PML tumor suppressor is regulated by HIPK2-mediated phosphorylation in response to DNA damage

Oncogene

Volumn 28, Issue 5, 2009, Pages 698-708

  Gresko, E ^a,b   Ritterhoff, S ^c   Sevilla Perez, J ^c   Roscic, A ^c,d   Frobius K ^c   Kotevic, I ^e   Vichalkovski, A ^b   Hess, D ^b   Hemmings, B A ^b   Schmitz, M L ^c  

^a UNIVERSITY OF BERN   (Switzerland)

^b FRIEDRICH MIESCHER INSTITUTE FOR BIOMEDICAL RESEARCH   (Switzerland)

^c JUSTUS LIEBIG UNIVERSITY   (Germany)

^d NOVARTIS PHARMA AG   (Switzerland)

^e ETH ZURICH   (Switzerland)

Author keywords

Cell death; HIPK2; Phosphorylation; PML

Indexed keywords

HOMEODOMAIN INTERACTING PROTEIN KINASE 2; HYBRID PROTEIN; PROMYELOCYTIC LEUKEMIA PROTEIN; RETINOIC ACID RECEPTOR ALPHA;

AMINO TERMINAL SEQUENCE; ARTICLE; CELL DEATH; CONTROLLED STUDY; DNA DAMAGE; ENZYME BINDING; HUMAN; HUMAN CELL; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN PHOSPHORYLATION; PROTEIN PROTEIN INTERACTION; PROTEIN STABILITY; SUMOYLATION;

CARRIER PROTEINS; CELL DEATH; CELLS, CULTURED; DNA DAMAGE; GENES, TUMOR SUPPRESSOR; HUMANS; NUCLEAR PROTEINS; PHOSPHORYLATION; PROTEIN BINDING; PROTEIN PROCESSING, POST-TRANSLATIONAL; PROTEIN STABILITY; PROTEIN STRUCTURE, TERTIARY; PROTEIN-SERINE-THREONINE KINASES; SERINE; SUMO-1 PROTEIN; TRANSCRIPTION FACTORS; TUMOR SUPPRESSOR PROTEINS;

EID: 59649100178     PISSN: 09509232     EISSN: 14765594     Source Type: Journal    
DOI: 10.1038/onc.2008.420     Document Type: Article

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