A dual role for TNF-α in type 1 diabetes: Islet-specific expression abrogates the ongoing autoimmune process when induced late but not early during pathogenesis

Journal of Immunology

Volumn 166, Issue 12, 2001, Pages 7023-7032

  Christen, U ^a   Wolfe, T ^a   Mohrle U ^a   Hughes, A C ^a   Rodrigo, E ^a   Green, E A ^a   Flavell, R A ^a   Von Herrath, M G ^a  

^a SCRIPPS RESEARCH INSTITUTE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

TUMOR NECROSIS FACTOR ALPHA;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CYTOKINE PRODUCTION; IMMUNE RESPONSE; IMMUNOHISTOCHEMISTRY; INCIDENCE; INSULIN DEPENDENT DIABETES MELLITUS; LYMPHOCYTE; LYMPHOCYTIC CHORIOMENINGITIS VIRUS; MOUSE; NONHUMAN; PATHOGENESIS; PRIORITY JOURNAL; TRANSGENIC MOUSE;

EID: 0035877181     PISSN: 00221767     EISSN: None     Source Type: Journal    
DOI: 10.4049/jimmunol.166.12.7023     Document Type: Article

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