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Volumn 20, Issue 17, 2001, Pages 2080-2090

Constitutively active STAT5 variants induce growth and survival of hematopoietic cells through a PI 3-kinase/Akt dependent pathway

(7) Santos, Susana Constantino Rosa a Lacronique, Virginie b Bouchaert, Isabelle a Monni, Richard b Bernard, Olivier b Gisselbrecht, Sylvie a Gouilleux, Fabrice a

a Hôpital Cochin ^* (France)

b FONDATION JEAN DAUSSET CEPH (France)

Author keywords

PI 3 kinase; STAT5; Survival; TEL JAK2

Indexed keywords

2 MORPHOLINO 8 PHENYLCHROMONE; HYBRID PROTEIN; ONCOPROTEIN; PHOSPHATIDYLINOSITOL 3 KINASE; STAT5 PROTEIN;

ANIMAL CELL; APOPTOSIS; ARTICLE; CELL GROWTH; CELL SURVIVAL; CELL TRANSFORMATION; DNA BINDING; HEMATOPOIETIC CELL; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROTEIN PHOSPHORYLATION; SIGNAL TRANSDUCTION;

1-PHOSPHATIDYLINOSITOL 3-KINASE; ANIMALS; CELL CYCLE; CELL DIVISION; CELL LINE; CELL SURVIVAL; DNA-BINDING PROTEINS; ENZYME ACTIVATION; ERYTHROPOIETIN; FUSION PROTEINS, BCR-ABL; HEMATOPOIETIC STEM CELLS; HUMANS; MICE; MILK PROTEINS; ONCOGENE PROTEINS, FUSION; PHOSPHORYLATION; PROTEIN-SERINE-THREONINE KINASES; PROTEIN-TYROSINE KINASES; PROTO-ONCOGENE PROTEINS; PROTO-ONCOGENE PROTEINS C-AKT; SIGNAL TRANSDUCTION; STAT5 TRANSCRIPTION FACTOR; TRANS-ACTIVATORS; TRANSFORMATION, GENETIC;

ANIMALIA;

EID: 0035912097 PISSN: 09509232 EISSN: None Source Type: Journal
DOI: 10.1038/sj.onc.1204308 Document Type: Article

Times cited : (68)

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