Protein kinase C iota protects neural cells against apoptosis induced by amyloid β-peptide

Molecular Brain Research

Volumn 82, Issue 1-2, 2000, Pages 107-113

  Xie, Jun ^a   Guo, Qing ^a   Zhu, Haiyan ^a   Wooten, Marie W ^b   Mattson, Mark P ^a,c  

^a UNIVERSITY OF KENTUCKY   (United States)

^b AUBURN UNIVERSITY   (United States)

^c NATIONAL INSTITUTE ON AGING   (United States)

Author keywords

Alzheimer's disease; Calcium; Caspase; Mitochondrial membrane potential; Oxidative stress

Indexed keywords

AMYLOID BETA PROTEIN; CALCIUM ION; CASPASE; PROTEIN KINASE C; PROTEIN KINASE C IOTA; UNCLASSIFIED DRUG;

ALZHEIMER DISEASE; ANIMAL CELL; APOPTOSIS; ARTICLE; BRAIN MITOCHONDRION; BRAIN PROTECTION; CELL SURVIVAL; CONTROLLED STUDY; ENZYME POLYMORPHISM; MEMBRANE POTENTIAL; MITOCHONDRIAL MEMBRANE; NERVE CELL PLASTICITY; NONHUMAN; OXIDATIVE STRESS; PRIORITY JOURNAL; PROTEIN EXPRESSION; RAT;

AMYLOID BETA-PROTEIN; ANIMAL; APOPTOSIS; CALCIUM; CASPASES; ISOENZYMES; MITOCHONDRIA; OXIDATIVE STRESS; PC12 CELLS; PEPTIDE FRAGMENTS; PROTEIN KINASE C; RATS; RECOMBINANT PROTEINS; SUPPORT, U.S. GOV'T, P.H.S.; TRANSFECTION;

ANIMALIA;

EID: 0034693333     PISSN: 0169328X     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0169-328X(00)00187-X     Document Type: Article

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