Absence of NOD2 receptor predisposes to intestinal inflammation by a deregulation in the immune response in hosts that are unable to control gut dysbiosis

Immunobiology

Volumn 223, Issue 10, 2018, Pages 577-585

  de Souza, Patrícia Reis ^a,b   Guimarães, Francielle Rodrigues ^a,b   Sales Campos, Helioswilton ^a,b,c   Bonfá, Giuliano ^a   Nardini, Viviani ^a   Chica, Javier Emilio Lazo ^b   Turato, Walter Miguel ^a   Silva, João Santana ^a   Zamboni, Dario Simões ^a   Cardoso, Cristina Ribeiro de Barros ^a  

^a UNIVERSITY OF SÃO PAULO   (Brazil)

^b FEDERAL UNIVERSITY OF TRIÂNGULO MINEIRO   (Brazil)

^c FEDERAL UNIVERSITY OF GOIÁS   (Brazil)

Author keywords

Inflammatory bowel disease; Innate immunity; Microbiota; NOD2

Indexed keywords

ANTIBIOTIC AGENT; CASPASE RECRUITMENT DOMAIN PROTEIN 15; CYTOTOXIC T LYMPHOCYTE ANTIGEN 4; GAMMA INTERFERON; INTERLEUKIN 1BETA; INTERLEUKIN 4; INTERLEUKIN 5; TRANSCRIPTION FACTOR FOXP3; CARD15 PROTEIN, MOUSE;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ANTIBIOTIC THERAPY; ARTICLE; CD4+ T LYMPHOCYTE; CD8+ T LYMPHOCYTE; COLITIS; CONTROLLED STUDY; DISEASE CONTROL; DISEASE PREDISPOSITION; DYSBIOSIS; ENTERITIS; EXPERIMENTAL COLITIS; GENE MUTATION; HOST RESISTANCE; IMMUNE RESPONSE; IMMUNOMODULATION; INTESTINE FLORA; MALE; MESENTERY LYMPH NODE; MOUSE; NONHUMAN; PRIORITY JOURNAL; SPLEEN; TH17 CELL; THERAPY EFFECT; ANIMAL; BIOSYNTHESIS; GENETICS; IMMUNOLOGY; INFLAMMATORY BOWEL DISEASE; KNOCKOUT MOUSE; MICROBIOLOGY;

ANIMALS; COLITIS; DYSBIOSIS; GASTROINTESTINAL MICROBIOME; INFLAMMATORY BOWEL DISEASES; INTERLEUKIN-1BETA; INTERLEUKIN-5; MICE; MICE, KNOCKOUT; NOD2 SIGNALING ADAPTOR PROTEIN;

EID: 85049443785     PISSN: 01712985     EISSN: 18783279     Source Type: Journal    
DOI: 10.1016/j.imbio.2018.07.003     Document Type: Article

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