Promyelocytic leukemia nuclear bodies behave as DNA damage sensors whose response to DNA double-strand breaks is regulated by NBS1 and the kinases ATM, Chk2, and ATR

Journal of Cell Biology

Volumn 175, Issue 1, 2006, Pages 55-66

  Dellaire, Graham ^a,b   Ching, Reagan W ^a   Ahmed, Kashif ^a   Jalali, Farid ^c   Tse, Kenneth C K ^c   Bristow, Robert G ^c,d   Bazett Jones, David P ^a  

^a HOSPITAL FOR SICK CHILDREN   (Canada)

^b DALHOUSIE UNIVERSITY   (Canada)

^c PRINCESS MARGARET HOSPITAL   (Canada)

^d UNIVERSITY OF TORONTO   (Canada)

Author keywords

[No Author keywords available]

Indexed keywords

ATM PROTEIN; ATR PROTEIN; CAFFEINE; CHECKPOINT KINASE 2; DOXORUBICIN; NIBRIN; PROMYELOCYTIC LEUKEMIA PROTEIN; PROTEIN P53; SUMO 1 PROTEIN;

ARTICLE; CANCER INHIBITION; CELL CYCLE; CELL CYCLE PROGRESSION; CELL CYCLE S PHASE; CELL NUCLEUS INCLUSION BODY; CHROMATIN; CONTROLLED STUDY; DNA DAMAGE; DNA REPAIR; DNA REPLICATION; DNA STRAND BREAKAGE; DNA TRANSCRIPTION; HUMAN; HUMAN CELL; PRIORITY JOURNAL; SUPRAMOLECULAR CHEMISTRY;

CAFFEINE; CELL CYCLE PROTEINS; CELL NUCLEUS STRUCTURES; CHROMATIN; DNA DAMAGE; DNA REPAIR; DNA-BINDING PROTEINS; HUMANS; NUCLEAR PROTEINS; PROTEIN BIOSYNTHESIS; PROTEIN-SERINE-THREONINE KINASES; TUMOR SUPPRESSOR PROTEIN P53; TUMOR SUPPRESSOR PROTEINS;

EID: 33749547681     PISSN: 00219525     EISSN: 00219525     Source Type: Journal    
DOI: 10.1083/jcb.200604009     Document Type: Article

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