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Volumn 914, Issue , 2000, Pages 92-103

Molecular footprints of neurotoxic amphetamine action

Author keywords

[No Author keywords available]

Indexed keywords

1,4 BENZOQUINONE; 3,4 METHYLENEDIOXYMETHAMPHETAMINE; AMPHETAMINE DERIVATIVE; CATECHOL; DOPAMINE; METHAMPHETAMINE; NITRIC OXIDE; NITROGEN; NUCLEOTIDE DERIVATIVE; PEROXYNITRITE; PROTEIN DERIVATIVE; REACTIVE OXYGEN METABOLITE; SEROTONIN; TRYPTOPHAN HYDROXYLASE; TYROSINE 3 MONOOXYGENASE;

EID: 0033710752     PISSN: 00778923     EISSN: None     Source Type: Book Series    
DOI: 10.1111/j.1749-6632.2000.tb05187.x     Document Type: Article
Times cited : (36)

References (57)
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  • 15
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    • Dopamine inactivates tryptophan hydroxylase and forms a redox-cycling quinoprotein: Possible endogenous toxin to serotonin neurons
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    • Molecular mechanism of the inactivation of tryptophan hydroxylase by nitric oxide: Attack on critical sulfhydryls that spare the enzyme iron center
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    • Kuhn, D.M.1    Arthur R., Jr.2
  • 33
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    • The role of temperature, stress, and other factors in the neurotoxicity of the substituted amphetamines 3,4-methylenedioxymethamphetamine and fenfluramine
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    • Miller, D.B.1    O'Callaghan, J.P.2


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.