Local expression of TNFα in neonatal NOD mice promotes diabetes by enhancing presentation of islet antigens

Immunity

Volumn 9, Issue 5, 1998, Pages 733-743

  Green, E Allison ^b   Eynon, Elizabeth E ^b   Flaveirti, Richard A ^a  

^a HOWARD HUGHES MEDICAL INSTITUTE   (United States)

^b NONE

Author keywords

[No Author keywords available]

Indexed keywords

CELL ANTIGEN; MAJOR HISTOCOMPATIBILITY ANTIGEN CLASS 1; TUMOR NECROSIS FACTOR ALPHA; AUTOANTIGEN;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ANTIGEN PRESENTATION; ARTICLE; AUTOIMMUNITY; B LYMPHOCYTE; CONTROLLED STUDY; DENDRITIC CELL; DIABETES MELLITUS; HISTOPATHOLOGY; IMMUNOLOGICAL TOLERANCE; IMMUNOPATHOGENESIS; INFLAMMATION; MACROPHAGE; MOUSE; NONHUMAN; PANCREAS ISLET CELL; PRIORITY JOURNAL; T LYMPHOCYTE; ANIMAL; APOPTOSIS; BIOSYNTHESIS; DISEASE COURSE; FEMALE; IMMUNOLOGY; INSULIN DEPENDENT DIABETES MELLITUS; LYMPHOCYTE ACTIVATION; MALE; METABOLISM; MOUSE MUTANT; NONOBESE DIABETIC MOUSE; PANCREAS ISLET; PATHOLOGY; PHYSIOLOGY; SENSITIVITY AND SPECIFICITY; TRANSGENE; TRANSGENIC MOUSE;

ANIMALS; ANTIGEN PRESENTATION; APOPTOSIS; AUTOANTIGENS; B-LYMPHOCYTES; DENDRITIC CELLS; DIABETES MELLITUS, TYPE 1; DISEASE PROGRESSION; FEMALE; ISLETS OF LANGERHANS; LYMPHOCYTE ACTIVATION; MALE; MICE; MICE, INBRED NOD; MICE, SCID; MICE, TRANSGENIC; SENSITIVITY AND SPECIFICITY; T-LYMPHOCYTES; TRANSGENES; TUMOR NECROSIS FACTOR-ALPHA;

EID: 0032214715     PISSN: 10747613     EISSN: None     Source Type: Journal    
DOI: 10.1016/S1074-7613(00)80670-6     Document Type: Article

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