Lipid mediator class switching during acute inflammation: Signals in resolution

Nature Immunology

Volumn 2, Issue 7, 2001, Pages 612-619

  Levy, B D ^a   Clish, C B ^a   Schmidt, B ^a   Gronert, K ^a   Serhan, C N ^a  

^a BRIGHAM AND WOMEN S HOSPITAL   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ARACHIDONATE 5 LIPOXYGENASE; ICOSANOID; LEUKOTRIENE; LEUKOTRIENE B4; LIPID; LIPOXIN; LIPOXIN A; PROSTAGLANDIN; PROSTAGLANDIN E2;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ANTIINFLAMMATORY ACTIVITY; ARTICLE; CLINICAL STUDY; CONTROLLED STUDY; EXPERIMENTATION; EXPOSURE; EXUDATE; FATTY ACID SYNTHESIS; HUMAN; HUMAN CELL; HUMAN TISSUE; INFLAMMATION; MALE; MOUSE; NEUTROPHIL; NEUTROPHIL CHEMOTAXIS; NONHUMAN; PLEURA FLUID; PLEURISY; PRIORITY JOURNAL; SIGNAL TRANSDUCTION;

ANIMALS; ARACHIDONATE 15-LIPOXYGENASE; BASE SEQUENCE; DINOPROSTONE; DNA, COMPLEMENTARY; HUMANS; HYDROXYEICOSATETRAENOIC ACIDS; LEUKOTRIENE B4; LIPID METABOLISM; LIPIDS; LIPOXINS; MALE; MICE; MICE, INBRED BALB C; MOLECULAR SEQUENCE DATA; MOLECULAR STRUCTURE; NEUTROPHILS; PLEURAL EFFUSION; RNA, MESSENGER; SIGNAL TRANSDUCTION;

EID: 0034916569     PISSN: 15292908     EISSN: None     Source Type: Journal    
DOI: 10.1038/89759     Document Type: Article

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