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Volumn 66, Issue 5, 1996, Pages 1836-1844
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Estrogens attenuate and corticosterone exacerbates excitotoxicity, oxidative injury, and amyloid β-peptide toxicity in hippocampal neurons
a a a a |
Author keywords
Alzheimer's disease; Confocal laser scanning microscope; Estradiol Free radicals; Glucocorticoids; Lipid peroxidation; Neuronal death; Progesterone; Testosterone; Thiobarbituric acid
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Indexed keywords
AMYLOID BETA PROTEIN;
CORTICOSTERONE;
ESTRADIOL;
ESTRIOL;
ESTROGEN;
EXCITOTOXIN;
FERROUS SULFATE;
FREE RADICAL;
GLUCOCORTICOID;
GLUCOCORTICOID RECEPTOR;
GLUCOSE;
GLUTAMIC ACID;
PROGESTERONE;
THIOBARBITURIC ACID;
ALZHEIMER DISEASE;
ANIMAL CELL;
ANIMAL TISSUE;
ANTIOXIDANT ACTIVITY;
ARTICLE;
ATTENUATION;
CALCIUM CELL LEVEL;
CALCIUM HOMEOSTASIS;
CALCIUM TRANSPORT;
CONFOCAL LASER MICROSCOPY;
DEGENERATIVE DISEASE;
HIPPOCAMPUS;
LIPID PEROXIDATION;
NONHUMAN;
OXIDATION;
PRIORITY JOURNAL;
RAT;
ANIMALIA;
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EID: 0029924284
PISSN: 00223042
EISSN: None
Source Type: Journal
DOI: 10.1046/j.1471-4159.1996.66051836.x Document Type: Article |
Times cited : (808)
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References (0)
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