BAX and BAK mediate p53-independent suppression of tumorigenesis

Cancer Cell

Volumn 2, Issue 3, 2002, Pages 193-203

  Degenhardt, Kurt ^b   Chen, Guanghua ^a,b   Lindsten, Tullia ^d   White, Eileen ^a,b,c  

^a RUTGERS UNIVERSITY   (United States)

^b RUTGERS UNIVERSITY   (United States)

^c RUTGERS CANCER INSTITUTE OF NEW JERSEY   (United States)

^d UNIVERSITY OF PENNSYLVANIA   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

E1A PROTEIN; PROTEIN BAK; PROTEIN BAX; PROTEIN P53; BAK1 PROTEIN, MOUSE; BAX PROTEIN, MOUSE; MEMBRANE PROTEIN; ONCOPROTEIN; PROTEIN BCL 2;

ADENOVIRUS; ANIMAL CELL; APOPTOSIS; ARTICLE; CANCER INHIBITION; CANCER INVASION; CARCINOGENESIS; CELL DISRUPTION; CELL TRANSFORMATION; CONTROLLED STUDY; EPITHELIUM CELL; KIDNEY EPITHELIUM; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN FUNCTION; PROTEIN SYNTHESIS INHIBITION; SIGNAL TRANSDUCTION; WILD TYPE; ANIMAL; CELL LINE; GENETICS; HETEROZYGOSITY LOSS; KIDNEY; METABOLISM; NEOPLASM; NUDE MOUSE; PATHOLOGY; PHYSIOLOGY; TUMOR SUPPRESSOR GENE; XENOGRAFT;

ADENOVIRIDAE;

ANIMALS; APOPTOSIS; BCL-2 HOMOLOGOUS ANTAGONIST-KILLER PROTEIN; BCL-2-ASSOCIATED X PROTEIN; CELL LINE, TRANSFORMED; EPITHELIAL CELLS; GENES, P53; KIDNEY; LOSS OF HETEROZYGOSITY; MEMBRANE PROTEINS; MICE; MICE, NUDE; NEOPLASMS; PROTO-ONCOGENE PROTEINS; PROTO-ONCOGENE PROTEINS C-BCL-2; TRANSPLANTATION, HETEROLOGOUS;

EID: 0036729911     PISSN: 15356108     EISSN: None     Source Type: Journal    
DOI: 10.1016/S1535-6108(02)00126-5     Document Type: Article

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