Senescence-associated phenotypes in Akita diabetic mice are enhanced by absence of bradykinin B2 receptors

Journal of Clinical Investigation

Volumn 116, Issue 5, 2006, Pages 1302-1309

  Kakoki, Masao ^a   Kizer, Catherine M ^a   Yi, Xianwen ^a   Takahashi, Nobuyuki ^a   Kim, Hyung Suk ^a   Bagnell, C Robert ^a   Edgell, Cora Jean S ^a   Maeda, Nobuyo ^a   Jennette, J Charles ^a   Smithies, Oliver ^a  

^a UNIVERSITY OF NORTH CAROLINA SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

8 HYDROXYDEOXYGUANOSINE; ALPHA SYNUCLEIN; BRADYKININ B2 RECEPTOR; CONNECTIVE TISSUE GROWTH FACTOR; GLUTATHIONE; MITOCHONDRIAL DNA; PROTEIN P53; THIOBARBITURIC ACID;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; APOPTOSIS; ARTICLE; CONTROLLED STUDY; DELETION MUTANT; DIABETES MELLITUS; DNA DAMAGE; ERYTHROCYTE; GENE; GENE EXPRESSION; INS2 GENE; MOUSE; NICK END LABELING; NONHUMAN; OXIDATIVE STRESS; PHENOTYPE; POINT MUTATION; PRIORITY JOURNAL; REVERSE TRANSCRIPTION POLYMERASE CHAIN REACTION; SENESCENCE;

ANIMALS; APOPTOSIS; CELL AGING; DEOXYGUANOSINE; DIABETES MELLITUS; DISEASE MODELS, ANIMAL; DNA, MITOCHONDRIAL; GENE EXPRESSION REGULATION; GENES, DOMINANT; MICE; MICE, INBRED C57BL; MICE, TRANSGENIC; OXIDATIVE STRESS; RECEPTOR, BRADYKININ B2;

EID: 33646408788     PISSN: 00219738     EISSN: 15588238     Source Type: Journal    
DOI: 10.1172/JCI26958     Document Type: Article

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