Apoptotic neutrophils and T cells sequester chemokines during immune response resolution through modulation of CCR5 expression

Nature Immunology

Volumn 7, Issue 11, 2006, Pages 1209-1216

  Ariel, Amiram ^a   Fredman, Gabrielle ^a   Sun, Yee Ping ^a   Kantarci, Alpdogan ^b   Van Dyke, Thomas E ^b   Luster, Andrew D ^c   Serhan, Charles N ^a  

^a BRIGHAM AND WOMEN S HOSPITAL   (United States)

^b BOSTON UNIVERSITY   (United States)

^c MASSACHUSETTS GENERAL HOSPITAL   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CD59 ANTIGEN; CHEMOKINE; CHEMOKINE RECEPTOR CCR5; LIPOXIN A; RANTES; TUMOR NECROSIS FACTOR;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; APOPTOSIS; ARTICLE; CELLULAR IMMUNITY; CONTROLLED STUDY; DOWN REGULATION; FEMALE; HUMAN; HUMAN CELL; LEUKOCYTE FUNCTION; MALE; MOUSE; NEUTROPHIL; NONHUMAN; PERITONITIS; POLYMORPHONUCLEAR CELL; PRIORITY JOURNAL; PROTEIN EXPRESSION; SIGNAL TRANSDUCTION; T LYMPHOCYTE; T LYMPHOCYTE ACTIVATION; UPREGULATION;

ANIMALS; APOPTOSIS; CHEMOKINES; FEMALE; HUMANS; INFLAMMATION MEDIATORS; MALE; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; NEUTROPHILS; RECEPTORS, CCR5; T-LYMPHOCYTES;

EID: 33750112087     PISSN: 15292908     EISSN: 15292916     Source Type: Journal    
DOI: 10.1038/ni1392     Document Type: Article

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