Chronic copper exposure exacerbates both amyloid and tau pathology and selectively dysregulates cdk5 in a mouse model of AD

Journal of Neurochemistry

Volumn 108, Issue 6, 2009, Pages 1550-1560

  Kitazawa, Masashi ^a   Cheng, David ^a   Laferla, Frank M ^a  

^a UNIVERSITY OF CALIFORNIA   (United States)

Author keywords

Alzheimer; Kinase; Metal; Oligomer

Indexed keywords

AMYLOID BETA PROTEIN; COPPER; CYCLIN DEPENDENT KINASE 5; OLIGOMER; PROTEIN P25; TAU PROTEIN;

ALZHEIMER DISEASE; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; ENZYME ACTIVATION; MOUSE; NERVE CELL; NONHUMAN; PRIORITY JOURNAL; PROTEIN DEGRADATION; PROTEIN PHOSPHORYLATION;

ALZHEIMER DISEASE; AMYLOID; AMYLOID BETA-PROTEIN; AMYLOID BETA-PROTEIN PRECURSOR; ANIMALS; BRAIN; CALPAIN; CELL LINE, TUMOR; COPPER; CYCLIN-DEPENDENT KINASE 5; DISEASE MODELS, ANIMAL; ENZYME-LINKED IMMUNOSORBENT ASSAY; HUMANS; IMMUNOPRECIPITATION; MICE; MICE, TRANSGENIC; MUTATION; PEPTIDE FRAGMENTS; SUPEROXIDE DISMUTASE; TAU PROTEINS; TIME FACTORS; TRACE ELEMENTS;

MUS;

EID: 61349110122     PISSN: 00223042     EISSN: 14714159     Source Type: Journal    
DOI: 10.1111/j.1471-4159.2009.05901.x     Document Type: Article

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