Neuroprotection against ischemic brain injury by a small peptide inhibitor of c-Jun N-terminal kinase (JNK) via nuclear and non-nuclear pathways

Neuroscience

Volumn 139, Issue 2, 2006, Pages 609-627

  Guan, Q H ^a,b   Pei, D S ^a,b   Zong, Y Y ^a   Xu, T L ^b,c   Zhang, G Y ^a,b  

^a XUZHOU MEDICAL UNIVERSITY   (China)

^b UNIVERSITY OF SCIENCE AND TECHNOLOGY OF CHINA   (China)

^c Shanghai Jiao Tong University Affiliated Sixth People s Hospital   (China)

Author keywords

c Jun N terminal protein kinase; cerebral ischemia reperfusion; JIP 1; JNK; JNK interacting protein 1; Tat JBD

Indexed keywords

ADENOSINE DIPHOSPHATE RIBOSE; PROTEIN; PROTEIN BCL 2; PROTEIN TAT JBD; STRESS ACTIVATED PROTEIN KINASE; STRESS ACTIVATED PROTEIN KINASE INHIBITOR; UNCLASSIFIED DRUG;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL TISSUE; APOPTOSIS; ARTICLE; BRAIN ISCHEMIA; CONTROLLED STUDY; ENZYME ACTIVATION; ENZYME BINDING; ENZYME PHOSPHORYLATION; GENETIC TRANSDUCTION; HIPPOCAMPUS; MALE; NEUROPROTECTION; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN PROTEIN INTERACTION; RAT;

ANALYSIS OF VARIANCE; ANIMALS; ANTIGENS, CD95; BCL-2-ASSOCIATED X PROTEIN; BRAIN INJURIES; BRAIN ISCHEMIA; CASPASE 3; CASPASES; CELLS, CULTURED; DISEASE MODELS, ANIMAL; EMBRYO; FAS LIGAND PROTEIN; GENE EXPRESSION; GENE PRODUCTS, TAT; HIPPOCAMPUS; JNK MITOGEN-ACTIVATED PROTEIN KINASES; MALE; MEMBRANE GLYCOPROTEINS; NEURONS; NEUROPROTECTIVE AGENTS; PEPTIDES; PROTO-ONCOGENE PROTEINS C-BCL-2; RATS; SIGNAL TRANSDUCTION; SUBCELLULAR FRACTIONS; TUMOR NECROSIS FACTORS;

EID: 33645870441     PISSN: 03064522     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.neuroscience.2005.11.067     Document Type: Article

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