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Oncogene

Volumn 19, Issue 47, 2000, Pages 5338-5347

Loss of p21(WAF1/CIP1) accelerates Ras oncogenesis in a transgenic/knockout mammary cancer model

(6) Adnane, Jalila a Jackson, Rosalind J a Nicosia, Santo V a Cantor, Alan B a Pledger, W Jack a Sebti, Saïd M a

a H LEE MOFFITT CANCER CENTER AND RESEARCH INSTITUTE (United States)

Author keywords

Mammary tumor model; P21(WAF1 CIP1); Ras oncogene

Indexed keywords

PROTEIN P21; RAS PROTEIN; CDKN1A PROTEIN, MOUSE; CYCLIN DEPENDENT KINASE INHIBITOR 1A; CYCLINE;

ANIMAL CELL; ANIMAL MODEL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; BREAST CANCER; CONTROLLED STUDY; HISTOPATHOLOGY; MITOSIS INDEX; MOUSE; NONHUMAN; ONCOGENE RAS; PRIORITY JOURNAL; PROTEIN DEFECT; PROTEIN PROTEIN INTERACTION; ADENOCARCINOMA; ANIMAL; DISEASE MODEL; EXPERIMENTAL NEOPLASM; FEMALE; GENE EXPRESSION; GENETICS; MALE; MOUSE MUTANT; PAGET NIPPLE DISEASE; PATHOLOGY; PATHOPHYSIOLOGY; PHYSIOLOGY; SALIVARY GLAND TUMOR; TRANSGENIC MOUSE;

ANIMALIA; MUS MUSCULUS;

ADENOCARCINOMA; ANIMALS; CARCINOMA, DUCTAL, BREAST; CYCLIN-DEPENDENT KINASE INHIBITOR P21; CYCLINS; DISEASE MODELS, ANIMAL; FEMALE; GENE EXPRESSION; GENES, RAS; MALE; MAMMARY NEOPLASMS, ANIMAL; MICE; MICE, KNOCKOUT; MICE, TRANSGENIC; ONCOGENE PROTEIN P21(RAS); SALIVARY GLAND NEOPLASMS;

EID: 0034626629 PISSN: 09509232 EISSN: None Source Type: Journal
DOI: 10.1038/sj.onc.1203956 Document Type: Article

Times cited : (83)

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