Iodoacetate protects hippocampal neurons against excitotoxic and oxidative injury: Involvement of heat-shock proteins and Bcl-2

Journal of Neurochemistry

Volumn 79, Issue 2, 2001, Pages 361-370

  Guo, Zhihong ^a   Lee, Jaewon ^a   Lane, Mark ^a   Mattson, Mark P ^a,b  

^a NATIONAL INSTITUTE ON AGING   (United States)

^b JOHNS HOPKINS UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

Apoptosis; Cerebral ischemia; Glutamate; Heat shock proteins 70 and 90; Membrane lipid peroxidation; Mitochondrial transmembrane potential

Indexed keywords

ADENOSINE TRIPHOSPHATE; GLUTAMIC ACID; GLYCERALDEHYDE 3 PHOSPHATE DEHYDROGENASE; HEAT SHOCK PROTEIN 70; HEAT SHOCK PROTEIN 90; IODOACETIC ACID; IRON; PROTEIN BCL 2;

ANIMAL CELL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; BRAIN INJURY; CELL DEATH; CONTROLLED STUDY; EMBRYO; HIPPOCAMPUS; IMMUNOBLOTTING; NERVE CELL CULTURE; NEUROPROTECTION; NONHUMAN; OXIDATIVE STRESS; PRIORITY JOURNAL; RAT;

ADENOSINE TRIPHOSPHATE; ANIMALS; CELL DEATH; CELLS, CULTURED; GLUTAMIC ACID; HIPPOCAMPUS; HSP70 HEAT-SHOCK PROTEINS; HSP90 HEAT-SHOCK PROTEINS; IODOACETATES; IODOACETIC ACID; IRON; MEMBRANE POTENTIALS; NEURONS; NEUROPROTECTIVE AGENTS; NEUROTOXINS; OSMOLAR CONCENTRATION; OXIDATIVE STRESS; PROTO-ONCOGENE PROTEINS C-BCL-2; RATS; RATS, SPRAGUE-DAWLEY;

ANIMALIA;

EID: 0034781048     PISSN: 00223042     EISSN: None     Source Type: Journal    
DOI: 10.1046/j.1471-4159.2001.00564.x     Document Type: Article

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