Programmed Cell Death in Amyotrophic Lateral Sclerosis: A Mechanism of Pathogenic and Therapeutic Importance

Neurologist

Volumn 10, Issue 1, 2004, Pages 1-7

  Przedborski, Serge ^a  

^a Och Spine at New York Presbyterian Hospitals   (United States)

Author keywords

ALS; Apoptosis; Neurodegeneration; Neuroprotection

Indexed keywords

CASPASE; PROTEIN BCL 2; PROTEIN BCL XL;

AMYOTROPHIC LATERAL SCLEROSIS; APOPTOSIS; CELL DEATH; ENZYME ACTIVITY; HUMAN; MOTONEURON; NERVE DEGENERATION; NEUROPROTECTION; PATHOPHYSIOLOGY; PRIORITY JOURNAL; REVIEW; SIGNAL TRANSDUCTION;

AMYOTROPHIC LATERAL SCLEROSIS; ANIMALS; APOPTOSIS; CASPASES; DNA FRAGMENTATION; GENES, BCL-2; HUMANS; MICE; MICE, TRANSGENIC; MITOGEN-ACTIVATED PROTEIN KINASES; MOTOR NEURONS;

EID: 0942287085     PISSN: 10747931     EISSN: None     Source Type: Journal    
DOI: 10.1097/01.nrl.0000106920.84668.37     Document Type: Review

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