-
1
-
-
0342819525
-
Compensatory hyperfunction of the heart and cardiac insufficiency
-
Meerson, F.Z. 1962. Compensatory hyperfunction of the heart and cardiac insufficiency. Circ. Res. 10:250-258.
-
(1962)
Circ. Res.
, vol.10
, pp. 250-258
-
-
Meerson, F.Z.1
-
2
-
-
0025280867
-
Prognostic implications of echocardiographically determined left ventricular mass in the Framingham Heart Study
-
Levy, D., Garrison, R.J., Savage, D.D., Kannel, W.B., and Castelli, W.P. 1990. Prognostic implications of echocardiographically determined left ventricular mass in the Framingham Heart Study. N. Engl. J. Med. 322:1561-1566.
-
(1990)
N. Engl. J. Med.
, vol.322
, pp. 1561-1566
-
-
Levy, D.1
Garrison, R.J.2
Savage, D.D.3
Kannel, W.B.4
Castelli, W.P.5
-
3
-
-
1842579393
-
Hypertrophy of the heart: A new therapeutic target?
-
Frey, N., Katus, H.A., Olson, E.N., and Hill, J.A. 2004. Hypertrophy of the heart: a new therapeutic target? Circulation. 109:1580-1589.
-
(2004)
Circulation
, vol.109
, pp. 1580-1589
-
-
Frey, N.1
Katus, H.A.2
Olson, E.N.3
Hill, J.A.4
-
4
-
-
0036143851
-
Genetic alterations that inhibit in vivo pressure-overload hypertrophy prevent cardiac dysfunction despite increased wall stress
-
Esposito, G., et al. 2002. Genetic alterations that inhibit in vivo pressure-overload hypertrophy prevent cardiac dysfunction despite increased wall stress. Circulation. 105:85-92.
-
(2002)
Circulation
, vol.105
, pp. 85-92
-
-
Esposito, G.1
-
5
-
-
0034688194
-
Effects of an angiotensin-converting-enzyme inhibitor, ramipril, on cardiovascular events in high-risk patients
-
The Heart Outcomes Prevention Evaluation Study Investigators
-
Yusuf, S., et al. 2000. Effects of an angiotensin-converting-enzyme inhibitor, ramipril, on cardiovascular events in high-risk patients. The Heart Outcomes Prevention Evaluation Study Investigators. N. Engl. J. Med. 342:145-153.
-
(2000)
N. Engl. J. Med.
, vol.342
, pp. 145-153
-
-
Yusuf, S.1
-
6
-
-
8544226268
-
Prognostic significance of left ventricular mass change during treatment of hypertension
-
Devereux, R.B., et al. 2004. Prognostic significance of left ventricular mass change during treatment of hypertension. JAMA. 292:2350-2356.
-
(2004)
JAMA
, vol.292
, pp. 2350-2356
-
-
Devereux, R.B.1
-
7
-
-
8544264609
-
Left ventricular hypertrophy: The next treatable, silent killer?
-
Gardin, J.M., and Lauer, M.S. 2004. Left ventricular hypertrophy: the next treatable, silent killer? JAMA. 292:2396-2398.
-
(2004)
JAMA
, vol.292
, pp. 2396-2398
-
-
Gardin, J.M.1
Lauer, M.S.2
-
8
-
-
0036172127
-
Fas receptor signaling inhibits glycogen synthase kinase 3β and induces cardiac hypertrophy following pressure overload
-
doi:10.1172/JCI200213779
-
Badorff, C., et al. 2002. Fas receptor signaling inhibits glycogen synthase kinase 3β and induces cardiac hypertrophy following pressure overload. J. Clin. Invest. 109:373-381. doi:10.1172/JCI200213779.
-
(2002)
J. Clin. Invest.
, vol.109
, pp. 373-381
-
-
Badorff, C.1
-
9
-
-
0037236642
-
Melusin, a muscle-specific integrin beta1-interacting protein, is required to prevent cardiac failure in response to chronic pressure overload
-
Brancaccio, M., et al. 2003. Melusin, a muscle-specific integrin beta1-interacting protein, is required to prevent cardiac failure in response to chronic pressure overload. Nat. Med. 9:68-75.
-
(2003)
Nat. Med.
, vol.9
, pp. 68-75
-
-
Brancaccio, M.1
-
10
-
-
0033574574
-
Loss of a gp130 cardiac muscle cell survival pathway is a critical event in the onset of heart failure during biomechanical stress
-
Hirota, H., et al. 1999. Loss of a gp130 cardiac muscle cell survival pathway is a critical event in the onset of heart failure during biomechanical stress. Cell. 97:189-198.
-
(1999)
Cell
, vol.97
, pp. 189-198
-
-
Hirota, H.1
-
11
-
-
0033515643
-
Cyclosporine attenuates pressure-overload hypertrophy in mice while enhancing susceptibility to decompensation and heart failure
-
Meguro, T., et al. 1999. Cyclosporine attenuates pressure-overload hypertrophy in mice while enhancing susceptibility to decompensation and heart failure. Circ. Res. 84:735-740.
-
(1999)
Circ. Res.
, vol.84
, pp. 735-740
-
-
Meguro, T.1
-
12
-
-
0032702983
-
RGS4 causes increased mortality and reduced cardiac hypertrophy in response to pressure overload
-
Rogers, J.H., et al. 1999. RGS4 causes increased mortality and reduced cardiac hypertrophy in response to pressure overload. J. Clin. Invest. 104:567-576.
-
(1999)
J. Clin. Invest.
, vol.104
, pp. 567-576
-
-
Rogers, J.H.1
-
13
-
-
85047693926
-
Inhibition of receptor-localized PI3K preserves cardiac β-adrenergic receptor function and ameliorates pressure overload heart failure
-
doi:10.1172/JCI200318213
-
Nienaber, J.J., et al. 2003. Inhibition of receptor-localized PI3K preserves cardiac β-adrenergic receptor function and ameliorates pressure overload heart failure. J. Clin. Invest. 112:1067-1079. doi:10.1172/ JCI200318213.
-
(2003)
J. Clin. Invest.
, vol.112
, pp. 1067-1079
-
-
Nienaber, J.J.1
-
14
-
-
13444292828
-
Level of betaadrenergic receptor kinase 1 inhibition determines degree of cardiac dysfunction after chronic pressure overload-induced heart failure
-
Tachibana, H., Naga Prasad, S.V., Lefkowitz, R.J., Koch, W.J., and Rockman, H.A. 2005. Level of betaadrenergic receptor kinase 1 inhibition determines degree of cardiac dysfunction after chronic pressure overload-induced heart failure. Circulation. 111:591-597.
-
(2005)
Circulation
, vol.111
, pp. 591-597
-
-
Tachibana, H.1
Naga Prasad, S.V.2
Lefkowitz, R.J.3
Koch, W.J.4
Rockman, H.A.5
-
15
-
-
0142027759
-
Phosphoinositide 3-kinase(p110alpha) plays a critical role for the induction of physiological, but not pathological, cardiac hypertrophy
-
McMullen, J.R., et al. 2003. Phosphoinositide 3-kinase(p110alpha) plays a critical role for the induction of physiological, but not pathological, cardiac hypertrophy. Proc. Natl. Acad. Sci. U. S. A. 100:12355-12360.
-
(2003)
Proc. Natl. Acad. Sci. U. S. A.
, vol.100
, pp. 12355-12360
-
-
McMullen, J.R.1
-
16
-
-
6044234749
-
Skeletal muscle adaptation in response to voluntary running in Ca2+/calmodulin-dependent protein kinase IV-deficient mice
-
Akimoto, T., Ribar, T.J., Williams, R.S., and Yan, Z. 2004. Skeletal muscle adaptation in response to voluntary running in Ca2+/calmodulin-dependent protein kinase IV-deficient mice. Am. J. Physiol. Cell Physiol. 287:C1311-C1319.
-
(2004)
Am. J. Physiol. Cell Physiol.
, vol.287
-
-
Akimoto, T.1
Ribar, T.J.2
Williams, R.S.3
Yan, Z.4
-
17
-
-
0025873338
-
Segregation of atrialspecific and inducible expression of an atrial natriuretic factor transgene in an in vivo murine model of cardiac hypertrophy
-
Rockman, H.A., et al. 1991. Segregation of atrialspecific and inducible expression of an atrial natriuretic factor transgene in an in vivo murine model of cardiac hypertrophy. Proc. Natl. Acad. Sci. U. S. A. 88:8277-8281.
-
(1991)
Proc. Natl. Acad. Sci. U. S. A.
, vol.88
, pp. 8277-8281
-
-
Rockman, H.A.1
-
18
-
-
0034857217
-
Important role of endogenous norepinephrine and epinephrine in the development of in vivo pressure-overload carcardiac hypertrophy
-
Rapacciuolo, A., et al. 2001. Important role of endogenous norepinephrine and epinephrine in the development of in vivo pressure-overload carcardiac hypertrophy. J. Am. Coll. Cardiol. 38:876-882.
-
(2001)
J. Am. Coll. Cardiol.
, vol.38
, pp. 876-882
-
-
Rapacciuolo, A.1
-
19
-
-
0020688573
-
Sympathetic nervous activity during exercise
-
Christensen, N.J., and Galbo, H. 1983. Sympathetic nervous activity during exercise. Annu. Rev. Physiol. 45:139-153.
-
(1983)
Annu. Rev. Physiol.
, vol.45
, pp. 139-153
-
-
Christensen, N.J.1
Galbo, H.2
-
20
-
-
0018778154
-
Conditioning effects of chronic infusions of dobutamine. Comparison with exercise training
-
Liang, C., Tuttle, R.R., Hood, W.B., Jr., and Gavras, H. 1979. Conditioning effects of chronic infusions of dobutamine. Comparison with exercise training. J. Clin. Invest. 64:613-619.
-
(1979)
J. Clin. Invest.
, vol.64
, pp. 613-619
-
-
Liang, C.1
Tuttle, R.R.2
Hood Jr., W.B.3
Gavras, H.4
-
21
-
-
0030685743
-
Changes in gene expression in the intact human heart. Downregulation of α-myosin heavy chain in hypertrophied, failing ventricular myocardium
-
Lowes, B.D., et al. 1997. Changes in gene expression in the intact human heart. Downregulation of α-myosin heavy chain in hypertrophied, failing ventricular myocardium. J. Clin. Invest. 100:2315-2324.
-
(1997)
J. Clin. Invest.
, vol.100
, pp. 2315-2324
-
-
Lowes, B.D.1
-
22
-
-
0030671503
-
Myosin heavy chain gene expression in human heart failure
-
Nakao, K., Minobe, W., Roden, R., Bristow, M.R., and Leinwand, L.A. 1997. Myosin heavy chain gene expression in human heart failure. J. Clin. Invest. 100:2362-2370.
-
(1997)
J. Clin. Invest.
, vol.100
, pp. 2362-2370
-
-
Nakao, K.1
Minobe, W.2
Roden, R.3
Bristow, M.R.4
Leinwand, L.A.5
-
23
-
-
0033529637
-
Defective beta-adrenergic receptor signaling precedes the development of dilated cardiomyopathy in transgenic mice with calsequestrin overexpression
-
Cho, M.C., et al. 1999. Defective beta-adrenergic receptor signaling precedes the development of dilated cardiomyopathy in transgenic mice with calsequestrin overexpression. J. Biol. Chem. 274:22251-22256.
-
(1999)
J. Biol. Chem.
, vol.274
, pp. 22251-22256
-
-
Cho, M.C.1
-
24
-
-
0037050009
-
Seven-transmembrane-spanning receptors and heart function
-
Rockman, H.A., Koch, W.J., and Lefkowitz, R.J. 2002. Seven-transmembrane- spanning receptors and heart function. Nature. 415:206-212.
-
(2002)
Nature
, vol.415
, pp. 206-212
-
-
Rockman, H.A.1
Koch, W.J.2
Lefkowitz, R.J.3
-
25
-
-
0030798818
-
Mechanism of beta-adrenergic receptor desensitization in cardiac hypertrophy is increased beta-adrenergic receptor kinase
-
Choi, D.J., Koch, W.J., Hunter, J.J., and Rockman, H.A. 1997. Mechanism of beta-adrenergic receptor desensitization in cardiac hypertrophy is increased beta-adrenergic receptor kinase. J. Biol. Chem. 272:17223-17229.
-
(1997)
J. Biol. Chem.
, vol.272
, pp. 17223-17229
-
-
Choi, D.J.1
Koch, W.J.2
Hunter, J.J.3
Rockman, H.A.4
-
26
-
-
20344402530
-
Targeted inhibition of beta-adrenergic receptor kinase-1-associated phosphoinositide-3 kinase activity preserves beta-adrenergic receptor signaling and prolongs survival in heart failure induced by calsequestrin overexpression
-
Perrino, C., Naga Prasad, S.V., Patel, M., Wolf, M.J., and Rockman, H.A. 2005. Targeted inhibition of beta-adrenergic receptor kinase-1-associated phosphoinositide-3 kinase activity preserves beta-adrenergic receptor signaling and prolongs survival in heart failure induced by calsequestrin overexpression. J. Am. Coll. Cardiol. 45:1862-1870.
-
(2005)
J. Am. Coll. Cardiol.
, vol.45
, pp. 1862-1870
-
-
Perrino, C.1
Naga Prasad, S.V.2
Patel, M.3
Wolf, M.J.4
Rockman, H.A.5
-
27
-
-
19644397920
-
Restoration of beta-adrenergic receptor signaling and contractile function in heart failure by disruption of the betaARK1/phosphoinositide 3-kinase complex
-
Perrino, C., et al. 2005. Restoration of beta-adrenergic receptor signaling and contractile function in heart failure by disruption of the betaARK1/phosphoinositide 3-kinase complex. Circulation. 111:2579-2587.
-
(2005)
Circulation
, vol.111
, pp. 2579-2587
-
-
Perrino, C.1
-
28
-
-
0036074371
-
The MEKK1-JNK pathway plays a protective role in pressure overload but does not mediate cardiac hypertrophy
-
doi:10.1172/JCI200214938
-
Sadoshima, J., et al. 2002. The MEKK1-JNK pathway plays a protective role in pressure overload but does not mediate cardiac hypertrophy. J. Clin. Invest. 110:271-279. doi:10.1172/JCI200214938.
-
(2002)
J. Clin. Invest.
, vol.110
, pp. 271-279
-
-
Sadoshima, J.1
-
29
-
-
0034105968
-
TAK1 is activated in the myocardium after pressure overload and is sufficient to provoke heart failure in transgenic mice
-
Zhang, D., et al. 2000. TAK1 is activated in the myocardium after pressure overload and is sufficient to provoke heart failure in transgenic mice. Nat. Med. 6:556-563.
-
(2000)
Nat. Med.
, vol.6
, pp. 556-563
-
-
Zhang, D.1
-
30
-
-
0036083359
-
Heart size-independent analysis of myocardial function in murine pressure overload hypertrophy
-
Takaoka, H., Esposito, G., Mao, L., Suga, H., and Rockman, H.A. 2002. Heart size-independent analysis of myocardial function in murine pressure overload hypertrophy. Am. J. Physiol. Heart Circ. Physiol. 282:H2190-H2197.
-
(2002)
Am. J. Physiol. Heart Circ. Physiol.
, vol.282
-
-
Takaoka, H.1
Esposito, G.2
Mao, L.3
Suga, H.4
Rockman, H.A.5
-
31
-
-
0346390805
-
Hyperfunction with normal inotropic state of the hypertrophied left ventricle
-
Sasayama, S., Franklin, D., and Ross, J., Jr. 1977. Hyperfunction with normal inotropic state of the hypertrophied left ventricle. Am. J. Physiol. 232:H418-H425.
-
(1977)
Am. J. Physiol.
, vol.232
-
-
Sasayama, S.1
Franklin, D.2
Ross Jr., J.3
-
32
-
-
4344714889
-
PI3Kgamma modulates the cardiac response to chronic pressure overload by distinct kinase-dependent and -independent effects
-
Patrucco, E., et al. 2004. PI3Kgamma modulates the cardiac response to chronic pressure overload by distinct kinase-dependent and -independent effects. Cell. 118:375-387.
-
(2004)
Cell
, vol.118
, pp. 375-387
-
-
Patrucco, E.1
-
33
-
-
23644439061
-
Disruption of coordinated cardiac hypertrophy and angiogenesis contributes to the transition to heart failure
-
doi:10.1172/JCI24682
-
Shiojima, I., et al. 2005. Disruption of coordinated cardiac hypertrophy and angiogenesis contributes to the transition to heart failure. J. Clin. Invest. 115:2108-2118. doi:10.1172/JCI24682.
-
(2005)
J. Clin. Invest.
, vol.115
, pp. 2108-2118
-
-
Shiojima, I.1
-
34
-
-
17644402459
-
Transduction of receptor signals by beta-arrestins
-
Lefkowitz, R.J., and Shenoy, S.K. 2005. Transduction of receptor signals by beta-arrestins. Science. 308:512-517.
-
(2005)
Science
, vol.308
, pp. 512-517
-
-
Lefkowitz, R.J.1
Shenoy, S.K.2
-
35
-
-
27644477496
-
When 7 transmembrane receptors are not G protein-coupled receptors
-
doi:10.1172/JCI26950
-
Rajagopal, K., Lefkowitz, R.J., and Rockman, H.A. 2005. When 7 transmembrane receptors are not G protein-coupled receptors. J. Clin. Invest. 115:2971-2974. doi:10.1172/JCI26950.
-
(2005)
J. Clin. Invest.
, vol.115
, pp. 2971-2974
-
-
Rajagopal, K.1
Lefkowitz, R.J.2
Rockman, H.A.3
-
36
-
-
2942751868
-
Mechanical stress activates angiotensin II type 1 receptor without the involvement of angiotensin II
-
Zou, Y., et al. 2004. Mechanical stress activates angiotensin II type 1 receptor without the involvement of angiotensin II. Nat. Cell Biol. 6:499-506.
-
(2004)
Nat. Cell Biol.
, vol.6
, pp. 499-506
-
-
Zou, Y.1
-
37
-
-
0034599128
-
Myosin heavy chain isoform expression in the failing and nonfailing human heart
-
Miyata, S., Minobe, W., Bristow, M.R., and Leinwand, L.A. 2000. Myosin heavy chain isoform expression in the failing and nonfailing human heart. Circ. Res. 86:386-390.
-
(2000)
Circ. Res.
, vol.86
, pp. 386-390
-
-
Miyata, S.1
Minobe, W.2
Bristow, M.R.3
Leinwand, L.A.4
-
38
-
-
0037007679
-
Myocardial gene expression in dilated cardiomyopathy treated with beta-blocking agents
-
Lowes, B.D., et al. 2002. Myocardial gene expression in dilated cardiomyopathy treated with beta-blocking agents. N. Engl. J. Med. 346:1357-1365.
-
(2002)
N. Engl. J. Med.
, vol.346
, pp. 1357-1365
-
-
Lowes, B.D.1
-
39
-
-
14644414790
-
Protein kinase cascades in the regulation of cardiac hypertrophy
-
doi:10.1172/JCI200524178
-
Dorn, G.W., II, and Force, T. 2005. Protein kinase cascades in the regulation of cardiac hypertrophy. J. Clin. Invest. 115:527-537. doi:10.1172/JCI200524178.
-
(2005)
J. Clin. Invest.
, vol.115
, pp. 527-537
-
-
Dorn II, G.W.1
Force, T.2
-
40
-
-
0034383952
-
The MEK1-ERK1/2 signaling pathway promotes compensated cardiac hypertrophy in transgenic mice
-
Bueno, O.F., et al. 2000. The MEK1-ERK1/2 signaling pathway promotes compensated cardiac hypertrophy in transgenic mice. EMBO J. 19:6341-6350.
-
(2000)
EMBO J.
, vol.19
, pp. 6341-6350
-
-
Bueno, O.F.1
-
41
-
-
0018157075
-
Time constant of isovolumic pressure fall: Determinants in the working left ventricle
-
Frederiksen, J.W., Weiss, J.L., and Weisfeldt, M.L. 1978. Time constant of isovolumic pressure fall: determinants in the working left ventricle. Am. J. Physiol. 235:H701-H706.
-
(1978)
Am. J. Physiol.
, vol.235
-
-
Frederiksen, J.W.1
Weiss, J.L.2
Weisfeldt, M.L.3
-
42
-
-
0019034028
-
Myocardial relaxation. II. Hemodynamic determinants of rate of left ventricular isovolumic pressure decline
-
Gaasch, W.H., Blaustein, A.S., Andrias, C.W., Donahue, R.P., and Avitall, B. 1980. Myocardial relaxation. II. Hemodynamic determinants of rate of left ventricular isovolumic pressure decline. Am. J. Physiol. 239:H1-H6.
-
(1980)
Am. J. Physiol.
, vol.239
-
-
Gaasch, W.H.1
Blaustein, A.S.2
Andrias, C.W.3
Donahue, R.P.4
Avitall, B.5
-
43
-
-
18644372367
-
Regulation of myocardial contractility and cell size by distinct PI3KPTEN signaling pathways
-
Crackower, M.A., et al. 2002. Regulation of myocardial contractility and cell size by distinct PI3KPTEN signaling pathways. Cell. 110:737-749.
-
(2002)
Cell
, vol.110
, pp. 737-749
-
-
Crackower, M.A.1
-
44
-
-
23144450820
-
Protein kinase activity of phosphoinositide 3-kinase regulates beta-adrenergic receptor endocytosis
-
Naga Prasad, S.V., Jayatilleke, A., Madamanchi, A., and Rockman, H.A. 2005. Protein kinase activity of phosphoinositide 3-kinase regulates beta-adrenergic receptor endocytosis. Nat. Cell Biol. 7:785-796.
-
(2005)
Nat. Cell Biol.
, vol.7
, pp. 785-796
-
-
Naga Prasad, S.V.1
Jayatilleke, A.2
Madamanchi, A.3
Rockman, H.A.4
-
45
-
-
0036327388
-
Phosphoinositide 3-kinase regulates beta2-adrenergic receptor endocytosis by AP-2 recruitment to the receptor/betaarrestin complex
-
Naga Prasad, S.V., et al. 2002. Phosphoinositide 3-kinase regulates beta2-adrenergic receptor endocytosis by AP-2 recruitment to the receptor/betaarrestin complex. J. Cell Biol. 158:563-575.
-
(2002)
J. Cell Biol.
, vol.158
, pp. 563-575
-
-
Naga Prasad, S.V.1
-
46
-
-
0028841211
-
Sympathetic neural mechanisms in obstructive sleep apnea
-
Somers, V.K., Dyken, M.E., Clary, M.P., and Abboud, F.M. 1995. Sympathetic neural mechanisms in obstructive sleep apnea. J. Clin. Invest. 96:1897-1904.
-
(1995)
J. Clin. Invest.
, vol.96
, pp. 1897-1904
-
-
Somers, V.K.1
Dyken, M.E.2
Clary, M.P.3
Abboud, F.M.4
-
47
-
-
0034925133
-
Impairment of vascular endothelial function and left ventricular filling: Association with the severity of apnea-induced hypoxemia during sleep
-
Kraiczi, H., Caidahl, K., Samuelsson, A., Peker, Y., and Hedner, J. 2001. Impairment of vascular endothelial function and left ventricular filling: association with the severity of apnea-induced hypoxemia during sleep. Chest. 119:1085-1091.
-
(2001)
Chest
, vol.119
, pp. 1085-1091
-
-
Kraiczi, H.1
Caidahl, K.2
Samuelsson, A.3
Peker, Y.4
Hedner, J.5
-
48
-
-
0035148723
-
Sleep-disordered breathing and cardiovascular disease: Cross-sectional results of the Sleep Heart Health Study
-
Shahar, E., et al. 2001. Sleep-disordered breathing and cardiovascular disease: cross-sectional results of the Sleep Heart Health Study. Am. J. Respir. Crit. Care Med. 163:19-25.
-
(2001)
Am. J. Respir. Crit. Care Med.
, vol.163
, pp. 19-25
-
-
Shahar, E.1
-
49
-
-
0035826710
-
Cardiac beta ARK1 inhibition prolongs survival and augments beta blocker therapy in a mouse model of severe heart failure
-
Harding, V.B., Jones, L.R., Lefkowitz, R.J., Koch, W.J., and Rockman, H.A. 2001. Cardiac beta ARK1 inhibition prolongs survival and augments beta blocker therapy in a mouse model of severe heart failure. Proc. Natl. Acad. Sci. U. S. A. 98:5809-5814.
-
(2001)
Proc. Natl. Acad. Sci. U. S. A.
, vol.98
, pp. 5809-5814
-
-
Harding, V.B.1
Jones, L.R.2
Lefkowitz, R.J.3
Koch, W.J.4
Rockman, H.A.5
-
50
-
-
0036070468
-
Ventricular expression of natriuretic peptides in Npr1(-/-) mice with cardiac hypertrophy and fibrosis
-
Ellmers, L.J., et al. 2002. Ventricular expression of natriuretic peptides in Npr1(-/-) mice with cardiac hypertrophy and fibrosis. Am. J. Physiol. Heart Circ. Physiol. 283:H707-H714.
-
(2002)
Am. J. Physiol. Heart Circ. Physiol.
, vol.283
-
-
Ellmers, L.J.1
-
51
-
-
0035063196
-
Pressure-independent enhancement of cardiac hypertrophy in natriuretic peptide receptor A-deficient mice
-
Knowles, J.W., et al. 2001. Pressure-independent enhancement of cardiac hypertrophy in natriuretic peptide receptor A-deficient mice. J. Clin. Invest. 107:975-984.
-
(2001)
J. Clin. Invest.
, vol.107
, pp. 975-984
-
-
Knowles, J.W.1
-
52
-
-
0141676781
-
Vascular rarefaction in peripheral skeletal muscle after experimental heart failure
-
Nusz, D.J., et al. 2003. Vascular rarefaction in peripheral skeletal muscle after experimental heart failure. Am. J. Physiol. Heart Circ. Physiol. 285:H1554-H1562.
-
(2003)
Am. J. Physiol. Heart Circ. Physiol.
, vol.285
-
-
Nusz, D.J.1
-
53
-
-
0032537621
-
Myocyte recovery after mechanical circulatory support in humans with end-stage heart failure
-
Dipla, K., Mattiello, J.A., Jeevanandam, V., Houser, S.R., and Margulies, K.B. 1998. Myocyte recovery after mechanical circulatory support in humans with end-stage heart failure. Circulation. 97:2316-2322.
-
(1998)
Circulation
, vol.97
, pp. 2316-2322
-
-
Dipla, K.1
Mattiello, J.A.2
Jeevanandam, V.3
Houser, S.R.4
Margulies, K.B.5
-
54
-
-
0035375104
-
Agonistdependent recruitment of phosphoinositide 3-kinase to the membrane by beta-adrenergic receptor kinase 1. A role in receptor sequestration
-
Naga Prasad, S.V., Barak, L.S., Rapacciuolo, A., Caron, M.G., and Rockman, H.A. 2001. Agonistdependent recruitment of phosphoinositide 3-kinase to the membrane by beta-adrenergic receptor kinase 1. A role in receptor sequestration. J. Biol. Chem. 276:18953-18959.
-
(2001)
J. Biol. Chem.
, vol.276
, pp. 18953-18959
-
-
Naga Prasad, S.V.1
Barak, L.S.2
Rapacciuolo, A.3
Caron, M.G.4
Rockman, H.A.5
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