Accumulation of SOD1 Mutants in Postnatal Motoneurons Does Not Cause Motoneuron Pathology or Motoneuron Disease

Journal of Neuroscience

Volumn 22, Issue 12, 2002, Pages 4825-4832

  Lino, Maria Maddalena ^a   Schneider, Corinna ^a   Caroni, Pico ^a  

^a FRIEDRICH MIESCHER INSTITUTE FOR BIOMEDICAL RESEARCH   (Switzerland)

Author keywords

Amyotrophic lateral sclerosis; Motoneuron; Mutant SOD1; Neurodegeneration; Neuronal pathology; Pathophysiological process

Indexed keywords

SUPEROXIDE DISMUTASE; COPPER ZINC SUPEROXIDE DISMUTASE; THY 1 ANTIGEN;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CLINICAL FEATURE; CONTROLLED STUDY; GENE MUTATION; MOTOR NEURON DISEASE; MOUSE; MUSCLE INNERVATION; NONHUMAN; PATHOGENESIS; PRIORITY JOURNAL; PROTEIN EXPRESSION; SPINAL CORD; TRANSGENIC MOUSE; AMYOTROPHIC LATERAL SCLEROSIS; ANIMAL; BIOLOGICAL MODEL; CYTOLOGY; DISEASE COURSE; GENETICS; KINETICS; METABOLISM; MOTONEURON; MUTATION; NEWBORN; PATHOLOGY;

AMYOTROPHIC LATERAL SCLEROSIS; ANIMALS; ANIMALS, NEWBORN; ANTIGENS, THY-1; DISEASE PROGRESSION; KINETICS; MICE; MICE, TRANSGENIC; MODELS, NEUROLOGICAL; MOTOR NEURONS; MUTATION; SUPEROXIDE DISMUTASE;

EID: 0037096354     PISSN: 02706474     EISSN: None     Source Type: Journal    
DOI: 10.1523/jneurosci.22-12-04825.2002     Document Type: Article

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