Homologous recombination contributes to the repair of DNA double-strand breaks induced by high-energy iron ions

Radiation Research

Volumn 173, Issue 1, 2010, Pages 27-39

  Zafar, Faria ^a   Seidler, Sara B ^a   Kronenberg, Amy ^a   Schild, David ^a   Wiese, Claudia ^a  

^a LAWRENCE BERKELEY NATIONAL LABORATORY   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

DOUBLE STRANDED DNA; IRON; RAD51 PROTEIN; SMALL INTERFERING RNA;

ANIMAL CELL; ANIMAL CELL CULTURE; ARTICLE; CELL CYCLE G1 PHASE; CELL CYCLE S PHASE; CELL SURVIVAL; CONTROLLED STUDY; CYTOTOXICITY; DNA REPAIR; DNA STRAND BREAKAGE; ENERGY METABOLISM; EXPOSURE; FLOW CYTOMETRY; GENE DELETION; HIGH ENERGY IRON ION; HOMOLOGOUS RECOMBINATION; HUMAN; HUMAN CELL; HUMAN CELL CULTURE; IRRADIATION; MUTAGENESIS; MUTATIONAL ANALYSIS; NONHUMAN; PRIORITY JOURNAL; RADIOSENSITIVITY; RAT; RNA INTERFERENCE;

ANIMALS; BASE SEQUENCE; CELL SURVIVAL; CHO CELLS; CRICETINAE; CRICETULUS; DNA BREAKS, DOUBLE-STRANDED; DNA REPAIR; G2 PHASE; GENE KNOCKDOWN TECHNIQUES; HUMANS; IRON; MUTAGENESIS; MUTATION; RAD51 RECOMBINASE; RADIATION TOLERANCE; RECOMBINATION, GENETIC; S PHASE; THYMIDINE KINASE; TUMOR SUPPRESSOR PROTEIN P53;

RODENTIA;

EID: 73349132033     PISSN: 00337587     EISSN: None     Source Type: Journal    
DOI: 10.1667/RR1910.1     Document Type: Article

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