Histone H2AX is integral to hypoxia-driven neovascularization

Nature Medicine

Volumn 15, Issue 5, 2009, Pages 553-558

  Economopoulou, Matina ^a   Langer, Harald F ^a   Celeste, Arkady ^a   Orlova, Valeria V ^a   Choi, Eun Young ^a   Ma, Mingchao ^b   Vassilopoulos, Athanassios ^c   Callen, Elsa ^a   Deng, Chuxia ^c   Bassing, Craig H ^d   Boehm, Manfred ^b   Nussenzweig, Andre ^a   Chavakis, Triantafyllos ^a  

^a NATIONAL CANCER INSTITUTE   (United States)

^b NATIONAL HEART LUNG AND BLOOD INSTITUTE   (United States)

^c NATIONAL INSTITUTES OF HEALTH   (United States)

^d UNIVERSITY OF PENNSYLVANIA   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

DNA; GAMMA HISTONE H2AX; HISTONE H2AX; UNCLASSIFIED DRUG;

ANGIOGENESIS; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; CELL PROLIFERATION; CONTROLLED STUDY; DNA DAMAGE; DNA REPAIR; ENDOTHELIUM CELL; GENE; GENE DELETION; H2AFX GENE; HYPOXIA; LEG ISCHEMIA; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROLIFERATIVE RETINOPATHY; PROTEIN FUNCTION; TUMOR VASCULARIZATION;

ANIMALS; ANOXIA; DNA DAMAGE; ENDOTHELIUM, VASCULAR; GENE DELETION; HINDLIMB; HISTONES; HUMANS; HYDROXYUREA; ISCHEMIA; MICE; MICE, KNOCKOUT; NEOVASCULARIZATION, PATHOLOGIC; PHOSPHORYLATION; RETINAL VESSELS; UMBILICAL VEINS;

MUS;

EID: 65649113763     PISSN: 10788956     EISSN: 1546170X     Source Type: Journal    
DOI: 10.1038/nm.1947     Document Type: Article

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