dorsal root ganglia of rats with radicular injury and pain
-
discussion 25
-
Abe M, Kurihara T, Han W, Shinomiya K, Tanabe T. Changes in expression of voltage-dependent ion channel subunits in dorsal root ganglia of rats with radicular injury and pain. Spine 2002, 27:1517-24. discussion 25
-
(2002)
Spine
, vol.27
, pp. 1517-1524
-
-
Abe, M.1
Kurihara, T.2
Han, W.3
Shinomiya, K.4
Tanabe, T.5
-
36
-
-
33644859588
-
Effects of a chronic compression of the dorsal root ganglion on voltage-gated Na+ and K+ currents in cutaneous afferent neurons
-
Tan ZY, Donnelly DF, LaMotte RH. Effects of a chronic compression of the dorsal root ganglion on voltage-gated Na+ and K+ currents in cutaneous afferent neurons. J Neurophysiol 2006, 95:1115-23.
-
(2006)
J Neurophysiol
, vol.95
, pp. 1115-1123
-
-
Tan, Z.Y.1
Donnelly, D.F.2
LaMotte, R.H.3
-
37
-
-
33646071787
-
The role of sodium channels in chronic inflammatory and neuropathic pain
-
Amir R, Argoff CE, Bennett GJ. The role of sodium channels in chronic inflammatory and neuropathic pain. J Pain 2006, 7:S1-29.
-
(2006)
J Pain
, vol.7
-
-
Amir, R.1
Argoff, C.E.2
Bennett, G.J.3
-
38
-
-
1542498432
-
Changes in the expression of tetrodotoxin-sensitive sodium channels within dorsal root ganglia neurons in inflammatory pain
-
Black JA, Liu S, Tanaka M, Cummins TR, Waxman SG. Changes in the expression of tetrodotoxin-sensitive sodium channels within dorsal root ganglia neurons in inflammatory pain. Pain 2004, 108:237-47.
-
(2004)
Pain
, vol.108
, pp. 237-247
-
-
Black, J.A.1
Liu, S.2
Tanaka, M.3
Cummins, T.R.4
Waxman, S.G.5
-
39
-
-
0036182217
-
Mild gastritis alters voltage-sensitive sodium currents in gastric sensory neurons in rats
-
Bielefeldt K, Ozaki N, Gebhart GF. Mild gastritis alters voltage-sensitive sodium currents in gastric sensory neurons in rats. Gastroenterology 2002, 122:752-61.
-
(2002)
Gastroenterology
, vol.122
, pp. 752-761
-
-
Bielefeldt, K.1
Ozaki, N.2
Gebhart, G.F.3
-
40
-
-
14844298657
-
Decrease in inflammatory hyperalgesia by herpes vector-mediated knockdown of Na(v)1.7 sodium channels in primary afferents
-
Yeomans DC, Levinson SR, Peters MC. Decrease in inflammatory hyperalgesia by herpes vector-mediated knockdown of Na(v)1.7 sodium channels in primary afferents. Hum Gene Ther 2005, 16:271-7.
-
(2005)
Hum Gene Ther
, vol.16
, pp. 271-277
-
-
Yeomans, D.C.1
Levinson, S.R.2
Peters, M.C.3
-
41
-
-
4344670206
-
Nociceptor-specific gene deletion reveals a major role for Nav1.7 (PN1) in acute and inflammatory pain
-
Nassar MA, Stirling LC, Forlani G. Nociceptor-specific gene deletion reveals a major role for Nav1.7 (PN1) in acute and inflammatory pain. Proc Natl Acad Sci USA 2004, 101:12706-11.
-
(2004)
Proc Natl Acad Sci USA
, vol.101
, pp. 12706-12711
-
-
Nassar, M.A.1
Stirling, L.C.2
Forlani, G.3
-
42
-
-
0033363999
-
The tetrodotoxin-resistant sodium channel SNS has a specialized function in pain pathways
-
Akopian AN, Souslova V, England S. The tetrodotoxin-resistant sodium channel SNS has a specialized function in pain pathways. Nat Neurosci 1999, 2:541-8.
-
(1999)
Nat Neurosci
, vol.2
, pp. 541-548
-
-
Akopian, A.N.1
Souslova, V.2
England, S.3
-
43
-
-
33748888226
-
Dissecting the role of sodium currents in visceral sensory neurons in a model of chronic hyperexcitability using Nav1.8 and Nav1.9 null mice
-
Hillsley K, Lin JH, Stanisz A. Dissecting the role of sodium currents in visceral sensory neurons in a model of chronic hyperexcitability using Nav1.8 and Nav1.9 null mice. J Physiol (Lond) 2006, 576:257-67.
-
(2006)
J Physiol (Lond)
, vol.576
, pp. 257-267
-
-
Hillsley, K.1
Lin, J.H.2
Stanisz, A.3
-
44
-
-
33744548162
-
Involvement of the TTX-resistant sodium channel Nav 1.8 in inflammatory and neuropathic, but not post-operative, pain states
-
Joshi SK, Mikusa JP, Hernandez G. Involvement of the TTX-resistant sodium channel Nav 1.8 in inflammatory and neuropathic, but not post-operative, pain states. Pain 2006, 123:75-82.
-
(2006)
Pain
, vol.123
, pp. 75-82
-
-
Joshi, S.K.1
Mikusa, J.P.2
Hernandez, G.3
-
45
-
-
0001059602
-
A comparison of the potential role of the tetrodotoxin-insensitive sodium channels, PN3/SNS and NaN/SNS2, in rat models of chronic pain [In Process Citation]
-
Porreca F, Lai J, Bian D. A comparison of the potential role of the tetrodotoxin-insensitive sodium channels, PN3/SNS and NaN/SNS2, in rat models of chronic pain [In Process Citation]. Proc Natl Acad Sci USA 1999, 96:7640-4.
-
(1999)
Proc Natl Acad Sci USA
, vol.96
, pp. 7640-7644
-
-
Porreca, F.1
Lai, J.2
Bian, D.3
-
46
-
-
0035503586
-
The involvement of the tetrodotoxin-resistant sodium channel Nav1.8 (PN3/SNS) in a rat model of visceral pain
-
Yoshimura N, Seki S, Novakovic SD. The involvement of the tetrodotoxin-resistant sodium channel Nav1.8 (PN3/SNS) in a rat model of visceral pain. J Neurosci 2001, 21:8690-6.
-
(2001)
J Neurosci
, vol.21
, pp. 8690-8696
-
-
Yoshimura, N.1
Seki, S.2
Novakovic, S.D.3
-
47
-
-
21544455502
-
Contribution of the tetrodotoxin-resistant voltage-gated sodium channel NaV1.9 to sensory transmission and nociceptive behavior
-
Priest BT, Murphy BA, Lindia JA. Contribution of the tetrodotoxin-resistant voltage-gated sodium channel NaV1.9 to sensory transmission and nociceptive behavior. Proc Natl Acad Sci USA 2005, 102:9382-7.
-
(2005)
Proc Natl Acad Sci USA
, vol.102
, pp. 9382-9387
-
-
Priest, B.T.1
Murphy, B.A.2
Lindia, J.A.3
-
48
-
-
33845631301
-
The voltage-gated sodium channel Na(v)1.9 is an effector of peripheral inflammatory pain hypersensitivity
-
Amaya F, Wang H, Costigan M. The voltage-gated sodium channel Na(v)1.9 is an effector of peripheral inflammatory pain hypersensitivity. J Neurosci 2006, 26:12852-60.
-
(2006)
J Neurosci
, vol.26
, pp. 12852-12860
-
-
Amaya, F.1
Wang, H.2
Costigan, M.3
-
49
-
-
0036894050
-
Changes of sodium channel expression in experimental painful diabetic neuropathy
-
Craner MJ, Klein JP, Renganathan M, Black JA, Waxman SG. Changes of sodium channel expression in experimental painful diabetic neuropathy. Ann Neurol 2002, 52:786-92.
-
(2002)
Ann Neurol
, vol.52
, pp. 786-792
-
-
Craner, M.J.1
Klein, J.P.2
Renganathan, M.3
Black, J.A.4
Waxman, S.G.5
-
50
-
-
3142761823
-
Early painful diabetic neuropathy is associated with differential changes in tetrodotoxin-sensitive and -resistant sodium channels in dorsal root ganglion neurons in the rat
-
Hong S, Morrow TJ, Paulson PE, Isom LL, Wiley JW. Early painful diabetic neuropathy is associated with differential changes in tetrodotoxin-sensitive and -resistant sodium channels in dorsal root ganglion neurons in the rat. J Biol Chem 2004, 279:29341-50.
-
(2004)
J Biol Chem
, vol.279
, pp. 29341-29350
-
-
Hong, S.1
Morrow, T.J.2
Paulson, P.E.3
Isom, L.L.4
Wiley, J.W.5
-
51
-
-
0032400799
-
Slow closed-state inactivation: A novel mechanism underlying ramp currents in cells expressing the hNE/PN1 sodium channel
-
Cummins TR, Howe JR, Waxman SG. Slow closed-state inactivation: A novel mechanism underlying ramp currents in cells expressing the hNE/PN1 sodium channel. J Neurosci 1998, 18:9607-19.
-
(1998)
J Neurosci
, vol.18
, pp. 9607-9619
-
-
Cummins, T.R.1
Howe, J.R.2
Waxman, S.G.3
-
52
-
-
0141646635
-
Distinct repriming and closed-state inactivation kinetics of Nav1.6 and Nav1.7 sodium channels in mouse spinal sensory neurons
-
Herzog RI, Cummins TR, Ghassemi F, Dib-Hajj SD, Waxman SG. Distinct repriming and closed-state inactivation kinetics of Nav1.6 and Nav1.7 sodium channels in mouse spinal sensory neurons. J Physiol (Lond) 2003, 551:741-50.
-
(2003)
J Physiol (Lond)
, vol.551
, pp. 741-750
-
-
Herzog, R.I.1
Cummins, T.R.2
Ghassemi, F.3
Dib-Hajj, S.D.4
Waxman, S.G.5
-
53
-
-
33847038041
-
Mutations in the sodium channel Nav1.7 underlie inherited erythromelalgia
-
Dib-Hajj S, Rush A, Cummins T, Waxman S. Mutations in the sodium channel Nav1.7 underlie inherited erythromelalgia. Drug Discov Today Dis Mech 2006, 3:343-50.
-
(2006)
Drug Discov Today Dis Mech
, vol.3
, pp. 343-350
-
-
Dib-Hajj, S.1
Rush, A.2
Cummins, T.3
Waxman, S.4
-
54
-
-
28244468409
-
Erythermalgia: Molecular basis for an inherited pain syndrome
-
Waxman SG, Dib-Hajj S. Erythermalgia: Molecular basis for an inherited pain syndrome. Trends Mol Med 2005, 11:555-62.
-
(2005)
Trends Mol Med
, vol.11
, pp. 555-562
-
-
Waxman, S.G.1
Dib-Hajj, S.2
-
55
-
-
33847168937
-
SCN9A mutations in paroxysmal extreme pain disorder: Allelic variants underlie distinct channel defects and phenotypes
-
Fertleman CR, Baker MD, Parker KA. SCN9A mutations in paroxysmal extreme pain disorder: Allelic variants underlie distinct channel defects and phenotypes. Neuron 2006, 52:767-74.
-
(2006)
Neuron
, vol.52
, pp. 767-774
-
-
Fertleman, C.R.1
Baker, M.D.2
Parker, K.A.3
-
56
-
-
33845901486
-
An SCN9A channelopathy causes congenital inability to experience pain
-
Cox JJ, Reimann F, Nicholas AK. An SCN9A channelopathy causes congenital inability to experience pain. Nature 2006, 444:894-8.
-
(2006)
Nature
, vol.444
, pp. 894-898
-
-
Cox, J.J.1
Reimann, F.2
Nicholas, A.K.3
-
57
-
-
34247874778
-
Loss-of-function mutations in the Nav1.7 gene underlie congenital indifference to pain in multiple human populations
-
Goldberg Y, Macfarlane J, Macdonald M. Loss-of-function mutations in the Nav1.7 gene underlie congenital indifference to pain in multiple human populations. Clin Genet 2007, 71:311-19.
-
(2007)
Clin Genet
, vol.71
, pp. 311-319
-
-
Goldberg, Y.1
Macfarlane, J.2
Macdonald, M.3
-
58
-
-
12144288410
-
Mutations in SCN9A, encoding a sodium channel alpha subunit, in patients with primary erythermalgia
-
Yang Y, Wang Y, Li S. Mutations in SCN9A, encoding a sodium channel alpha subunit, in patients with primary erythermalgia. J Med Genet 2004, 41:171-4.
-
(2004)
J Med Genet
, vol.41
, pp. 171-174
-
-
Yang, Y.1
Wang, Y.2
Li, S.3
-
59
-
-
35748935573
-
From genes to pain: Nav1.7 and human pain disorders
-
Dib-Hajj SD, Cummins TR, Black JA, Waxman SG. From genes to pain: Nav1.7 and human pain disorders. Trends Neurosci 2007, 30:555-63.
-
(2007)
Trends Neurosci
, vol.30
, pp. 555-563
-
-
Dib-Hajj, S.D.1
Cummins, T.R.2
Black, J.A.3
Waxman, S.G.4
-
60
-
-
36849036949
-
Mutations in sodium-channel gene SCN9A cause a spectrum of human genetic pain disorders
-
Drenth JP, Waxman SG. Mutations in sodium-channel gene SCN9A cause a spectrum of human genetic pain disorders. J Clin Invest 2007, 117:3603-9.
-
(2007)
J Clin Invest
, vol.117
, pp. 3603-3609
-
-
Drenth, J.P.1
Waxman, S.G.2
-
61
-
-
23444443202
-
Gain-of-function mutation in Nav1.7 in familial erythromelalgia induces bursting of sensory neurons
-
Dib-Hajj SD, Rush AM, Cummins TR. Gain-of-function mutation in Nav1.7 in familial erythromelalgia induces bursting of sensory neurons. Brain 2005, 128:1847-54.
-
(2005)
Brain
, vol.128
, pp. 1847-1854
-
-
Dib-Hajj, S.D.1
Rush, A.M.2
Cummins, T.R.3
-
62
-
-
33644826050
-
Sporadic onset of erythermalgia: A gain-of-function mutation in Nav1.7
-
Han C, Rush AM, Dib-Hajj SD. Sporadic onset of erythermalgia: A gain-of-function mutation in Nav1.7. Ann Neurol 2006, 59:553-8.
-
(2006)
Ann Neurol
, vol.59
, pp. 553-558
-
-
Han, C.1
Rush, A.M.2
Dib-Hajj, S.D.3
-
63
-
-
33845271929
-
NaV1.7 mutant A863P in erythromelalgia: Effects of altered activation and steady-state inactivation on excitability of nociceptive dorsal root ganglion neurons
-
Harty TP, Dib-Hajj SD, Tyrrell L. NaV1.7 mutant A863P in erythromelalgia: Effects of altered activation and steady-state inactivation on excitability of nociceptive dorsal root ganglion neurons. J Neurosci 2006, 26:12566-75.
-
(2006)
J Neurosci
, vol.26
, pp. 12566-12575
-
-
Harty, T.P.1
Dib-Hajj, S.D.2
Tyrrell, L.3
-
64
-
-
20544476041
-
SCN9A mutations define primary erythermalgia as a neuropathic disorder of voltage gated sodium channels
-
Drenth JP, Te Morsche RH, Guillet G. SCN9A mutations define primary erythermalgia as a neuropathic disorder of voltage gated sodium channels. J Invest Dermatol 2005, 124:1333-8.
-
(2005)
J Invest Dermatol
, vol.124
, pp. 1333-1338
-
-
Drenth, J.P.1
Te Morsche, R.H.2
Guillet, G.3
-
65
-
-
26444466085
-
Autosomal dominant erythermalgia associated with a novel mutation in the voltage-gated sodium channel alpha subunit Nav1.7
-
Michiels JJ, te Morsche RH, Jansen JB, Drenth JP. Autosomal dominant erythermalgia associated with a novel mutation in the voltage-gated sodium channel alpha subunit Nav1.7. Arch Neurol 2005, 62:1587-90.
-
(2005)
Arch Neurol
, vol.62
, pp. 1587-1590
-
-
Michiels, J.J.1
te Morsche, R.H.2
Jansen, J.B.3
Drenth, J.P.4
-
66
-
-
33847666143
-
Characterization of a familial case with primary erythromelalgia from Taiwan
-
Lee MJ, Yu HS, Hsieh ST. Characterization of a familial case with primary erythromelalgia from Taiwan. J Neurol 2007, 254:210-14.
-
(2007)
J Neurol
, vol.254
, pp. 210-214
-
-
Lee, M.J.1
Yu, H.S.2
Hsieh, S.T.3
-
67
-
-
36649037802
-
A case of primary erythermalgia, wintry hypothermia and encephalopathy
-
Takahashi K, Saitoh M, Hoshino H. A case of primary erythermalgia, wintry hypothermia and encephalopathy. Neuropediatrics 2007, 38:157-9.
-
(2007)
Neuropediatrics
, vol.38
, pp. 157-159
-
-
Takahashi, K.1
Saitoh, M.2
Hoshino, H.3
-
68
-
-
33750976383
-
Inherited erythermalgia. Limb pain from an S4 charge-neutral Na channelopathy
-
Choi JS, Dib-Hajj SD, Waxman SG. Inherited erythermalgia. Limb pain from an S4 charge-neutral Na channelopathy. Neurology 2006, 67:1563-7.
-
(2006)
Neurology
, vol.67
, pp. 1563-1567
-
-
Choi, J.S.1
Dib-Hajj, S.D.2
Waxman, S.G.3
-
69
-
-
4644268452
-
Electrophysiological properties of mutant Nav1.7 sodium channels in a painful inherited neuropathy
-
Cummins TR, Dib-Hajj SD, Waxman SG. Electrophysiological properties of mutant Nav1.7 sodium channels in a painful inherited neuropathy. J Neurosci 2004, 24:8232-6.
-
(2004)
J Neurosci
, vol.24
, pp. 8232-8236
-
-
Cummins, T.R.1
Dib-Hajj, S.D.2
Waxman, S.G.3
-
70
-
-
33846021646
-
Size matters: Erythromelalgia mutation S241T in Nav1.7 alters channel gating
-
Lampert A, Dib-Hajj SD, Tyrrell L, Waxman SG. Size matters: Erythromelalgia mutation S241T in Nav1.7 alters channel gating. J Biol Chem 2006, 281:36029-35.
-
(2006)
J Biol Chem
, vol.281
, pp. 36029-36035
-
-
Lampert, A.1
Dib-Hajj, S.D.2
Tyrrell, L.3
Waxman, S.G.4
-
71
-
-
40449087926
-
Mutation I136V alters electrophysiological properties of the NaV1.7 channel in a family with onset of erythromelalgia in the second decade
-
Cheng X, Dib-Hajj SD, Tyrrell L, Waxman SG. Mutation I136V alters electrophysiological properties of the NaV1.7 channel in a family with onset of erythromelalgia in the second decade. Mol Pain 2008, 4:1.
-
(2008)
Mol Pain
, vol.4
, pp. 1
-
-
Cheng, X.1
Dib-Hajj, S.D.2
Tyrrell, L.3
Waxman, S.G.4
-
72
-
-
34249980057
-
A Nav1.7 channel mutation associated with hereditary erythromelalgia contributes to neuronal hyperexcitability and displays reduced lidocaine sensitivity
-
Sheets PL, Jackson Ii JO, Waxman SG, Dib-Hajj S, Cummins TR. A Nav1.7 channel mutation associated with hereditary erythromelalgia contributes to neuronal hyperexcitability and displays reduced lidocaine sensitivity. J Physiol (Lond) 2007, 581:1019-31.
-
(2007)
J Physiol (Lond)
, vol.581
, pp. 1019-1031
-
-
Sheets, P.L.1
Jackson Ii, J.O.2
Waxman, S.G.3
Dib-Hajj, S.4
Cummins, T.R.5
-
73
-
-
33744454923
-
A single sodium channel mutation produces hyper- or hypoexcitability in different types of neurons
-
Rush AM, Dib-Hajj SD, Liu S. A single sodium channel mutation produces hyper- or hypoexcitability in different types of neurons. Proc Natl Acad Sci USA 2006, 103:8245-50.
-
(2006)
Proc Natl Acad Sci USA
, vol.103
, pp. 8245-8250
-
-
Rush, A.M.1
Dib-Hajj, S.D.2
Liu, S.3
-
74
-
-
0030041548
-
A tetrodotoxin-resistant voltage-gated sodium channel expressed by sensory neurons
-
Akopian AN, Sivilotti L, Wood JN. A tetrodotoxin-resistant voltage-gated sodium channel expressed by sensory neurons. Nature 1996, 379:257-62.
-
(1996)
Nature
, vol.379
, pp. 257-262
-
-
Akopian, A.N.1
Sivilotti, L.2
Wood, J.N.3
-
75
-
-
13344294399
-
Structure and function of a novel voltage-gated, tetrodoxtoxin-resistant sodium channel specific to sensory neurons
-
Sangameswaran L, Delgado SG, Fish LM. Structure and function of a novel voltage-gated, tetrodoxtoxin-resistant sodium channel specific to sensory neurons. J Biol Chem 1996, 271:5953-6.
-
(1996)
J Biol Chem
, vol.271
, pp. 5953-5956
-
-
Sangameswaran, L.1
Delgado, S.G.2
Fish, L.M.3
-
76
-
-
33846974357
-
Multiple sodium channels and their roles in electrogenesis within dorsal root ganglion neurons
-
Pt 1
-
Rush AM, Cummins TR, Waxman SG. Multiple sodium channels and their roles in electrogenesis within dorsal root ganglion neurons. J Physiol (Lond) 2007, 579:1-14. Pt 1
-
(2007)
J Physiol (Lond)
, vol.579
, pp. 1-14
-
-
Rush, A.M.1
Cummins, T.R.2
Waxman, S.G.3
-
77
-
-
30344431782
-
Familial rectal pain: A familial autonomic disorder as a cause of paroxysmal attacks in the newborn baby
-
Bednarek N, Arbues AS, Motte J. Familial rectal pain: A familial autonomic disorder as a cause of paroxysmal attacks in the newborn baby. Epileptic Disord 2005, 7:360-2.
-
(2005)
Epileptic Disord
, vol.7
, pp. 360-362
-
-
Bednarek, N.1
Arbues, A.S.2
Motte, J.3
-
78
-
-
33750440145
-
What's in a name-familial rectal pain syndrome becomes paroxysmal extreme pain disorder
-
Fertleman CR, Ferrie CD. What's in a name-familial rectal pain syndrome becomes paroxysmal extreme pain disorder. J Neurol Neurosurg Psychiatry 2006, 77:1294-5.
-
(2006)
J Neurol Neurosurg Psychiatry
, vol.77
, pp. 1294-1295
-
-
Fertleman, C.R.1
Ferrie, C.D.2
-
79
-
-
58149154859
-
Nav1.7 gain-of-function mutations as a continuum: A1632E displays physiological changes associated with erythromelalgia and paroxysmal extreme pain disorder mutations and produces symptoms of both disorders
-
Estacion M, Dib-Hajj SD, Benke PJ. Nav1.7 gain-of-function mutations as a continuum: A1632E displays physiological changes associated with erythromelalgia and paroxysmal extreme pain disorder mutations and produces symptoms of both disorders. J Neurosci 2008, 28:11079-88.
-
(2008)
J Neurosci
, vol.28
, pp. 11079-11088
-
-
Estacion, M.1
Dib-Hajj, S.D.2
Benke, P.J.3
-
80
-
-
53349171632
-
Paroxysmal extreme pain disorder M1627K mutation in human Nav1.7 renders DRG neurons hyperexcitable
-
Dib-Hajj SD, Estacion M, Jarecki BW. Paroxysmal extreme pain disorder M1627K mutation in human Nav1.7 renders DRG neurons hyperexcitable. Mol Pain 2008, 4:37.
-
(2008)
Mol Pain
, vol.4
, pp. 37
-
-
Dib-Hajj, S.D.1
Estacion, M.2
Jarecki, B.W.3
-
81
-
-
51749085389
-
Paroxysmal extreme pain disorder mutations within the D3/S4-S5 linker of Nav1.7 cause moderate destabilization of fast inactivation
-
Jarecki BW, Sheets PL, Jackson JO, Cummins TR. Paroxysmal extreme pain disorder mutations within the D3/S4-S5 linker of Nav1.7 cause moderate destabilization of fast inactivation. J Physiol 2008, 586:4137-53.
-
(2008)
J Physiol
, vol.586
, pp. 4137-4153
-
-
Jarecki, B.W.1
Sheets, P.L.2
Jackson, J.O.3
Cummins, T.R.4
-
82
-
-
34548419652
-
A stop codon mutation in SCN9A causes lack of pain sensation
-
Ahmad S, Dahllund L, Eriksson AB. A stop codon mutation in SCN9A causes lack of pain sensation. Hum Mol Genet 2007, 16:2114-21.
-
(2007)
Hum Mol Genet
, vol.16
, pp. 2114-2121
-
-
Ahmad, S.1
Dahllund, L.2
Eriksson, A.B.3
-
83
-
-
0030608445
-
Spinal sensory neurons express multiple sodium channel alpha-subunit mRNAs
-
Black JA, Dib-Hajj S, McNabola K. Spinal sensory neurons express multiple sodium channel alpha-subunit mRNAs. Mol Brain Res 1996, 43:117-31.
-
(1996)
Mol Brain Res
, vol.43
, pp. 117-131
-
-
Black, J.A.1
Dib-Hajj, S.2
McNabola, K.3
-
84
-
-
0033572137
-
A novel persistent tetrodotoxin-resistant sodium current in SNS-null and wild-type small primary sensory neurons
-
Cummins TR, Dib-Hajj SD, Black JA. A novel persistent tetrodotoxin-resistant sodium current in SNS-null and wild-type small primary sensory neurons. J Neurosci 1999, 19:RC43.
-
(1999)
J Neurosci
, vol.19
-
-
Cummins, T.R.1
Dib-Hajj, S.D.2
Black, J.A.3
-
85
-
-
0033529222
-
Sodium channels and pain
-
Waxman SG, Dib-Hajj S, Cummins TR, Black JA. Sodium channels and pain. Proc Natl Acad Sci USA 1999, 96:7635-9.
-
(1999)
Proc Natl Acad Sci USA
, vol.96
, pp. 7635-7639
-
-
Waxman, S.G.1
Dib-Hajj, S.2
Cummins, T.R.3
Black, J.A.4
|